Idiopathic Avascular Necrosis of the Scaphoid: Preiser's Disease

Hand Clinics ◽  
2006 ◽  
Vol 22 (4) ◽  
pp. 475-484 ◽  
Author(s):  
Anthony J. Lauder ◽  
Thomas E. Trumble
2019 ◽  
Vol 5 (1) ◽  
pp. 20
Author(s):  
John McMurtry ◽  
Jessica Frankenhoff

Reports of bilateral Preiser’s disease-avascular necrosis of the scaphoid -- are rare with only 4 cases reported in the English literature. We present the fifth reported case of bilateral Preiser’s disease in the setting of a previously undiagnosed hypercoagulability disorder.  


1994 ◽  
Vol 19 (2) ◽  
pp. 174-182 ◽  
Author(s):  
T. J. HERBERT ◽  
M. LANZETTA

Idiopathic avascular necrosis of the scaphoid is a rare condition. A review of the literature shows a variety of conditions labelled as spontaneous avascular necrosis or Preiser’s disease. In this paper we report on a study of eight patients with idiopathic avascular necrosis affecting only the proximal pole of the scaphoid. Seven of these patients had positive ulnar variance. The possible aetiology is discussed and the natural history has been studied. A staging system is proposed, as this helps to determine the prognosis and appropriate management. Two of our patients were managed conservatively; the others were treated successfully by partial silastic replacement of the scaphoid


2013 ◽  
Vol 54 (7) ◽  
pp. 374-376 ◽  
Author(s):  
M. J. Aiken ◽  
J. E. Stewart ◽  
A. A. Anderson

2013 ◽  
Vol 39 (7) ◽  
pp. 770-776 ◽  
Author(s):  
A. M. Kallen ◽  
S. D. Strackee

In 1910, Georg Preiser (1876–1913) described five cases of rarifying osteitis. Based on his imaging studies, he diagnosed post-traumatic avascular necrosis (AVN) of the scaphoid without any sign of primary fracture. This was followed by an article in 1911 in which Preiser related his findings to Kienböck’s disease and Köhler’s disease of the tarsal navicular. Upon searching the literature, we found descriptions and discussions of Preiser’s imaging; however, the original images have never been published. We reproduce Preiser’s original imaging in this current review. All of these appear to show a fracture and no signs of AVN, suggesting that Georg Preiser misinterpreted his findings. There is no apparent uniformity in the literature regarding the definition, description, or aetiology of Preiser’s disease, and it is for this reason that we find the use of eponyms to be confusing.


1999 ◽  
Vol 41 (2) ◽  
pp. 381
Author(s):  
Hae Woong Jeong ◽  
Jeong Hee Yoon ◽  
Chang Soo Kim

Sign in / Sign up

Export Citation Format

Share Document