California sea lions (Zalophus californianus californianus) have lower chlorinated hydrocarbon contents in northern Baja California, México, than in California, USA

2006 ◽  
Vol 142 (1) ◽  
pp. 83-92 ◽  
Author(s):  
Ligeia Del Toro ◽  
Gisela Heckel ◽  
Víctor F. Camacho-Ibar ◽  
Yolanda Schramm
1983 ◽  
Vol 64 (3) ◽  
pp. 513-518 ◽  
Author(s):  
D. Aurioles ◽  
F. Sinsel ◽  
C. Fox ◽  
E. Alvarado ◽  
O. Maravilla

1975 ◽  
Vol 32 (12) ◽  
pp. 2391-2397 ◽  
Author(s):  
Donald R. Buhler ◽  
Robert R. Claeys ◽  
Bruce R. Mate

Samples of various tissues and organs from healthy California sea lions (Zalophus californianus californianus) and sick animals (apparently with leptospirosis) collected along the central Oregon coast in 1970, 1971, and 1973 were analyzed for total mercury, methylmercury, cadmium, and chlorinated hydrocarbons. Maximum mercury concentrations of 74–170 ppm occurred in sea lion liver, but only 1.6–3.7% of this was present as methylmercury. Cadmium was concentrated primarily in the kidney which contained 7.2–12.0 ppm of the metal. Chlorinated hydrocarbon residues in sea lion fat ranged between 253–475 ppm DDE, and 21.2 and 34.1 ppm PCB. Although mercury, cadmium, and chlorinated hydrocarbon residues in some of the sick sea lions were significantly higher than those present in healthy animals, it is not possible to relate these differences to the onset of leptospirosis.


2005 ◽  
Vol 5 (1) ◽  
pp. 140-142 ◽  
Author(s):  
F. D. HERNANDEZ-VELAZQUEZ ◽  
C. E. GALINDO-SANCHEZ ◽  
M. I. TAYLOR ◽  
J. DE LA ROSA-VELEZ ◽  
I. M. COTE ◽  
...  

2001 ◽  
Vol 79 (6) ◽  
pp. 1080-1087 ◽  
Author(s):  
Anthony J Orr ◽  
James T Harvey

The purpose of this study was to quantify the errors associated with using fecal samples to determine the diet of the California sea lion (Zalophus californianus). Fishes and squids of known size and number were fed to five sea lions held in enclosures with seawater-filled pools. Enclosures were washed and pools were drained periodically so that sea lion feces could be collected using a 0.5 mm mesh bag. Fish otoliths and squid beaks were collected from feces and used to estimate number and size of prey eaten. An average of 50.7% (SE = 6.4%) of 430 fishes and 73.5% (SE = 12.0%) of 49 cephalopods fed to sea lions were represented by otoliths and beaks in feces, respectively. Estimated lengths of fish from feces were less than lengths of fish fed to sea lions by an average of 30.1% (SE = 2.8%). Beaks were not digested significantly; estimated lengths of squid were underestimated by an average of only 3.3% (SE = 1.5%) relative to actual lengths. Passage rates of otoliths varied, but more than 70% were recovered within 48 h after the fish was consumed. Passage rates of beaks were generally less than those of otoliths; six beaks (11%) were collected in feces 4 days after the squid were eaten. Correction factors were created to more reliably estimate the number and size of fishes and cephalopods eaten by California sea lions.


2018 ◽  
Vol 54 (3) ◽  
pp. 581-586 ◽  
Author(s):  
Alissa C. Deming ◽  
Kathleen M. Colegrove ◽  
Padraig J. Duignan ◽  
Ailsa J. Hall ◽  
James F. X. Wellehan ◽  
...  

2011 ◽  
Vol 94 (3) ◽  
pp. 243-248 ◽  
Author(s):  
T Goldstein ◽  
KM Colegrove ◽  
M Hanson ◽  
FMD Gulland

2008 ◽  
Vol 39 (4) ◽  
pp. 542-547 ◽  
Author(s):  
Sophie Dennison ◽  
Martin Haulena ◽  
D. Colette Williams ◽  
John Dawson ◽  
Brian S. Yandell ◽  
...  

2005 ◽  
Vol 42 (2) ◽  
pp. 184-191 ◽  
Author(s):  
P. A. Silvagni ◽  
L. J. Lowenstine ◽  
T. Spraker ◽  
T. P. Lipscomb ◽  
F. M. D. Gulland

Over 100 free-ranging adult California sea lions ( Zalophus californianus) and one Northern fur seal ( Callorhinus ursinus), predominantly adult females, were intoxicated by domoic acid (DA) during three harmful algal blooms between 1998 and 2000 in central and northern California coastal waters. The vector prey item was Northern anchovy ( Engraulis mordax) and the primary DA-producing algal diatom was Psuedonitzschia australis. Postmortem examination revealed gross and histologic findings that were distinctive and aided in diagnosis. A total of 109 sea lions were examined, dying between 1 day and 10 months after admission to a marine mammal rehabilitation center. Persistent seizures with obtundation were the main clinical findings. Frequent gross findings in animals dying acutely consisted of piriform lobe malacia, myocardial pallor, bronchopneumonia, and complications related to pregnancy. Gross findings in animals dying months after intoxication included bilateral hippocampal atrophy. Histologic observations implicated limbic system seizure injury consistent with excitotoxin exposure. Peracutely, there was microvesicular hydropic degeneration within the neuropil of the hippocampus, amygdala, pyriform lobe, and other limbic structures. Acutely, there was ischemic neuronal necrosis, particularly apparent in the granular cells of the dentate gyrus and the pyramidal cells within the hippocampus cornu ammonis (CA) sectors CA4, CA3, and CA1. Dentate granular cell necrosis has not been reported in human or experimental animal DA toxicity and may be unique to sea lions. Chronically, there was gliosis, mild nonsuppurative inflammation, and loss of laminar organization in affected areas.


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