Cellular localization of neuropeptide Y mRNA and peptide in the brain of the Japanese quail and domestic chicken

1998 ◽  
Vol 293 (1) ◽  
pp. 31-38 ◽  
Author(s):  
T. Boswell ◽  
J. R. Millam ◽  
Q. Li ◽  
I. C. Dunn
Keyword(s):  
Neuropeptide Y ◽  
Japanese Quail ◽  
Cellular Localization ◽  
Domestic Chicken ◽  
The Brain

Localization of neuropeptide Y-immunoreactive cells and fibres in the brain of the Japanese quail

1991 ◽  
Vol 265 (2) ◽  
pp. 219-230 ◽  
Author(s):  
N. Aste ◽  
C. Viglietti-Panzica ◽  
A. Fasolo ◽  
C. Andreone ◽  
H. Vaudry ◽  
...  
Keyword(s):  
Neuropeptide Y ◽  
Japanese Quail ◽  
The Brain

scholarly journals The Role of Neuropeptide Y in the Nucleus Accumbens

2021 ◽  
Vol 22 (14) ◽  
pp. 7287
Author(s):  
Masaki Tanaka ◽  
Shunji Yamada ◽  
Yoshihisa Watanabe
Keyword(s):  
Nervous System ◽  
Nucleus Accumbens ◽  
Neuropeptide Y ◽  
Emotional Behavior ◽  
Characteristic Functions ◽  
Receptor Subtypes ◽  
Neuroendocrine Mechanisms ◽  
Fear And Anxiety ◽  
The Brain

Neuropeptide Y (NPY), an abundant peptide in the central nervous system, is expressed in neurons of various regions throughout the brain. The physiological and behavioral effects of NPY are mainly mediated through Y1, Y2, and Y5 receptor subtypes, which are expressed in regions regulating food intake, fear and anxiety, learning and memory, depression, and posttraumatic stress. In particular, the nucleus accumbens (NAc) has one of the highest NPY concentrations in the brain. In this review, we summarize the role of NPY in the NAc. NPY is expressed principally in medium-sized aspiny neurons, and numerous NPY immunoreactive fibers are observed in the NAc. Alterations in NPY expression under certain conditions through intra-NAc injections of NPY or receptor agonists/antagonists revealed NPY to be involved in the characteristic functions of the NAc, such as alcohol intake and drug addiction. In addition, control of mesolimbic dopaminergic release via NPY receptors may take part in these functions. NPY in the NAc also participates in fat intake and emotional behavior. Accumbal NPY neurons and fibers may exert physiological and pathophysiological actions partly through neuroendocrine mechanisms and the autonomic nervous system.

Multifaces of neuropeptide Y in the brain – Neuroprotection, neurogenesis and neuroinflammation

Neuropeptides ◽  
2012 ◽  
Vol 46 (6) ◽  
pp. 299-308 ◽  
Author(s):  
J.O. Malva ◽  
S. Xapelli ◽  
S. Baptista ◽  
J. Valero ◽  
F. Agasse ◽  
...  
Keyword(s):  
Neuropeptide Y ◽  
The Brain

Neuropeptide Y Receptors: Autoradiographic Distribution in the Brain and Structure-Activity Relationships

1990 ◽  
Vol 611 (1 Central and P) ◽  
pp. 58-72 ◽  
Author(s):  
RÉMI QUIRION ◽  
JEAN-CLAUDE MARTEL ◽  
YVAN DUMONT ◽  
ALAIN CADIEUX ◽  
FRANCOIS JOLICOEUR ◽  
...  
Keyword(s):  
Neuropeptide Y ◽  
Structure Activity Relationships ◽  
Structure Activity ◽  
The Brain ◽  
Y Receptors ◽  
Neuropeptide Y Receptors

Dexamethasone rapidly increases hypothalamic neuropeptide Y secretion in adrenalectomized ob/ob mice

1996 ◽  
Vol 271 (1) ◽  
pp. E151-E158 ◽  
Author(s):  
H. L. Chen ◽  
D. R. Romsos
Keyword(s):  
Neuropeptide Y ◽  
Arcuate Nucleus ◽  
Rapid Onset ◽  
Brown Adipose ◽  
Specific Enhancement ◽  
Brown Adipose Tissue Thermogenesis ◽  
The Central Nervous System ◽  
Rapid Transport ◽  
The Brain

A single intracerebroventricular injection of dexamethasone (DEX) rapidly (within 30 min) suppresses brown adipose tissue thermogenesis and increases plasma insulin concentrations in adrenal-ectomized (ADX) ob/ob mice but not in ADX lean mice. Intracerebroventricular neuropeptide Y (NPY) administered intracerebroventricularly causes these same metabolic changes within 30 min in both ob/ob and lean ADX mice. We therefore hypothesized that DEX exerts these rapid-onset metabolic actions in ob/ob mice via a phenotype-specific enhancement of NPY secretion within the central nervous system. In support of this hypothesis, DEX (a type II glucocorticoid receptor agonist) administered intracerebroventricularly selectively lowered NPY concentrations in the whole hypothalamus of ADX ob/ob mice by 35% and in the arcuate nucleus region by approximately 70% within 30 min but not in the brain stem or hippocampus or in any of these regions of lean mice. DEX also functioned in vitro to enhance depolarization-dependent release of NPY from hypothalamic blocks of ADX ob/ob mice but not of ADX lean mice. Thus DEX acts in the hypothalamus of ob/ob mice in a phenotype-specific manner to evoke rapid transport of NPY from cell bodies within the arcuate nucleus to terminal regions including the dorsomedial and ventromedial hypothalamic regions for release.

scholarly journals Cyclooxygenase in the brain: their cellular localization and functions

Ensho Saisei ◽  
2001 ◽  
Vol 21 (3) ◽  
pp. 209-217
Author(s):  
Kiyoshi Matsumura ◽  
Shigeo Kobayashi
Keyword(s):  
Cellular Localization ◽  
The Brain

Interaction between neuropeptide Y immunoreactive neurons and galanin immunoreactive neurons in the brain of the masu salmon, Oncorhynchus masou

2009 ◽  
Vol 462 (1) ◽  
pp. 33-38 ◽  
Author(s):  
Masafumi Amano ◽  
Noriko Amiya ◽  
Mikiko Hiramatsu ◽  
Takuma Tomioka ◽  
Yoshitaka Oka
Keyword(s):  
Neuropeptide Y ◽  
Masu Salmon ◽  
Oncorhynchus Masou ◽  
The Brain ◽  
Masu Salmon Oncorhynchus Masou

Characterization of the Brain Penetrant Neuropeptide Y Y2 Receptor Antagonist SF-11

2019 ◽  
Vol 10 (8) ◽  
pp. 3454-3463 ◽  
Author(s):  
Helena Domin ◽  
Natalia Piergies ◽  
Ewa Pięta ◽  
Elżbieta Wyska ◽  
Bartłomiej Pochwat ◽  
...  
Keyword(s):  
Neuropeptide Y ◽  
Receptor Antagonist ◽  
Y2 Receptor ◽  
The Brain
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