Evolution through bursts: Network structure develops through localized bursts in time and space

2016 ◽  
Vol 4 (3) ◽  
pp. 293-313 ◽  
Author(s):  
HILLA BROT ◽  
LEV MUCHNIK ◽  
JACOB GOLDENBERG ◽  
YORAM LOUZOUN

Models of network evolution are based on the implicit assumption that network growth is continuous, uniform, and steady. Using the data collected from a large online-blogging platform, we show that the addition and removal of network ties by users do not occur sporadically at isolated nodes spread all over the network, as assumed by the vast majority of stochastic network models, but rather occur in brief bursts of intense local activity.These bursts of network growth and attrition (addition and removal of network ties) are highly localized around focal nodes. Such network changes coincide with nearly instantaneous densification of the ties between the affected nodes, resulting in an increase of local clustering. Furthermore, we find that these network changes are tightly coupled to the dynamics of individual attributes, particularly the increase in homology between neighboring nodes (homophily) within the scope of the burst. Coincidence of the localized network change with the increase in homophily suggests a strong coupling between the selection and influence processes that lead to simultaneous elevation of assortativity and clustering.

2019 ◽  
Vol 23 (3) ◽  
pp. 511-534 ◽  
Author(s):  
Yuval Kalish

Stochastic actor-oriented (SAO) models are a family of models for network dynamics that enable researchers to test multiple, often competing explanations for network change and estimate the extent and relative power of various influences on network evolution. SAO models for the co-evolution of network ties and actor behavior, the most comprehensive category of SAO models, examine how networks and actor attributes—their behavior, performance, or attitudes—influence each other over time. While these models have been widely used in the social sciences, and particularly in educational settings, their use in organizational scholarship has been extremely limited. This paper provides a layperson introduction to SAO models for the co-evolution of networks and behavior and the types of research questions they can address. The models and their underpinnings are explained in nonmathematical terms, and theoretical explanations are supported by a concrete, detailed example that includes step-by-step model building and hypothesis testing, alongside an R script.


2019 ◽  
pp. 1-9 ◽  
Author(s):  
Jill de Ron ◽  
Eiko I. Fried ◽  
Sacha Epskamp

Abstract Background In clinical research, populations are often selected on the sum-score of diagnostic criteria such as symptoms. Estimating statistical models where a subset of the data is selected based on a function of the analyzed variables introduces Berkson's bias, which presents a potential threat to the validity of findings in the clinical literature. The aim of the present paper is to investigate the effect of Berkson's bias on the performance of the two most commonly used psychological network models: the Gaussian Graphical Model (GGM) for continuous and ordinal data, and the Ising Model for binary data. Methods In two simulation studies, we test how well the two models recover a true network structure when estimation is based on a subset of the data typically seen in clinical studies. The network is based on a dataset of 2807 patients diagnosed with major depression, and nodes in the network are items from the Hamilton Rating Scale for Depression (HRSD). The simulation studies test different scenarios by varying (1) sample size and (2) the cut-off value of the sum-score which governs the selection of participants. Results The results of both studies indicate that higher cut-off values are associated with worse recovery of the network structure. As expected from the Berkson's bias literature, selection reduced recovery rates by inducing negative connections between the items. Conclusion Our findings provide evidence that Berkson's bias is a considerable and underappreciated problem in the clinical network literature. Furthermore, we discuss potential solutions to circumvent Berkson's bias and their pitfalls.


2021 ◽  
Vol 8 (1) ◽  
Author(s):  
Vesa Kuikka

AbstractWe present methods for analysing hierarchical and overlapping community structure and spreading phenomena on complex networks. Different models can be developed for describing static connectivity or dynamical processes on a network topology. In this study, classical network connectivity and influence spreading models are used as examples for network models. Analysis of results is based on a probability matrix describing interactions between all pairs of nodes in the network. One popular research area has been detecting communities and their structure in complex networks. The community detection method of this study is based on optimising a quality function calculated from the probability matrix. The same method is proposed for detecting underlying groups of nodes that are building blocks of different sub-communities in the network structure. We present different quantitative measures for comparing and ranking solutions of the community detection algorithm. These measures describe properties of sub-communities: strength of a community, probability of formation and robustness of composition. The main contribution of this study is proposing a common methodology for analysing network structure and dynamics on complex networks. We illustrate the community detection methods with two small network topologies. In the case of network spreading models, time development of spreading in the network can be studied. Two different temporal spreading distributions demonstrate the methods with three real-world social networks of different sizes. The Poisson distribution describes a random response time and the e-mail forwarding distribution describes a process of receiving and forwarding messages.


2021 ◽  
Vol 11 (1) ◽  
Author(s):  
Naomi A. Arnold ◽  
Raul J. Mondragón ◽  
Richard G. Clegg

AbstractDiscriminating between competing explanatory models as to which is more likely responsible for the growth of a network is a problem of fundamental importance for network science. The rules governing this growth are attributed to mechanisms such as preferential attachment and triangle closure, with a wealth of explanatory models based on these. These models are deliberately simple, commonly with the network growing according to a constant mechanism for its lifetime, to allow for analytical results. We use a likelihood-based framework on artificial data where the network model changes at a known point in time and demonstrate that we can recover the change point from analysis of the network. We then use real datasets and demonstrate how our framework can show the changing importance of network growth mechanisms over time.


2019 ◽  
Vol 5 ◽  
pp. 237802311987979 ◽  
Author(s):  
George Wood ◽  
Daria Roithmayr ◽  
Andrew V. Papachristos

Conventional explanations of police misconduct generally adopt a microlevel focus on deviant officers or a macrolevel focus on the top-down organization of police departments. Between these levels are social networks of misconduct. This study recreates these networks using data on 16,503 complaints and 15,811 police officers over a six-year period in Chicago. We examine individual-level factors associated with receiving a complaint, the basic properties of these misconduct networks, and factors related to officer co-naming in complaints. We find that the incidence of police misconduct is associated with attributes including race, age, and tenure and that almost half of police officers are connected in misconduct ties in broader networks of misconduct. We also find that certain dyadic factors, especially seniority and race, strongly predict network ties and the incidence of group misconduct. Our results provide actionable information regarding possible ways to leverage the co-complaint network structure to reduce misconduct.


2017 ◽  
Vol 284 (1854) ◽  
pp. 20162302 ◽  
Author(s):  
Evan C. Fricke ◽  
Joshua J. Tewksbury ◽  
Elizabeth M. Wandrag ◽  
Haldre S. Rogers

The global decline of mutualists such as pollinators and seed dispersers may cause negative direct and indirect impacts on biodiversity. Mutualistic network models used to understand the stability of mutualistic systems indicate that species with low partner diversity are most vulnerable to coextinction following mutualism disruption. However, existing models have not considered how species vary in their dependence on mutualistic interactions for reproduction or survival, overlooking the potential influence of this variation on species' coextinction vulnerability and on network stability. Using global databases and field experiments focused on the seed dispersal mutualism, we found that plants and animals that depend heavily on mutualistic interactions have higher partner diversity. Under simulated network disruption, this empirical relationship strongly reduced coextinction because the species most likely to lose mutualists depend least on their mutualists. The pattern also reduced the importance of network structure for stability; nested network structure had little effect on coextinction after simulations incorporated the empirically derived relationship between partner diversity and mutualistic dependence. Our results highlight a previously unknown source of stability in mutualistic networks and suggest that differences among species in their mutualistic strategy, rather than network structure, primarily accounts for stability in mutualistic communities.


2019 ◽  
Author(s):  
Jill de Ron ◽  
Eiko I Fried ◽  
Sacha Epskamp

In clinical research, populations are often selected on the sum-score of diagnostic criteria such as symptoms. Estimating statistical models where a subset of the data is selected based on a function of the analyzed variables introduces Berkson’s bias, which presents a potential threat to the validity of findings in the clinical literature. The aim of the present paper is to investigate the effect of Berkson’s bias on the performance of the two most commonly used psychological network models: the Gaussian Graphical Model (GGM) for continuous and ordinal data, and the Ising Model for binary data. In two simulation studies, we test how well the two models recover a true network structure when estimation is based on a subset of the data typically seen in clinical studies. The network is based on a dataset of 2,807 patients diagnosed with major depression, and nodes in the network are items from the Hamilton Rating Scale for Depression (HRSD). The simulation studies test different scenarios by varying (1) sample size and (2) the cut-off value of the sum-score which governs the selection of participants. The results of both studies indicate that higher cut-off values are associated with worse recovery of the network structure. As expected from the Berkson’s bias literature, selection reduced recovery rates by inducing negative connections between the items. Our findings provide evidence that Berkson’s bias is a considerable and underappreciated problem in the clinical network literature. Furthermore, we discuss potential solutions to circumvent Berkson’s bias and their pitfalls.


2019 ◽  
Author(s):  
Carter J. Funkhouser ◽  
Kelly Correa

The popularity of network analysis in psychopathology research has increased exponentially in recent years. Yet, little research has examined the replicability of cross-sectional psychopathology network models, and those that have used single items for symptoms rather than multi-item scales. The present study therefore examined the replicability and generalizability of regularized partial correlation networks of internalizing symptoms within and across five samples (total N = 2,573) using the Inventory for Depression and Anxiety Symptoms, a factor analytically-derived measure of individual internalizing symptoms. As different metrics may yield different conclusions about the replicability of network parameters, we examined both global and specific metrics of similarity between networks. Correlations within and between nonclinical samples suggested considerable global similarities in network structure (rss = .53-.87) and centrality strength (rss = .37-.86), but weaker similarities in network structure (rss = .36-.66) and centrality (rss = .04-.54) between clinical and nonclinical samples. Global strength (i.e., connectivity) did not significantly differ across all five networks and few edges (0-5.5%) significantly differed between networks. Specific metrics of similarity indicated that, on average, approximately 80% of edges were consistently estimated within and between all five samples. The most central symptom (i.e., dysphoria) was consistent within and across samples, but there were few other matches in centrality rank-order. In sum, there were considerable similarities in network structure, the presence and sign of individual edges, and the most central symptom within and across internalizing symptom networks estimated from nonclinical samples, but global metrics suggested network structure and symptom centrality had weak to moderate generalizability from nonclinical to clinical samples.


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