Chemical Speciation of Fe and Ni in Residual Oil Fly Ash Fine Particulate Matter Using X-ray Absorption Spectroscopy

2012 ◽  
Vol 46 (23) ◽  
pp. 12927-12935 ◽  
Author(s):  
Sidhartha Pattanaik ◽  
Frank E. Huggins ◽  
Gerald P. Huffman
Keyword(s):  
Particulate Matter ◽  
Fly Ash ◽  
Chemical Speciation ◽  
Fine Particulate Matter ◽  
Residual Oil ◽  
X Ray ◽  
Residual Oil Fly Ash ◽  
Fine Particulate ◽  
Oil Fly Ash ◽  
X Ray Absorption

Nickel Speciation of Residual Oil Fly Ash and Ambient Particulate Matter Using X-ray Absorption Spectroscopy

2000 ◽  
Vol 50 (11) ◽  
pp. 1876-1886 ◽  
Author(s):  
Kevin C. Galbreath, ◽  
Donald L. Toman ◽  
Christopher J. Zygarlicke ◽  
Frank E. Huggins ◽  
Gerald P. Huffman ◽  
...  
Keyword(s):  
Particulate Matter ◽  
Fly Ash ◽  
Absorption Spectroscopy ◽  
Ambient Particulate Matter ◽  
Residual Oil ◽  
X Ray ◽  
Residual Oil Fly Ash ◽  
Oil Fly Ash ◽  
X Ray Absorption

EFFECT OF INHALED RESIDUAL OIL FLY ASH ON THE ELECTROCARDIOGRAM OF DOGS

2000 ◽  
Vol 12 (11) ◽  
pp. 189-208 ◽  
Author(s):  
B. A. Muggenburg ◽  
E. B. Barr ◽  
Y. S. Cheng ◽  
J. C. Seagrave ◽  
L. P. Tilley ◽  
...  
Keyword(s):  
Fly Ash ◽  
Residual Oil ◽  
Residual Oil Fly Ash ◽  
Oil Fly Ash

Induction of prostaglandin H synthase 2 in human airway epithelial cells exposed to residual oil fly ash

1996 ◽  
Vol 141 (1) ◽  
pp. 159-168 ◽  
Author(s):  
James M. Samet ◽  
William Reed ◽  
Andrew J. Ghio ◽  
Robert B. Devlin ◽  
Jacqueline D. Carter ◽  
...  
Keyword(s):  
Fly Ash ◽  
Epithelial Cells ◽  
Airway Epithelial Cells ◽  
Airway Epithelial ◽  
Residual Oil ◽  
Prostaglandin H Synthase ◽  
Residual Oil Fly Ash ◽  
Human Airway ◽  
Prostaglandin H ◽  
Oil Fly Ash

Anti-oxidants reduce the acute adverse effects of residual oil fly ash on the frog palate mucociliary epithelium

2005 ◽  
Vol 98 (3) ◽  
pp. 349-354 ◽  
Author(s):  
Regiani Carvalho-Oliveira ◽  
Mitiko Saiki ◽  
Ruy C. Pires-Neto ◽  
Geraldo Lorenzi-Filho ◽  
Mariangela Macchione ◽  
...  
Keyword(s):  
Fly Ash ◽  
Adverse Effects ◽  
Residual Oil ◽  
Residual Oil Fly Ash ◽  
Frog Palate ◽  
Oil Fly Ash

Role of Toll-like receptor-4 in genetic susceptibility to lung injury induced by residual oil fly ash

2005 ◽  
Vol 22 (1) ◽  
pp. 108-117 ◽  
Author(s):  
Hye-Youn Cho ◽  
Anne E. Jedlicka ◽  
Robert Clarke ◽  
Steven R. Kleeberger
Keyword(s):  
Fly Ash ◽  
Lung Injury ◽  
Genetic Background ◽  
Signal Molecules ◽  
Residual Oil ◽  
Toll Like Receptor ◽  
Toll Like Receptor 4 ◽  
Residual Oil Fly Ash ◽  
Oil Fly Ash

The mechanisms of susceptibility to particle-induced lung injury are not clearly understood. To evaluate the contribution of genetic background to pulmonary pathogenesis, we compared the lung injury responses to residual oil fly ash (ROFA) in inbred mouse strains and calculated heritability estimates. Significant interstrain (genetic) variation was observed in ROFA-induced lung inflammation and hyperpermeability phenotypes; broad-sense heritability ranged from ∼0.43 to 0.62, and the coefficient of genetic determination ranged from 0.28 to 0.45. C3H/HeJ (HeJ) mice were most resistant to the ROFA-induced injury responses. This was particularly important, as HeJ mice contain a dominant negative mutation in Toll-like receptor-4 ( Tlr4). We then characterized ROFA-induced injury and TLR4 signaling in HeJ mice and its coisogenic strain C3H/HeOuJ (OuJ; Tlr4 normal) to understand the potential role of Tlr4 in this model. ROFA-induced lung injury was significantly greater in OuJ mice compared with HeJ mice. ROFA also significantly enhanced transcript and protein levels of lung TLR4 in OuJ but not in HeJ mice. Greater activation of downstream signal molecules (i.e., MYD88, TRAF6, IRAK-1, NF-κB, MAPK, AP-1) was observed in OuJ mice than in HeJ mice before the development of ROFA-induced pulmonary injury. Putative TLR4-dependent inflammatory genes that were differentially induced by ROFA in the two strains include interleukin-1β and tumor necrosis factor-α. Results support an important contribution of genetic background to particle-mediated lung injury, and Tlr4 is a candidate susceptibility gene.

Role of metal-induced reactive oxygen species generation in lung responses caused by residual oil fly ash

2003 ◽  
Vol 28 (1) ◽  
pp. 13-18 ◽  
Author(s):  
Anthony B. Lewis ◽  
Michael D. Taylor ◽  
Jenny R. Roberts ◽  
Stephen S. Leonard ◽  
Xianglin Shi ◽  
...  
Keyword(s):  
Reactive Oxygen Species ◽  
Fly Ash ◽  
Reactive Oxygen Species Generation ◽  
Reactive Oxygen ◽  
Oxygen Species ◽  
Residual Oil ◽  
Residual Oil Fly Ash ◽  
Oil Fly Ash

scholarly journals Residual oil fly ash worsens pulmonary hyperreactivity in chronic allergic mice

2011 ◽  
Vol 179 (2-3) ◽  
pp. 151-157 ◽  
Author(s):  
Mariana Barcellos Avila ◽  
Flavia Mazzoli-Rocha ◽  
Clarissa Bichara Magalhães ◽  
Paulo Hilário Nascimento Saldiva ◽  
Alysson Roncally Carvalho ◽  
...  
Keyword(s):  
Fly Ash ◽  
Residual Oil ◽  
Residual Oil Fly Ash ◽  
Oil Fly Ash
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