Protective effects of gabion wall against blast waves from large TNT-equivalent explosions

2021 ◽  
Vol 249 ◽  
pp. 113389
Author(s):  
Rongzheng Xu ◽  
Li Chen ◽  
Qin Fang ◽  
Yuzhou Zheng ◽  
Zhan Li ◽  
...  
2013 ◽  
Vol 850-851 ◽  
pp. 368-372
Author(s):  
Zhi Zhong Li ◽  
De Gao Tang ◽  
Zheng Liu ◽  
Yu Long Xue ◽  
Wei Wei Li

For the comparative analysis of composite blast walls on the protective effects of blast wave, Overpressure protective effectiveness coefficient was put forward to analyze protective effectiveness and . It was studied that Protective effectiveness of composite blast walls under blast waves using 3D numerical simulation method and analysis of the amount of drugs, explosive distance, height and other structural parameters on protective effect of composite structures. The influence of several factors on forecasting formula was analyzed, such as scaled blast distance and scaled height of wall. Via fitting the simulation results overpressure formula of the back of composite blast walls was obtained. The result indicate that protective effectiveness of composite structure increases when walls height increases or scaled blast distance decreases.


Author(s):  
Paulina Iwan ◽  
Jan Stepniak ◽  
Malgorzata Karbownik-Lewinska

Abstract. Iodine is essential for thyroid hormone synthesis. Under normal iodine supply, calculated physiological iodine concentration in the thyroid is approx. 9 mM. Either potassium iodide (KI) or potassium iodate (KIO3) are used in iodine prophylaxis. KI is confirmed as absolutely safe. KIO3 possesses chemical properties suggesting its potential toxicity. Melatonin (N-acetyl-5-methoxytryptamine) is an effective antioxidant and free radical scavenger. Study aims: to evaluate potential protective effects of melatonin against oxidative damage to membrane lipids (lipid peroxidation, LPO) induced by KI or KIO3 in porcine thyroid. Homogenates of twenty four (24) thyroids were incubated in presence of either KI or KIO3 without/with melatonin (5 mM). As melatonin was not effective against KI-induced LPO, in the next step only KIO3 was used. Homogenates were incubated in presence of KIO3 (200; 100; 50; 25; 20; 15; 10; 7.5; 5.0; 2.5; 1.25 mM) without/with melatonin or 17ß-estradiol. Five experiments were performed with different concentrations of melatonin (5.0; 2.5; 1.25; 1.0; 0.625 mM) and one with 17ß-estradiol (1.0 mM). Malondialdehyde + 4-hydroxyalkenals (MDA + 4-HDA) concentration (LPO index) was measured spectrophotometrically. KIO3 increased LPO with the strongest damaging effect (MDA + 4-HDA level: ≈1.28 nmol/mg protein, p < 0.05) revealed at concentrations of around 15 mM, thus corresponding to physiological iodine concentrations in the thyroid. Melatonin reduced LPO (MDA + 4-HDA levels: from ≈0.97 to ≈0,76 and from ≈0,64 to ≈0,49 nmol/mg protein, p < 0.05) induced by KIO3 at concentrations of 10 mM or 7.5 mM. Conclusion: Melatonin can reduce very strong oxidative damage to membrane lipids caused by KIO3 used in doses resulting in physiological iodine concentrations in the thyroid.


2009 ◽  
Author(s):  
Phillip A. Ianni ◽  
Kenneth E. Hart ◽  
Stephen Hibbard ◽  
Michelle Carroll ◽  
Tobi Wilson ◽  
...  

2009 ◽  
Author(s):  
Pauline Garcia-Reid ◽  
Christina Hamme Peterson ◽  
Robert James Reid ◽  
Paul W. Speer ◽  
N. Andrew Peterson

2001 ◽  
Vol 11 (4) ◽  
pp. 183-193 ◽  
Author(s):  
Tedine Ranich ◽  
Sam J. Bhathena ◽  
Manuel T. Velasquez

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