Microscopic polyarteritis

2011 ◽  
Keyword(s):  
Microscopic Polyarteritis

scholarly journals AN AUTOPSY CASE OF MICROSCOPIC POLYARTERITIS NODOSA

1980 ◽  
Vol 69 (6) ◽  
pp. 738-744
Author(s):  
Hidemitsu MATSUURA ◽  
Takayoshi TANABE ◽  
Hiroyuki OHMORI ◽  
Tsuneo NANBA ◽  
Katsuhiko KIMOTO ◽  
...  
Keyword(s):  
Polyarteritis Nodosa ◽  
Autopsy Case ◽  
Microscopic Polyarteritis

scholarly journals A case of microscopic polyarteritis associated with recurrent pulmonary hemorrhage.

1991 ◽  
Vol 38 (4) ◽  
pp. 389-395
Author(s):  
Ji Youn Bae ◽  
Sang Soon Lim ◽  
Yoon Suk Lee ◽  
Kwang Ho In ◽  
Se Hwa Yoo ◽  
...  
Keyword(s):  
Pulmonary Hemorrhage ◽  
Microscopic Polyarteritis

Microscopic Polyarteritis

1992 ◽  
Vol 128 (9) ◽  
pp. 1223 ◽  
Author(s):  
Pamela Bostic Homas
Keyword(s):  
Microscopic Polyarteritis

Improved Prognosis of Renal Microscopic Polyarteritis in Recent Years

1988 ◽  
Vol 3 (4) ◽  
pp. 383-391 ◽  
Author(s):  
G. Fuiano ◽  
J. S. Cameron ◽  
M. Raftery ◽  
B. H. Hartley ◽  
D. G. Williams ◽  
...  
Keyword(s):  
Microscopic Polyarteritis

Clinical Outcome and Follow-Up of Patients with Renal Damage in Microscopic Polyarteritis

2015 ◽  
pp. 166-176
Author(s):  
Nicoletta Saporiti ◽  
Mietta Meroni ◽  
Adalberto Sessa ◽  
Angela Volpi ◽  
Ferruccio Conte ◽  
...  
Keyword(s):  
Clinical Outcome ◽  
Renal Damage ◽  
Microscopic Polyarteritis

α1-Antitrypsin Phenotypes in Patients with Anti-Neutrophil Cytoplasmic Antibody-Positive Vasculitis

1994 ◽  
Vol 87 (6) ◽  
pp. 693-695 ◽  
Author(s):  
Karl Lhotta ◽  
Wolfgang Vogel ◽  
Thomas Meisl ◽  
Martina Buxbaum ◽  
Ulrich Neyer ◽  
...  
Keyword(s):  
Systemic Vasculitis ◽  
Clinical Signs ◽  
Rapidly Progressive Glomerulonephritis ◽  
Control Group ◽  
Severe Deficiency ◽  
High Prevalence ◽  
Antitrypsin Deficiency ◽  
Hardy Weinberg Equilibrium ◽  
Microscopic Polyarteritis ◽  
A Minor

1. The genetic background of anti-neutrophil cytoplasmic antibody (ANCA)-associated systemic vasculitis remains largely unknown. Recently a very high prevalence of medium and severe deficiency of α1-antitrypsin was described in a small group of patients with Wegener's granulomatosis and c-ANCA. c-ANCAs are autoantibodies against proteinase 3, and α1-antitrypsin is the main inhibitor of this enzyme. 2. α1-Antitrypsin phenotypic polymorphism was determined by isoelectric focusing in 32 patients with c-ANCA-associated systemic vasculitis. Twenty-nine patients had Wegener's disease, two had microscopic polyarteritis and one suffered from idiopathic rapidly progressive glomerulonephritis. 3. Two patients were homozygous PiZZ and three were heterozygous PiMZ. These phenotype frequencies differed significantly from expected values, assuming Hardy-Weinberg equilibrium (P < 0.01). Compared with a control group of 868 healthy blood donors, these results meant a significant increase in the PiZ allele (0.0138 versus 0.1094, P< 0.001). 4. Furthermore, the serum of 47 patients with severe α1-antitrypsin deficiency (PiZZ) was tested for the presence of ANCA. All sera were negative for c-ANCA and p-ANCA. None of the patients showed clinical signs of systemic vasculitis. 5. In conclusion, these data indicate that α1-antitrypsin deficiency, despite being significantly more common in patients with c-ANCA-associated systemic vasculitis, is only a minor genetic risk factor for the development of this disease.

Microscopic polyarteritis with renal and cerebral involvement

2000 ◽  
Vol 15 (1-2) ◽  
pp. 134-135 ◽  
Author(s):  
P. V. Deshpande ◽  
R. Gilbert ◽  
H. Alton ◽  
D. V. Milford
Keyword(s):  
Cerebral Involvement ◽  
Microscopic Polyarteritis
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