Fire-mediated effects of shrubs, lichens and herbs on the demography of Hypericum cumulicola in patchy Florida scrub

Oecologia ◽  
1997 ◽  
Vol 112 (2) ◽  
pp. 263-271 ◽  
Author(s):  
Pedro F. Quintana-Ascencio ◽  
Marina Morales-Hernández
1998 ◽  
Vol 86 (4) ◽  
pp. 640-651 ◽  
Author(s):  
Pedro F. Quintana-Ascencio ◽  
Rebecca W. Dolan ◽  
Eric S. Menges

2005 ◽  
Vol 132 (2) ◽  
pp. 204-213 ◽  
Author(s):  
Matthew D. Trager ◽  
Eric S. Menges ◽  
Pedro F. Quintana-Ascencio ◽  
Carl W. Weekley

2010 ◽  
Vol 2010 ◽  
pp. 1-12 ◽  
Author(s):  
Eric S. Menges ◽  
Rebecca W. Dolan ◽  
Roberta Pickert ◽  
Rebecca Yahr ◽  
Doria R. Gordon

If genetic variation is often positively correlated with population sizes and the presence of nearby populations and suitable habitats, landscape proxies could inform conservation decisions without genetic analyses. For six Florida scrub endemic plants (Dicerandra frutescens, Eryngium cuneifolium, Hypericum cumulicola, Liatris ohlingerae, Nolina brittoniana, and Warea carteri), we relate two measures of genetic variation, expected heterozygosity and alleles per polymorphic locus (APL), to population size and landscape variables. Presettlement areas were estimated based on soil preferences and GIS soils maps. Four species showed no genetic patterns related to population or landscape factors. The other two species showed significant but inconsistent patterns. ForLiatris ohlingerae, APL was negatively related to population density and weakly, positively related to remaining presettlement habitat within 32 km. ForNolina brittoniana, APL increased with population size. The rather weak effects of population area/size and both past and current landscape structures suggest that genetic variation needs to be directly measured and not inferred for conservation planning.


1984 ◽  
Vol 51 (01) ◽  
pp. 089-092 ◽  
Author(s):  
M A Boogaerts ◽  
J Van de Broeck ◽  
H Deckmyn ◽  
C Roelant ◽  
J Vermylen ◽  
...  

SummaryThe effect of alfa-tocopherol on the cell-cell interactions at the vessel wall were studied, using an in vitro model of human umbilical vein endothelial cell cultures (HUEC). Immune triggered granulocytes (PMN) will adhere to and damage HUEC and platelets enhance this PMN mediated endothelial injury. When HUEC are cultured in the presence of vitamin E, 51Cr-leakage induced by complement stimulated PMN is significantly decreased and the enhanced cytotoxicity by platelets is completely abolished (p <0.001).The inhibition of PMN induced endothelial injury is directly correlated to a diminished adherence of PMN to vitamin E- cultured HUEC (p <0.001), which may be mediated by an increase of both basal and stimulated endogenous prostacyclin (PGI2) from alfa-tocopherol-treated HUEC (p <0.025). The vitamin E-effect is abolished by incubation of HUEC with the irreversible cyclo-oxygenase inhibitor, acetylsalicylic acid, but the addition of exogenous PGI2 could not reproduce the vitamin E-mediated effects.We conclude that vitamin E exerts a protective effect on immune triggered endothelial damage, partly by increasing the endogenous anti-oxidant potential, partly by modulating intrinsic endothelial prostaglandin production. The failure to reproduce vitamin E-protection by exogenously added PGI2 may suggest additional, not yet elucidated vitamin E-effects on endothelial metabolism.


2006 ◽  
Vol 114 (S 1) ◽  
Author(s):  
N Perwitz ◽  
B Meier ◽  
M Drenckhan ◽  
M Fasshauer ◽  
J Klein

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