Mild hypohydration induced by exercise in the heat attenuates autonomic thermoregulatory responses to the heat, but not thermal pleasantness in humans

2010 ◽  
Vol 100 (4) ◽  
pp. 340-345 ◽  
Author(s):  
Ken Tokizawa ◽  
Saki Yasuhara ◽  
Mayumi Nakamura ◽  
Yuki Uchida ◽  
Larry I. Crawshaw ◽  
...  
Keyword(s):  
Thermoregulatory Responses ◽  
Thermal Pleasantness

Heat balance and thermoregulatory responses in pinnaless rabbits

1980 ◽  
Vol 5 (1) ◽  
pp. 37-39 ◽  
Author(s):  
M. Caputa ◽  
W. Kądziela ◽  
J. Narąbski
Keyword(s):  
Heat Balance ◽  
Thermoregulatory Responses

Submandibular glands: novel structures participating in thermoregulatory responses

1997 ◽  
Vol 75 (5) ◽  
pp. 407-413 ◽  
Author(s):  
R D Mathison ◽  
T Malkinson ◽  
K E Cooper ◽  
J S Davison
Keyword(s):  
Submandibular Glands ◽  
Thermoregulatory Responses ◽  
Novel Structures

Thermoregulatory responses during competitive marathon running

1977 ◽  
Vol 42 (6) ◽  
pp. 909-914 ◽  
Author(s):  
M. B. Maron ◽  
J. A. Wagner ◽  
S. M. Horvath
Keyword(s):  
Skin Temperature ◽  
Marathon Running ◽  
Heat Illness ◽  
Mean Skin Temperature ◽  
Total Water ◽  
Water Losses ◽  
Thermoregulatory Responses ◽  
Marked Disparity ◽  
Skin Temperatures ◽  
Cool Conditions

To assess thermoregulatory responses occuring under actual marathon racing conditions, rectal (Tre) and five skin temperatures were measured in two runners approximately every 9 min of a competitive marathon run under cool conditions. Race times and total water losses were: runner 1 = 162.7 min, 3.02 kg; runner 2 = 164.6 min, 2.43 kg. Mean skin temperature was similar throughout the race in the two runners, although they exhibited a marked disparity in temperature at individual skin sites. Tre plateaued after 35--45 min (runner 1 = 40.0--40.1, runner 2 = 38.9--39.2 degrees C). While runner 2 maintained a relatively constant level for the remainder of the race, runner 1 exhibited a secondary increase in Tre. Between 113 and 119 min there was a precipitous rise in Tre from 40.9 to 41.9 degrees C. Partitional calorimetric calculations suggested that a decrease in sweating was responsible for this increment. However, runner 1's ability to maintain his high Tre and running pace for the remaining 44 min of the race and exhibit no signs of heat illness indicated thermoregulation was intact.

Ten-day endurance training attenuates the hyperosmotic suppression of cutaneous vasodilation during exercise but not sweating

2005 ◽  
Vol 99 (1) ◽  
pp. 237-243 ◽  
Author(s):  
Takashi Ichinose ◽  
Kazunobu Okazaki ◽  
Shizue Masuki ◽  
Hiroyuki Mitono ◽  
Mian Chen ◽  
...  
Keyword(s):  
Endurance Training ◽  
Exercise Intensity ◽  
Plasma Osmolality ◽  
Young Male ◽  
Cycle Ergometer ◽  
Peak Oxygen Consumption ◽  
Cutaneous Vasodilation ◽  
Thermoregulatory Responses ◽  
Before And After ◽  
The Individual

It is well known that hyperosmolality suppresses thermoregulatory responses and that plasma osmolality (Posmol) increases with exercise intensity. We examined whether the decreased esophageal temperature thresholds for cutaneous vasodilation (THFVC) and sweating (THSR) after 10-day endurance training (ET) are caused by either attenuated increase in Posmol at a given exercise intensity or blunted sensitivity of hyperosmotic suppression. Nine young male volunteers exercised on a cycle ergometer at 60% peak oxygen consumption rate (V̇o2 peak) for 1 h/day for 10 days at 30°C. Before and after ET, thermoregulatory responses were measured during 20-min exercise at pretraining 70% V̇o2 peak in the same environment as during ET under isoosmotic or hyperosmotic conditions. Hyperosmolality by ∼10 mosmol/kgH2O was attained by acute hypertonic saline infusion. After ET, V̇o2 peak and blood volume (BV) both increased by ∼4% ( P < 0.05), followed by a decrease in THFVC ( P < 0.05) but not by that in THSR. Although there was no significant decrease in Posmol at the thresholds after ET, the sensitivity of increase in THFVC at a given increase in Posmol [ΔTHFVC/ΔPosmol,°C·(mosmol/kgH2O)−1], determined by hypertonic infusion, was reduced to 0.021 ± 0.005 from 0.039 ± 0.004 before ET ( P < 0.05). The individual reductions in ΔTHFVC/ΔPosmol after ET were highly correlated with their increases in BV around THFVC ( r = −0.89, P < 0.005). In contrast, there was no alteration in the sensitivity of the hyperosmotic suppression of sweating after ET. Thus the downward shift of THFVC after ET was partially explained by the blunted sensitivity to hyperosmolality, which occurred in proportion to the increase in BV.

Thermoregulatory Responses To Racewalking Vs. Running In A Hot Environment

2010 ◽  
Vol 42 ◽  
pp. 278
Author(s):  
Juan F. Ortega ◽  
Nassim Hamouti ◽  
Jesus Gimenez ◽  
Ricardo Mora-Rodriguez
Keyword(s):  
Hot Environment ◽  
Thermoregulatory Responses

Role of central adenosine in the respiratory and thermoregulatory responses to hypoxia

Neuroreport ◽  
2000 ◽  
Vol 11 (1) ◽  
pp. 193-197 ◽  
Author(s):  
Renata C. H. Barros ◽  
Luiz G. S. Branco
Keyword(s):  
Thermoregulatory Responses
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