Effects of the Ca2+ sensitizers EMD 57033 and CGP 48506 on myocardial contractility and Ca2+ transients in human ventricular and atrial myocardium

2002 ◽  
Vol 91 (4) ◽  
pp. 312-318 ◽  
Author(s):  
Klara Brixius ◽  
Sebastian Reicke ◽  
Hannes Reuter ◽  
Robert H. G. Schwinger
Keyword(s):  
Myocardial Contractility ◽  
Atrial Myocardium ◽  
Cgp 48506

304 Fibrosis is an important mechanism of gap junction disorganisation in rat atrial myocardium during heart failure

2003 ◽  
Vol 2 (1) ◽  
pp. 54
Author(s):  
C RUCKERMARTIN ◽  
P MILLIEZ ◽  
N DEANGELIS ◽  
C GOYENVALLE ◽  
J RENAUD ◽  
...  
Keyword(s):  
Heart Failure ◽  
Gap Junction ◽  
Atrial Myocardium

Methylene blue reduces myocardial contractility

2012 ◽  
Vol 60 (S 01) ◽  
Author(s):  
H Weiler ◽  
O Moeller ◽  
M Wohlhoefer ◽  
LO Conzelmann ◽  
J Albers ◽  
...  
Keyword(s):  
Methylene Blue ◽  
Myocardial Contractility

scholarly journals Morpho-functional changes of cardiac telocytes in isolated atrial amyloidosis in patients with atrial fibrillation

2021 ◽  
Vol 11 (1) ◽  
Author(s):  
Tatyana V. Sukhacheva ◽  
Natalia V. Nizyaeva ◽  
Maria V. Samsonova ◽  
Andrey L. Cherniaev ◽  
Artem A. Burov ◽  
...  
Keyword(s):  
Atrial Fibrillation ◽  
Mitral Valve Regurgitation ◽  
Ultrastructural Level ◽  
Cardiomyocyte Hypertrophy ◽  
Myocardial Remodeling ◽  
Valve Regurgitation ◽  
Atrial Myocardium ◽  
Functional Changes ◽  
Age Related ◽  
Transitional Forms

AbstractTelocytes are interstitial cells with long, thin processes by which they contact each other and form a network in the interstitium. Myocardial remodeling of adult patients with different forms of atrial fibrillation (AF) occurs with an increase in fibrosis, age-related isolated atrial amyloidosis (IAA), cardiomyocyte hypertrophy and myolysis. This study aimed to determine the ultrastructural and immunohistochemical features of cardiac telocytes in patients with AF and AF + IAA. IAA associated with accumulation of atrial natriuretic factor was detected in 4.3–25% biopsies of left (LAA) and 21.7–41.7% of right (RAA) atrial appendage myocardium. Telocytes were identified at ultrastructural level more often in AF + IAA, than in AF group and correlated with AF duration and mitral valve regurgitation. Telocytes had ultrastructural signs of synthetic, proliferative, and phagocytic activity. Telocytes corresponded to CD117+, vimentin+, CD34+, CD44+, CD68+, CD16+, S100-, CD105- immunophenotype. No significant differences in telocytes morphology and immunophenotype were found in patients with various forms of AF. CD68-positive cells were detected more often in AF + IAA than AF group. We assume that in aged AF + IAA patients remodeling of atrial myocardium provoked transformation of telocytes into “transitional forms” combining the morphological and immunohistochemical features with signs of fibroblast-, histiocyte- and endotheliocyte-like cells.

Evaluation of the length constant in the atrial myocardium and pulmonary vein myocardium in mammals

2015 ◽  
Vol 460 (1) ◽  
pp. 8-11 ◽  
Author(s):  
V. S. Kuzmin ◽  
Yu. V. Egorov ◽  
V. M. Karimova ◽  
L. V. Rosenshtraukh
Keyword(s):  
Pulmonary Vein ◽  
Atrial Myocardium ◽  
Length Constant

Ca-movement controlling myocardial contractility I

1973 ◽  
Vol 338 (3) ◽  
pp. 207-231 ◽  
Author(s):  
H. Tritthart ◽  
R. Kaufmann ◽  
H. -P. Volkmer ◽  
R. Bayer ◽  
H. Krause
Keyword(s):  
Myocardial Contractility
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