Corrigendum to “Krill oil and xanthigen separately inhibit high fat diet induced obesity and hepatic triacylglycerol accumulation in mice” [J. Funct. Foods 19 (B) (2015) 913–921]

2017 ◽  
Vol 28 ◽  
pp. 328-330
Author(s):  
Ming-Fen Lee ◽  
Ching-Shu Lai ◽  
An-Chin Cheng ◽  
Jain-Sin Hou ◽  
Vladimir Badmaev ◽  
...  
Keyword(s):  
High Fat Diet ◽  
Krill Oil ◽  
High Fat ◽  
Diet Induced Obesity ◽  
Triacylglycerol Accumulation ◽  
Hepatic Triacylglycerol

Krill oil and xanthigen separately inhibit high fat diet induced obesity and hepatic triacylglycerol accumulation in mice

2015 ◽  
Vol 19 ◽  
pp. 913-921 ◽  
Author(s):  
Ming-Fen Lee ◽  
Ching-Shu Lai ◽  
An-Chin Cheng ◽  
Jain-Sin Hou ◽  
Vladimir Badmaev ◽  
...  
Keyword(s):  
High Fat Diet ◽  
Krill Oil ◽  
High Fat ◽  
Diet Induced Obesity ◽  
Triacylglycerol Accumulation ◽  
Hepatic Triacylglycerol

scholarly journals Modulation of the gut microbiota by the mixture of fish oil and krill oil in high-fat diet-induced obesity mice

PLoS ONE ◽  
2017 ◽  
Vol 12 (10) ◽  
pp. e0186216 ◽  
Author(s):  
Chenxi Cui ◽  
Yanyan Li ◽  
Hang Gao ◽  
Hongyan Zhang ◽  
Jiaojiao Han ◽  
...  
Keyword(s):  
Gut Microbiota ◽  
Fish Oil ◽  
High Fat Diet ◽  
Krill Oil ◽  
High Fat ◽  
Diet Induced Obesity

scholarly journals Germ-Free Swiss Webster Mice on a High-Fat Diet Develop Obesity, Hyperglycemia, and Dyslipidemia

2020 ◽  
Vol 8 (4) ◽  
pp. 520
Author(s):  
Isabelle E. Logan ◽  
Gerd Bobe ◽  
Cristobal L. Miranda ◽  
Stephany Vasquez-Perez ◽  
Jaewoo Choi ◽  
...  
Keyword(s):  
High Fat Diet ◽  
Glucose Absorption ◽  
Feed Conversion ◽  
High Fat ◽  
Germ Free ◽  
Low Fat Diet ◽  
Established Risk Factor ◽  
Diet Induced Obesity ◽  
Hepatic Triacylglycerol

A calorie-dense diet is a well-established risk factor for obesity and metabolic syndrome (MetS), whereas the role of the intestinal microbiota (IMB) in the development of diet-induced obesity (DIO) is not completely understood. To test the hypothesis that Swiss Webster (Tac:SW) mice can develop characteristics of DIO and MetS in the absence of the IMB, we fed conventional (CV) and germ-free (GF) male Tac:SW mice either a low-fat diet (LFD; 10% fat derived calories) or a high-fat diet (HFD; 60% fat derived calories) for 10 weeks. The HFD increased feed conversion and body weight in GF mice independent of the increase associated with the microbiota in CV mice. In contrast to CV mice, GF mice did not decrease feed intake on the HFD and possessed heavier fat pads. The HFD caused hyperglycemia, hyperinsulinemia, and impaired glucose absorption in GF mice independent of the increase associated with the microbiota in CV mice. A HFD also elevated plasma LDL-cholesterol and increased hepatic triacylglycerol, free fatty acids, and ceramides in all mice, whereas hypertriglyceridemia and increased hepatic medium and long-chain acylcarnitines were only observed in CV mice. Therefore, GF male Tac:SW mice developed several detrimental effects of obesity and MetS from a high-fat, calorie dense diet.

scholarly journals Effects of DGAT1 deficiency on energy and glucose metabolism are independent of adiponectin

2006 ◽  
Vol 291 (2) ◽  
pp. E388-E394 ◽  
Author(s):  
Ryan S. Streeper ◽  
Suneil K. Koliwad ◽  
Claudio J. Villanueva ◽  
Robert V. Farese
Keyword(s):  
Insulin Sensitivity ◽  
Glucose Metabolism ◽  
High Fat Diet ◽  
Acid Oxidation ◽  
Glucose And Lipid Metabolism ◽  
Diet Induced Obesity ◽  
Triacylglycerol Synthesis ◽  
Triacylglycerol Accumulation ◽  
Hepatic Triacylglycerol ◽  
Deficient Mice

Mice lacking acyl-CoA:diacylglycerol acyltransferase 1 (DGAT1), an enzyme that catalyzes the terminal step in triacylglycerol synthesis, have enhanced insulin sensitivity and are protected from obesity, a result of increased energy expenditure. In these mice, factors derived from white adipose tissue (WAT) contribute to the systemic changes in metabolism. One such factor, adiponectin, increases fatty acid oxidation and enhances insulin sensitivity. To test the hypothesis that adiponectin is required for the altered energy and glucose metabolism in DGAT1-deficient mice, we generated adiponectin-deficient mice and introduced adiponectin deficiency into DGAT1-deficient mice by genetic crosses. Although adiponectin-deficient mice fed a high-fat diet were heavier, exhibited worse glucose tolerance, and had more hepatic triacylglycerol accumulation than wild-type controls, mice lacking both DGAT1 and adiponectin, like DGAT1-deficient mice, were protected from diet-induced obesity, glucose intolerance, and hepatic steatosis. These findings indicate that adiponectin is required for normal energy, glucose, and lipid metabolism but that the metabolic changes induced by DGAT1-deficient WAT are independent of adiponectin and are likely due to other WAT-derived factors. Our findings also suggest that the pharmacological inhibition of DGAT1 may be useful for treating human obesity and insulin resistance associated with low circulating adiponectin levels.

Soyasaponin ameliorates obesity and reduces hepatic triacylglycerol accumulation by suppressing lipogenesis in high‐fat diet‐fed mice

2021 ◽  
Author(s):  
Wei‐Ting Tsai ◽  
Yuki Nakamura ◽  
Taiki Akasaka ◽  
Yoshinori Katakura ◽  
Yasutake Tanaka ◽  
...  
Keyword(s):  
High Fat Diet ◽  
High Fat ◽  
Triacylglycerol Accumulation ◽  
Hepatic Triacylglycerol

scholarly journals Korean Pine Nut Oil Attenuated Hepatic Triacylglycerol Accumulation in High-Fat Diet-Induced Obese Mice

Nutrients ◽  
2016 ◽  
Vol 8 (1) ◽  
pp. 59 ◽  
Author(s):  
Soyoung Park ◽  
Sunhye Shin ◽  
Yeseo Lim ◽  
Jae Shin ◽  
Je Seong ◽  
...  
Keyword(s):  
High Fat Diet ◽  
Korean Pine ◽  
Obese Mice ◽  
High Fat ◽  
Pine Nut ◽  
Pine Nut Oil ◽  
Triacylglycerol Accumulation ◽  
Hepatic Triacylglycerol

Glucagon under high fat diet induced obesity

2007 ◽  
Vol 115 (S 1) ◽  
Author(s):  
LC Bollheimer ◽  
H Wobser ◽  
CE Wrede ◽  
A Schäffler ◽  
J Schölmerich ◽  
...  
Keyword(s):  
High Fat Diet ◽  
High Fat ◽  
Diet Induced Obesity
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