scholarly journals CORRIGENDUM “Metabolic abnormality in the right dorsolateral prefrontal cortex in patients with obsessive-compulsive disorder: proton magnetic resonance spectroscopy”; [Acta Neuropsychiatr 2017;29(3):164-169]

2018 ◽  
Vol 30 (2) ◽  
pp. 123-123
Author(s):  
Shin-Eui Park ◽  
Nam-Gil Choi ◽  
Gwang-Woo Jeong
Keyword(s):  
Prefrontal Cortex ◽  
Magnetic Resonance ◽  
Dorsolateral Prefrontal Cortex ◽  
Proton Magnetic Resonance ◽  
Proton Magnetic Resonance Spectroscopy ◽  
Resonance Spectroscopy ◽  
Obsessive Compulsive ◽  
Compulsive Disorder ◽  
Dorsolateral Prefrontal ◽  
The Right

Metabolic abnormality in the right dorsolateral prefrontal cortex in patients with obsessive–compulsive disorder: proton magnetic resonance spectroscopy

2016 ◽  
Vol 29 (3) ◽  
pp. 164-169 ◽  
Author(s):  
Shin-Eui Park ◽  
Nam-Gil Choi ◽  
Gwang-Woo Jeong
Keyword(s):  
Prefrontal Cortex ◽  
Dorsolateral Prefrontal Cortex ◽  
Proton Magnetic Resonance ◽  
Proton Magnetic Resonance Spectroscopy ◽  
Resonance Spectroscopy ◽  
Healthy Controls ◽  
Obsessive Compulsive ◽  
Compulsive Disorder ◽  
Dorsolateral Prefrontal ◽  
The Right

ObjectiveProton magnetic resonance spectroscopy (1H-MRS) was used to evaluate metabolic changes in the dorsolateral prefrontal cortex (DLPFC) in patients with obsessive–compulsive disorder (OCD).MethodsIn total, 14 OCD patients (mean age 28.9±7.2 years) and 14 healthy controls (mean age 32.6±7.1 years) with no history of neurological and psychiatric illness participated in this study. Brain metabolite concentrations were measured from a localised voxel on the right DLPFC using a 3-Tesla 1H-MRS.ResultsThe metabolic concentration of myo-inositol in patients with OCD increased significantly by 52% compared with the healthy controls, whereas glutamine/glutamate was decreased by 11%. However, there were no significant differences in N-acetylaspartate, choline, lactate and lipid between the two groups.ConclusionThese findings would be helpful to understand the pathophysiology of OCD associated with the brain metabolic abnormalities in the right DLPFC.

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