Reply to the Comments of P. Tothill on “DXA in vivo BMD methodology: An erroneous and misleading research and clinical gauge of bone mineral status, bone fragility, and bone remodelling” by H. H. Bolotin (Bone 2007;41:138–154)

Bone ◽  
2008 ◽  
Vol 42 (1) ◽  
pp. 239-241
Author(s):  
H.H. Bolotin
Keyword(s):  
Bone Mineral ◽  
Bone Remodelling ◽  
Bone Fragility ◽  
Mineral Status ◽  
Bone Mineral Status

Effects of vanadium (V) and magnesium (Mg) on rat bone tissue: mineral status and micromorphology. Consequences of V–Mg interactions

Metallomics ◽  
2014 ◽  
Vol 6 (12) ◽  
pp. 2260-2278 ◽  
Author(s):  
Agnieszka Ścibior ◽  
Agnieszka Adamczyk ◽  
Robert Mroczka ◽  
Irmina Niedźwiecka ◽  
Dorota Gołębiowska ◽  
...  
Keyword(s):  
Bone Tissue ◽  
Experimental Model ◽  
Bone Mineral ◽  
Mineral Status ◽  
Bone Mineral Status ◽  
Rat Bone

Effects of vanadium and magnesium on bone mineral status and micromorphology were shown in anin vivoexperimental model.

Bone mineral status in children with turner syndrome. The effects of human growth hormone (hGH)

1992 ◽  
Vol 17 ◽  
pp. 227
Author(s):  
I. Voskaki-Voulgari ◽  
A. Al Qadtreh ◽  
N. Georgopoulos ◽  
G. Xecalou ◽  
C. Dakou-Voutetaki
Keyword(s):  
Growth Hormone ◽  
Turner Syndrome ◽  
Bone Mineral ◽  
Human Growth Hormone ◽  
Human Growth ◽  
Mineral Status ◽  
Bone Mineral Status

Bone mineral status of women with Marfan syndrome

1993 ◽  
Vol 95 (6) ◽  
pp. 568-572 ◽  
Author(s):  
Lynn Kohlmeier ◽  
Cheryll Gasner ◽  
Robert Marcus
Keyword(s):  
Marfan Syndrome ◽  
Bone Mineral ◽  
Mineral Status ◽  
Bone Mineral Status ◽  
Status Of Women

Effect of nitric oxide synthase inhibitors on bone metabolism in growing rats

1996 ◽  
Vol 270 (5) ◽  
pp. E840-E845 ◽  
Author(s):  
H. Tsukahara ◽  
M. Miura ◽  
S. Tsuchida ◽  
I. Hata ◽  
K. Hata ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Bone Mass ◽  
Bone Mineral ◽  
Normal Serum ◽  
Bone Mineral Status ◽  
Growing Rats ◽  
Bone Mineral Mass ◽  
Bone Degradation ◽  
Cross Links

We examined the effects of chronic nitric oxide (NO) blockade on bone mineral status in growing rats. Oral administration of NG-nitro-L-arginine methyl ester (L-NAME) for 4 wk caused hypertension and a significant reduction in urinary NO2- and NO3- excretion. Four-week oral aminoguanidine (AG, 400 mg/dl of drinking water) did not alter blood pressure but caused a significant decrease in urinary NO2- and NO3-. Rats treated with L-NAME at doses of 20 and 50 mg/dl had normal bone mineral mass in the lumbar spine, but the highest dose (80 mg/dl) caused a slight decrease in bone mass. Chronic AG induced a significant spine osteopenia. This effect of AG was abolished by the simultaneous administration of L-arginine (2.0 g/dl). AG-induced osteopenia was associated with a significant increase in urine excretion of collagen cross-links with normal serum osteocalcin. These findings indicate that chronic AG administration can cause an imbalance between bone resorption and formation, resulting in a decrease in bone mass in growing rats, and suggest that NO produced by inducible NO synthase plays an important role in basal osteoclast bone degradation activity in vivo.

Dual-energy X-ray absorptiometry studies of bone mineral status in newborn infants

2009 ◽  
Vol 11 (7) ◽  
pp. 997-1002 ◽  
Author(s):  
Winston W. K. Koo ◽  
Jocelyn Walters ◽  
Andrew J. Bush ◽  
Russell W. Chesney ◽  
Susan E. Carlson
Keyword(s):  
Bone Mineral ◽  
Newborn Infants ◽  
Dual Energy ◽  
Mineral Status ◽  
X Ray ◽  
Bone Mineral Status
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