Impact of maternal depressive symptoms and infant temperament on early infant growth and motor development: Results from a population based study in Bangladesh

2013 ◽  
Vol 146 (2) ◽  
pp. 254-261 ◽  
Author(s):  
Hashima-E Nasreen ◽  
Zarina Nahar Kabir ◽  
Yvonne Forsell ◽  
Maigun Edhborg
PLoS ONE ◽  
2015 ◽  
Vol 10 (3) ◽  
pp. e0121459 ◽  
Author(s):  
Rebecca Giallo ◽  
Salma Bahreinian ◽  
Stephanie Brown ◽  
Amanda Cooklin ◽  
Dawn Kingston ◽  
...  

2016 ◽  
Vol 31 (3) ◽  
pp. 346-355 ◽  
Author(s):  
Hanan El Marroun ◽  
Tonya J White ◽  
Guillen Fernandez ◽  
Vincent WV Jaddoe ◽  
Frank C Verhulst ◽  
...  

Selective serotonin reuptake Inhibitors (SSRIs) are frequently used during pregnancy. Evidence about the long-term consequences of prenatal SSRI exposure on child neurodevelopment is controversial. We prospectively investigated whether prenatal SSRI exposure was associated with childhood non-verbal cognition in a population-based study, and contrasted it to exposure to depressive symptoms (without SSRIs). We included 71 children prenatally exposed to SSRIs, 385 children prenatally exposed to maternal depressive symptoms and 5427 unexposed children. Child executive functioning was assessed by maternal report at 4 years ( n=4020). Non-verbal intelligence was measured at 5 years ( n=5001) and children were tested with a neuropsychological battery at 7 years ( n=1194). Prenatal SSRI exposure was not related to maternal reported executive function at 4 years, nor was it related with observed non-verbal intelligence at age 5 or neuropsychological function at 7 years. Exposure to untreated maternal depressive symptoms was related to maternal reported shifting problems and emotional control problems at 4 years. No associations between exposure to depressive symptoms and observed non-verbal IQ at 5 years or neuropsychological function at 7 years were found. This population-based study suggests that neither SSRI use nor untreated depressive symptoms during pregnancy had a major impact on child non-verbal cognition.


2014 ◽  
Vol 205 (2) ◽  
pp. 95-102 ◽  
Author(s):  
Hanan El Marroun ◽  
Tonya J. H. White ◽  
Noortje J. F. van der Knaap ◽  
Judith R. Homberg ◽  
Guillén Fernández ◽  
...  

BackgroundSelective serotonin reuptake inhibitors (SSRIs) are considered safe and are frequently used during pregnancy. However, two case–control studies suggested an association between prenatal SSRI exposure with childhood autism.AimsTo prospectively determine whether intra-uterine SSSRI exposure is associated with childhood autistic symptoms in a population-based study.MethodA total of 376 children prenatally exposed to maternal depressive symptoms (no SSRI exposure), 69 children prenatally exposed to SSRIs and 5531 unexposed children were included. Child pervasive developmental and affective problems were assessed by parental report with the Child Behavior Checklist at ages 1.5, 3 and 6. At age 6, we assessed autistic traits using the Social Responsiveness Scale (n= 4264).ResultsPrenatal exposure to maternal depressive symptoms without SSRIs was related to both pervasive developmental (odds ratio (OR) = 1.44, 95% CI 1.07–1.93) and affective problems (OR = 1.44, 95% CI 1.15–1.81). Compared with unexposed children, those prenatally exposed to SSRIs also were at higher risk for developing pervasive developmental problems (OR = 1.91, 95% CI 1.13–3.47), but not for affective problems. Children prenatally exposed to SSRIs also had more autistic traits (B= 0.15, 95% CI 0.08–0.22) compared with those exposed to depressive symptoms only.ConclusionsOur results suggest an association between prenatal SSRI exposure and autistic traits in children. Prenatal depressive symptoms without SSRI use were also associated with autistic traits, albeit this was weaker and less specific. Long-term drug safety trials are needed before evidence-based recommendations are possible.


2016 ◽  
Vol 26 (7) ◽  
pp. 467-473 ◽  
Author(s):  
Pamela J. Surkan ◽  
Kwame Sakyi ◽  
Donna M. Strobino ◽  
Sucheta Mehra ◽  
Alain Labrique ◽  
...  

2021 ◽  
Vol 279 ◽  
pp. 282-291
Author(s):  
Alexandre Vallée ◽  
Emmanuel Wiernik ◽  
Sofiane Kab ◽  
Cédric Lemogne ◽  
Marcel Goldberg ◽  
...  

Neurology ◽  
1999 ◽  
Vol 53 (9) ◽  
pp. 1998-1998 ◽  
Author(s):  
A- K. Berger ◽  
L. Fratiglioni ◽  
Y. Forsell ◽  
B. Winblad ◽  
L. Backman

2011 ◽  
Vol 15 (6) ◽  
pp. 1087-1092 ◽  
Author(s):  
Alan C Tsai ◽  
Tsui-Lan Chang ◽  
Shu-Hwang Chi

AbstractObjectiveThe study evaluated the association between consumption frequencies of the major food categories and the risk of new depression four years later in older Taiwanese.DesignA prospective cohort study with multistage random sampling. Logistic regression analysis evaluated the significance of the longitudinal associations of intake frequencies of the major food categories with future (4 years later) risk of new depression, controlled for possible confounding factors with or without adjustment for cognitive status.SettingPopulation-based free-living elderly.SubjectsMen and women (n 1609) ≥65 years of age.ResultsIn a regression model that controlled for demographic, socio-economic, lifestyle and disease/health-related variables but not cognitive status, both fruits (OR = 0·66, 95 % CI 0·45, 0·98, P = 0·038) and vegetables (OR = 0·38, 95 % CI 0·17, 0·86, P = 0·021) were protective against depressive symptoms 4 years later. However, when the same regression model was also adjusted for cognitive status, only vegetables (OR = 0·40, 95 % CI 0·17, 0·95, P = 0·039) were protective against depressive symptoms. Higher consumption of eggs was close to being significant in both regression models (P = 0·087 and 0·069, respectively). Other food categories including meat/poultry, fish, seafood, dairy, legumes, grains and tea showed no significant associations.ConclusionsResults suggest that although confounding factors cannot be totally ruled out, more frequent consumption of vegetables seems to be protective against depressive symptoms in the elderly. Further studies are needed to elucidate the causal role and the mechanism of the association.


Sign in / Sign up

Export Citation Format

Share Document