scholarly journals Electrophysiological mapping of rat sensorimotor lumbosacral spinal networks after complete paralysis☆

2015 ◽  
pp. 199-212 ◽  
Author(s):  
Parag Gad ◽  
Roland R. Roy ◽  
Jaehoon Choe ◽  
Hui Zhong ◽  
Mandheeraj Singh Nandra ◽  
...  
Keyword(s):  
Complete Paralysis

The Therapeutic Action of Hyoscyamine

1876 ◽  
Vol 22 (99) ◽  
pp. 434-439
Keyword(s):  
Therapeutic Action ◽  
The West ◽  
Complete Paralysis

Dr. Lawson, of the West Riding Asylum, contributes to the “Practitioner” of July last his experience of the action of this drug. In sufficient quantities it produces a subdued form of mania accompanied by almost complete paralysis of the voluntary muscles, and ending in quiet and refreshing sleep. Having regard to these effects, he thought it probable that it might be useful in certain forms of violent sthenic mania, which, notwithstanding the virtues of ergot of rye, do appear to be met with in the West Riding Asylum, and be useful in this way—by substituting for the extreme forms of excitement a quieter form of mania, which, on disappearing in its turn, might leave the patient quiet. Experience seems to have established the correctness of this surmise.

Acute Otitis Media and Facial Paralysis in Children

1996 ◽  
Vol 105 (1) ◽  
pp. 58-62 ◽  
Author(s):  
Clark A. Elliott ◽  
George H. Zalzal ◽  
Wendy R. Gottlieb
Keyword(s):  
Facial Nerve ◽  
Otitis Media ◽  
Facial Paralysis ◽  
Acute Otitis Media ◽  
Complete Recovery ◽  
Rapid Improvement ◽  
Nerve Decompression ◽  
Complete Paralysis ◽  
Prolonged Recovery ◽  
Facial Nerve Decompression

We reviewed 10 children who presented with facial paralysis after the onset of acute otitis media. The objective of the study was to examine the outcome of facial paralysis in children with acute otitis media treated without facial nerve decompression. Two groups were identified: 8 patients with incomplete paralysis and 2 with complete paralysis. Seven of the 8 patients with incomplete paralysis had rapid return of function after myringotomy and intravenous antibiotics. The eighth patient had delayed recovery requiring 9 months before complete return of function. The 2 patients with complete paralysis required mastoidectomy to control otorrhea and fever after initial myringotomy and antibiotics. Both patients had a prolonged recovery requiring 3 and 7 months for complete recovery. Patients with incomplete paralysis generally show rapid improvement following wide myringotomy and antibiotic treatment. A more protracted recovery may be expected in patients with complete paralysis; excellent return of function is expected when mastoidectomy without facial nerve decompression is employed.

scholarly journals Botulism with Unusual Rapid Progression to Complete Paralysis in a Child

2015 ◽  
Vol 56 (6) ◽  
pp. 425-428 ◽  
Author(s):  
Hui-Ju Tsai ◽  
Wen-Chen Liang ◽  
Chien-Hua Wang ◽  
Po-Ching Chou ◽  
Jong-Hau Hsu ◽  
...  
Keyword(s):  
Rapid Progression ◽  
Complete Paralysis

scholarly journals Primary spinal epidural non-Hodgkin’s lymphoma causing complete paralysis

QJM ◽  
2018 ◽  
Vol 111 (11) ◽  
pp. 813-814
Author(s):  
S -Z Liu ◽  
X Zhou ◽  
A Song ◽  
Z Huo ◽  
Y -P Wang ◽  
...  
Keyword(s):  
Hodgkin’S Lymphoma ◽  
Hodgkin's Lymphoma ◽  
Non Hodgkin’S Lymphoma ◽  
Non Hodgkin's Lymphoma ◽  
Complete Paralysis ◽  
Spinal Epidural

Common problems and symptoms

2020 ◽  
pp. 151-174
Keyword(s):  
Nervous System ◽  
Muscle Weakness ◽  
Cognitive Abilities ◽  
Management Strategies ◽  
Disease Process ◽  
Signs And Symptoms ◽  
Complete Paralysis ◽  
Common Problems ◽  
The Brain ◽  
Neurological System

Any damage to the neurological system due to a disease process, infection, or injury frequently results in certain abnormal signs and symptoms. Depending on the degree of damage and the region of the brain or peripheral nervous system affected, neurological disorders can result in partial or complete paralysis, muscle weakness, pain and spasticity, seizures, and abnormal cognitive abilities. This chapter present some of the more commonly occurring problems with some management strategies.

scholarly journals Altering spinal cord excitability enables voluntary movements after chronic complete paralysis in humans

Brain ◽  
2014 ◽  
Vol 137 (5) ◽  
pp. 1394-1409 ◽  
Author(s):  
Claudia A. Angeli ◽  
V. Reggie Edgerton ◽  
Yury P. Gerasimenko ◽  
Susan J. Harkema
Keyword(s):  
Spinal Cord ◽  
Voluntary Movements ◽  
Complete Paralysis

scholarly journals Electrophysiological biomarkers of neuromodulatory strategies to recover motor function after spinal cord injury

2015 ◽  
Vol 113 (9) ◽  
pp. 3386-3396 ◽  
Author(s):  
Parag Gad ◽  
Roland R. Roy ◽  
Jaehoon Choe ◽  
Jack Creagmile ◽  
Hui Zhong ◽  
...  
Keyword(s):  
Spinal Cord ◽  
Motor Control ◽  
Evoked Potentials ◽  
Motor Performance ◽  
Weight Bearing ◽  
Chronic Administration ◽  
Pharmacological Agents ◽  
Extensor Muscles ◽  
Cord Injury ◽  
Complete Paralysis

The spinal cord contains the circuitry to control posture and locomotion after complete paralysis, and this circuitry can be enabled with epidural stimulation [electrical enabling motor control (eEmc)] and/or administration of pharmacological agents [pharmacological enabling motor control (fEmc)] when combined with motor training. We hypothesized that the characteristics of the spinally evoked potentials after chronic administration of both strychnine and quipazine under the influence of eEmc during standing and stepping can be used as biomarkers to predict successful motor performance. To test this hypothesis we trained rats to step bipedally for 7 wk after paralysis and characterized the motor potentials evoked in the soleus and tibialis anterior (TA) muscles with the rats in a non-weight-bearing position, standing and stepping. The middle responses (MRs) to spinally evoked stimuli were suppressed with either or both drugs when the rat was suspended, whereas the addition of either or both drugs resulted in an overall activation of the extensor muscles during stepping and/or standing and reduced the drag duration and cocontraction between the TA and soleus muscles during stepping. The administration of quipazine and strychnine in concert with eEmc and step training after injury resulted in larger-amplitude evoked potentials [MRs and late responses (LRs)] in flexors and extensors, with the LRs consisting of a more normal bursting pattern, i.e., randomly generated action potentials within the bursts. This pattern was linked to more successful standing and stepping. Thus it appears that selected features of the patterns of potentials evoked in specific muscles with stimulation can serve as effective biomarkers and predictors of motor performance.

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