Anticollagen antibodies

2011 ◽  
Keyword(s):  
Anticollagen Antibodies

scholarly journals Interactions Between Anticollagen Antibodies and Chondrocytes

1992 ◽  
Vol 35 (2) ◽  
pp. 224-230 ◽  
Author(s):  
Toshitaka Takagi ◽  
Hugo E. Jasin
Keyword(s):  
Anticollagen Antibodies

Anticollagen antibodies in patients with juvenile chronic arthritis

1988 ◽  
Vol 7 (3) ◽  
pp. 378-383 ◽  
Author(s):  
J. Svobodová ◽  
R. Bečvář ◽  
J. Novotná ◽  
S. Havelka ◽  
M. Adam
Keyword(s):  
Juvenile Chronic Arthritis ◽  
Chronic Arthritis ◽  
Anticollagen Antibodies

scholarly journals Anticollagen Antibodies in Sera of Patients with Periodontal Disease

1979 ◽  
Vol 21 (4) ◽  
pp. 445-454
Author(s):  
Shinichi ANAMURA ◽  
Takeshi UCHIDA ◽  
Tadashi SHIRANE ◽  
Midori KUBOTA ◽  
Keiji NAKANISHI ◽  
...  
Keyword(s):  
Periodontal Disease ◽  
Anticollagen Antibodies

Demonstration of anticollagen antibodies in rheumatoid arthritis synovial fluids by14c-radioimmunoassay

1978 ◽  
Vol 21 (2) ◽  
pp. 243-248 ◽  
Author(s):  
Johannes Menzel ◽  
Carl Steffen ◽  
Gernot Kolarz ◽  
Marina Kojer ◽  
Josef Smolen
Keyword(s):  
Rheumatoid Arthritis ◽  
Synovial Fluids ◽  
Anticollagen Antibodies

Collagen Types and Anticollagen-Antibodies in Dupuytren's Disease

HAND ◽  
1979 ◽  
Vol os-11 (3) ◽  
pp. 243-248 ◽  
Author(s):  
E. J. Menzel ◽  
H. Piza ◽  
C. Zielinski ◽  
A. T. Endler ◽  
C. Steffen ◽  
...  
Keyword(s):  
Relative Proportion ◽  
Collagen Type I ◽  
Dupuytren’S Disease ◽  
Type Iii Collagen ◽  
Type I ◽  
Dupuytren's Disease ◽  
Type Iii ◽  
Low Concentrations ◽  
Collagen Antibodies ◽  
Anticollagen Antibodies

The relative proportion of collagen type I and type III in the aponeurosis of twenty-four patients with Dupuytren's disease was determined and compared with the aponeurosis of normal persons. The presence of considerable amounts of type III collagen was found in the Dupuytren's disease patients. The sera of the patients were screened for circulating anti-collagen antibodies using a sensitive radioimmunoassay. In seven out of the twenty-four patients low concentrations of these antibodies were found.

scholarly journals Induction and Suppression of Collagen-Induced Arthritis Is Dependent on Distinct Fcγ Receptors

2000 ◽  
Vol 191 (9) ◽  
pp. 1611-1616 ◽  
Author(s):  
Sandra Kleinau ◽  
Pernilla Martinsson ◽  
Birgitta Heyman
Keyword(s):  
Cell Activation ◽  
Negative Regulator ◽  
Human Rheumatoid Arthritis ◽  
Collagen Induced Arthritis ◽  
Effector Functions ◽  
Ig G ◽  
Anticollagen Antibodies ◽  
Deficient Mice

Receptors for immunoglobulin (Ig)G (FcγRs) are important for the antibody-mediated effector functions of the immune system. FcγRI and FcγRIII trigger cell activation through a common γ chain, whereas FcγRII acts as a negative regulator of antibody production and immune complex–triggered activation. Here we describe the in vivo consequences of FcγR deficiency in a mouse model of human rheumatoid arthritis. FcRγ chain–deficient mice on arthritis-susceptible DBA/1 background were immunized with collagen for induction of collagen-induced arthritis. The DBA/1 mice lacking FcRγ chain were protected from collagen-induced arthritis in contrast to wild-type mice, although both groups produced similar levels of IgG anticollagen antibodies. In comparison, DBA/1 mice lacking FcγRII developed an augmented IgG anticollagen response and arthritis. These observations suggest a crucial role of FcγRI and FcγRIII in triggering autoimmune arthritis.

scholarly journals Serum transfer of collagen-induced arthritis in mice.

1983 ◽  
Vol 158 (2) ◽  
pp. 378-392 ◽  
Author(s):  
J M Stuart ◽  
F J Dixon
Keyword(s):  
Articular Surface ◽  
Type Ii Collagen ◽  
Host Factors ◽  
Early Disease ◽  
Collagen Induced Arthritis ◽  
Serum Transfer ◽  
Immunofluorescence Studies ◽  
Resistant Strains ◽  
Arthritic Joints ◽  
Anticollagen Antibodies

Immunization of DBA/1 mice with native chick type II collagen resulted in development of polyarthritis 4-5 wk later. Sera of these mice contained high levels of anticollagen antibodies, and immunoglobulin concentrates of their sera transferred arthritis to unimmunized recipients. Histopathologically, this passively transferred arthritis resembled the early disease of immunized donors. Immunofluorescence studies demonstrated the deposition of IgG and C3 on the articular surface but not in synovial tissue of arthritic joints. Transferred, isotopically labeled anticollagen antibodies rapidly localized to the limbs and to other cartilage-containing tissues. When transfer concentrate was administered to arthritis-resistant strains, they also developed arthritis. Indeed, immunoglobulin concentrates from rats with collagen-induced arthritis transferred arthritis to naive mice. The amount of concentrate required for transfer to B10.D2 resistant mice was reduced by immunizing them with collagen 4 wk before transfer. Although susceptibility to arthritis from immunization is H-2 linked, these studies clearly demonstrate that passive transfer of arthritis depends upon injection of specific antibody and not on other host factors.

Anticollagen Antibodies Following Thermal Trauma

1969 ◽  
Vol 130 (2) ◽  
pp. 394-398 ◽  
Author(s):  
J. P. Bray ◽  
F. Estess ◽  
J. A. Bass
Keyword(s):  
Thermal Trauma ◽  
Anticollagen Antibodies
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