scholarly journals Probing the Formation of Reactive Oxygen Species by a Porous Self-Assembled Benzophenone Bis-Urea Host

ACS Omega ◽  
2019 ◽  
Vol 4 (5) ◽  
pp. 8290-8298 ◽  
Author(s):  
Baillie A. DeHaven ◽  
Hannah K. Liberatore ◽  
Alexander Greer ◽  
Susan D. Richardson ◽  
Linda S. Shimizu
Keyword(s):  
Reactive Oxygen Species ◽  
Reactive Oxygen ◽  
Oxygen Species ◽  
Self Assembled

Toxic Reactive Oxygen Species Enhanced Synergistic Combination Therapy by Self-Assembled Metal-Phenolic Network Nanoparticles

2018 ◽  
Vol 30 (8) ◽  
pp. 1704877 ◽  
Author(s):  
Yunlu Dai ◽  
Zhen Yang ◽  
Siyuan Cheng ◽  
Zhongliang Wang ◽  
Ruili Zhang ◽  
...  
Keyword(s):  
Reactive Oxygen Species ◽  
Combination Therapy ◽  
Reactive Oxygen ◽  
Oxygen Species ◽  
Synergistic Combination ◽  
Self Assembled

Phthalocyanine functionalized poly(glycidyl methacrylate) nano-assemblies for photodynamic inactivation of bacteria

2019 ◽  
Vol 7 (5) ◽  
pp. 1905-1918 ◽  
Author(s):  
Wei Tong ◽  
Yanhua Xiong ◽  
Shun Duan ◽  
Xiaokang Ding ◽  
Fu-Jian Xu
Keyword(s):  
Reactive Oxygen Species ◽  
Aqueous Solution ◽  
Glycidyl Methacrylate ◽  
Bacterial Infections ◽  
Reactive Oxygen ◽  
Photodynamic Inactivation ◽  
Oxygen Species ◽  
Self Assembled ◽  
Inactivation Of Bacteria

Self-assembled PGED-Pc nanoparticles are able to inactivate bacteria via the generation of reactive oxygen species in aqueous solution, while a facile immobilization strategy sheds light on the engineering of self-sterilizing surfaces to combat bacterial infections.

Mitochondria as a source of reactive oxygen species

2009 ◽  
pp. c3 ◽  
Author(s):  
Helena M. Cochemé ◽  
Michael P. Murphy
Keyword(s):  
Reactive Oxygen Species ◽  
Reactive Oxygen ◽  
Oxygen Species

Clinical aspects of reactive oxygen and nitrogen species

2004 ◽  
Vol 71 ◽  
pp. 121-133 ◽  
Author(s):  
Ascan Warnholtz ◽  
Maria Wendt ◽  
Michael August ◽  
Thomas Münzel
Keyword(s):  
Oxidative Stress ◽  
Reactive Oxygen Species ◽  
Risk Factor ◽  
Endothelial Dysfunction ◽  
Vascular Tissue ◽  
Rapid Development ◽  
Reactive Oxygen ◽  
Clinical Aspects ◽  
No Production ◽  
Oxygen Species

Endothelial dysfunction in the setting of cardiovascular risk factors, such as hypercholesterolaemia, hypertension, diabetes mellitus and chronic smoking, as well as in the setting of heart failure, has been shown to be at least partly dependent on the production of reactive oxygen species in endothelial and/or smooth muscle cells and the adventitia, and the subsequent decrease in vascular bioavailability of NO. Superoxide-producing enzymes involved in increased oxidative stress within vascular tissue include NAD(P)H-oxidase, xanthine oxidase and endothelial nitric oxide synthase in an uncoupled state. Recent studies indicate that endothelial dysfunction of peripheral and coronary resistance and conductance vessels represents a strong and independent risk factor for future cardiovascular events. Ways to reduce endothelial dysfunction include risk-factor modification and treatment with substances that have been shown to reduce oxidative stress and, simultaneously, to stimulate endothelial NO production, such as inhibitors of angiotensin-converting enzyme or the statins. In contrast, in conditions where increased production of reactive oxygen species, such as superoxide, in vascular tissue is established, treatment with NO, e.g. via administration of nitroglycerin, results in a rapid development of endothelial dysfunction, which may worsen the prognosis in patients with established coronary artery disease.

Fas ligand expression in rat kupffer cells in response to lipopolysaccharide is mediated by reactive oxygen species

2001 ◽  
Vol 120 (5) ◽  
pp. A361-A361
Author(s):  
K UCHIKURA ◽  
T WADA ◽  
Z SUN ◽  
S HOSHINO ◽  
G BULKLEY ◽  
...  
Keyword(s):  
Reactive Oxygen Species ◽  
Kupffer Cells ◽  
Fas Ligand ◽  
Reactive Oxygen ◽  
Oxygen Species ◽  
Ligand Expression
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