Ureteral implantation technique and urologic complications in adult kidney transplantion

2003 ◽  
Vol 35 (7) ◽  
pp. 2420-2422 ◽  
Author(s):  
G.N Tzimas ◽  
H Hayati ◽  
J.I Tchervenkov ◽  
P.P Metrakos
Urology ◽  
2007 ◽  
Vol 70 (5) ◽  
pp. 893-897 ◽  
Author(s):  
Panco Georgiev ◽  
Christian Böni ◽  
Felix Dahm ◽  
Christine F. Maurus ◽  
Stefan Wildi ◽  
...  

2007 ◽  
Vol 177 (4S) ◽  
pp. 62-63
Author(s):  
Oleg Sukonko ◽  
Sergey Krasny ◽  
Sergey Polaykov ◽  
Alexandr Rolevich ◽  
Carsten H. Ohlmann ◽  
...  

2005 ◽  
Vol 173 (4S) ◽  
pp. 24-24
Author(s):  
Igor Frank ◽  
Bradley C. Leibovich ◽  
Christine M. Lohse ◽  
Jose R. Colombo ◽  
Horst Zincke ◽  
...  

1997 ◽  
Vol 272 (5) ◽  
pp. F602-F609 ◽  
Author(s):  
J. Van Adelsberg ◽  
S. Chamberlain ◽  
V. D'Agati

Mutations in PKD1 cause autosomal dominant polycystic kidney disease (ADPKD), a common genetic disease in which cysts form from kidney tubules. The predicted product of this gene is a novel protein with cell-adhesive and membrane-spanning domains. To test the hypothesis that polycystin, the product of the PKD1 gene, is a cell adhesion molecule, we raised antibodies against peptides derived from the unduplicated, membrane-spanning portion of the predicted amino acid sequence. These antibodies recognized membrane-associated polypeptides of 485 and 245 kDa in human fetal kidney homogenates. Expression was greater in fetal than adult kidney by both Western blot analysis and immunofluorescence. In fetal kidney, polycystin was localized to the plasma membranes of ureteric bud and comma and S-shaped bodies. However, in more mature tubules in fetal kidney, in adult kidney, and in polycystic kidney, the majority of polycystin staining was intracellular. The temporal and spatial regulation of polycystin expression during renal development lead us to speculate that polycystin may play a role in nephrogenesis.


EP Europace ◽  
2020 ◽  
Vol 22 (Supplement_2) ◽  
pp. ii3-ii9
Author(s):  
Wei Hua ◽  
Shu Zhang ◽  
Dejia Huang

2014 ◽  
Vol 13 (1) ◽  
pp. eV64 ◽  
Author(s):  
R.M. Bauer ◽  
S. Herschorn ◽  
T.B. Olmedo ◽  
O.D. Reyes ◽  
W. Huebner

Diagnostics ◽  
2021 ◽  
Vol 11 (3) ◽  
pp. 390
Author(s):  
Camilo G. Sotomayor ◽  
Stan Benjamens ◽  
Hildebrand Dijkstra ◽  
Derya Yakar ◽  
Cyril Moers ◽  
...  

Ultrasound examination is advised for early post-kidney transplant assessment. Grayscale median (GSM) quantification is novel in the kidney transplant field, with no systematic assessment previously reported. In this prospective cohort study, we measured the post-operative GSM in a large cohort of adult kidney transplant recipients (KTR) who consecutively underwent Doppler ultrasound directly after transplantation (within 24 h), compared it with GSM in nontransplanted patients, and investigated its association with baseline and follow-up characteristics. B-mode images were used to calculate the GSM in KTR and compared with GSM data in nontransplanted patients, as simulated from summary statistics of the literature using a Mersenne twister algorithm. The association of GSM with baseline and 1-year follow-up characteristics were studied by means of linear regression analyses. In 282 KTR (54 ± 15 years old, 60% male), the median (IQR) GSM was 55 (45–69), ranging from 22 to 124 (coefficient of variation = 7.4%), without differences by type of donation (p = 0.28). GSM in KTR was significantly higher than in nontransplanted patients (p < 0.001), and associated with systolic blood pressure, history of cardiovascular disease, and donor age (std. β = 0.12, −0.20, and 0.13, respectively; p < 0.05 for all). Higher early post-kidney transplant GSM was not associated with 1-year post-kidney transplant function parameters (e.g., measured and estimated glomerular filtration rate). The data provided in this study could be used as first step for further research on the application of early postoperative ultrasound in KTR.


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