scholarly journals Extensive subcutaneous fat necrosis after selective head cooling in a newborn with hypoxic ischemic encephalopathy

2016 ◽  
Vol 34 (4) ◽  
pp. 211-212
Author(s):  
Kai-Lung Chen ◽  
Chien-Yi Chen ◽  
Hsien-Ching Chiu
2014 ◽  
Vol 34 (6) ◽  
pp. 492-493 ◽  
Author(s):  
E Pérez Martínez ◽  
M Camprubí Camprubí ◽  
M Ramos Cebrián ◽  
J Antón López ◽  
A Apodaca Saracho ◽  
...  

2013 ◽  
Vol 55 (2) ◽  
pp. e23-e24 ◽  
Author(s):  
Toru Kuboi ◽  
Takashi Kusaka ◽  
Kaoru Okazaki ◽  
Uiko Kaku ◽  
Ryota Kakinuma ◽  
...  

2014 ◽  
Vol 38 (2) ◽  
pp. 104-108
Author(s):  
AFMM Rahman ◽  
AA Ameri ◽  
HSA Shehri ◽  
MAA Mridha

Abstract not available DOI: http://dx.doi.org/10.3329/bjch.v38i2.21146 Bangladesh J Child Health 2014; VOL 38 (2) : 101-103


Author(s):  
L. VERBEKE ◽  
K. KAMOEN ◽  
L. AEYELS ◽  
J. VERAMME ◽  
N. DE CONO

Subcutaneous fat necrosis of the newborn with follow-up of the hypercalcemia A thirteen-day-old boy is diagnosed with subcutaneous fat necrosis of the newborn after the appearance of erythematous, subcutaneous plaques on the right shoulder. This rare, usually self-limiting cutaneous disease is mainly diagnosed in term and postterm infants. The diagnosis is confirmed by a skin biopsy. Various etiological hypotheses are suggested, although the exact pathogenesis needs to be elucidated. The disease might be associated with several maternal and foetal predisposing factors, including therapeutic hypothermia at birth as part of hypoxic ischemic encephalopathy. It is a benign inflammation of the subcutaneous adipocytes that can be accompanied by life-threatening complications, such as hypercalcemia and thrombocytopenia. The latter requires rigorous follow-up and, in some cases, also treatment. The skin damage should only be treated symptomatically with analgesia.


1982 ◽  
Vol 48 (03) ◽  
pp. 245-246 ◽  
Author(s):  
V Hofmann ◽  
P G Frick

SummaryA female patient is described who developed skin and subcutaneous fat necrosis on two occasions after intake of acenocoumarol.Several months later identical skin changes occurred during an episode of cholestasis associated with a prolongation of the prothrombin time to an extent comparable with therapeutic anticoagulation; intake of oral anticoagulants could be excluded.This association gives new insights in the pathogenetic mechanisms responsible for the so-called coumarin necrosis and indicates that it may be not due to drug toxicity or allergy.


2007 ◽  
Vol 24 (1) ◽  
pp. 93-93 ◽  
Author(s):  
LARISSA L. ZAULYANOV ◽  
SHARON E. JACOB ◽  
GEORGE W. ELGART ◽  
LAWRENCE SCHACHNER

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