Portal vein calcification and associated biliary stricture in idiopathic portal hypertension (Banti's syndrome)

1998 ◽  
Vol 23 (2) ◽  
pp. 180-182 ◽  
Author(s):  
P. J. Pickhardt ◽  
D. M. Balfe
Keyword(s):  
Portal Hypertension ◽  
Portal Vein ◽  
Biliary Stricture ◽  
Idiopathic Portal Hypertension ◽  
Banti's Syndrome

Development of Portal Vein Thrombosis Complicating Idiopathic Portal Hypertension

1985 ◽  
Vol 88 (4) ◽  
pp. 1034-1040 ◽  
Author(s):  
Kunihiko Ohnishi ◽  
Masayuki Saito ◽  
Hidetaka Terabayashi ◽  
Fumio Nomura ◽  
Kunio Okuda
Keyword(s):  
Portal Hypertension ◽  
Portal Vein ◽  
Portal Vein Thrombosis ◽  
Idiopathic Portal Hypertension ◽  
Vein Thrombosis

Postoperative Portal Vein Obstruction in Patients with Idiopathic Portal Hypertension

1990 ◽  
Vol 12 (5) ◽  
pp. 607 ◽  
Author(s):  
Hector Orozco ◽  
Takeshi Takahashi ◽  
Miguel Angel Mercado ◽  
Guadalupe Garcia-Tsao ◽  
Aurora Gonzalez-Lopez-Lira ◽  
...  
Keyword(s):  
Portal Hypertension ◽  
Portal Vein ◽  
Idiopathic Portal Hypertension ◽  
Portal Vein Obstruction

scholarly journals Deficiencies in proteins C and S in a patient with idiopathic portal hypertension accompanied by portal vein thrombosis

2010 ◽  
Vol 16 (2) ◽  
pp. 176 ◽  
Author(s):  
Sena Hwang ◽  
Do Young Kim ◽  
Minju Kim ◽  
Young Eun Chon ◽  
Hyun Jung Lee ◽  
...  
Keyword(s):  
Portal Hypertension ◽  
Portal Vein ◽  
Portal Vein Thrombosis ◽  
Idiopathic Portal Hypertension ◽  
Vein Thrombosis

Pathomorphologic study on the extrahepatic portal vein in idiopathic portal hypertension

2008 ◽  
Vol 10 (4) ◽  
pp. 209-216 ◽  
Author(s):  
Masayoshi Kage ◽  
Masahiro Arakawa ◽  
Kazunori Fukuda ◽  
Masamichi Kojiro
Keyword(s):  
Portal Hypertension ◽  
Portal Vein ◽  
Idiopathic Portal Hypertension

Colorectal Variceal Bleeding in Patients With Extrahepatic Portal Vein Thrombosis and Idiopathic Portal Hypertension

1992 ◽  
Vol 14 (2) ◽  
pp. 139-143 ◽  
Author(s):  
Hector Orozco ◽  
Takeshi Takahashi ◽  
Miguel A. Mercado ◽  
Eduardo Prado-Orozco ◽  
Hector Ferral ◽  
...  
Keyword(s):  
Portal Hypertension ◽  
Portal Vein ◽  
Portal Vein Thrombosis ◽  
Variceal Bleeding ◽  
Idiopathic Portal Hypertension ◽  
Vein Thrombosis

Portal hemodynamics in idiopathic portal hypertension (Banti's syndrome)

1987 ◽  
Vol 92 (3) ◽  
pp. 751-758 ◽  
Author(s):  
Kunihiko Ohnishi ◽  
Masayuki Saito ◽  
Shinichi Sato ◽  
Hidetaka Terabayashi ◽  
Shinji Iida ◽  
...  
Keyword(s):  
Portal Hypertension ◽  
Idiopathic Portal Hypertension ◽  
Portal Hemodynamics ◽  
Banti's Syndrome

A Comparative Clinical Study of Idiopathic Portal Hypertension, Extrahepatic Portal Vein Thrombosis, and Cirrhosis

1994 ◽  
Vol 19 (3) ◽  
pp. 217-221 ◽  
Author(s):  
Hector Orozco ◽  
Takeshi Takahashi ◽  
Guadalupe Garcia-Tsao ◽  
Miguel Angel Mercado ◽  
Eduardo Prado ◽  
...  
Keyword(s):  
Portal Hypertension ◽  
Clinical Study ◽  
Portal Vein ◽  
Portal Vein Thrombosis ◽  
Idiopathic Portal Hypertension ◽  
Vein Thrombosis ◽  
Comparative Clinical Study

scholarly journals Non-cirrhotic portal hypertension: current concepts and modern management

2016 ◽  
Vol 43 (3) ◽  
pp. 170-176
Author(s):  
Md Ismail Patwary ◽  
Matiur Rahman ◽  
Kaushik Mojumder
Keyword(s):  
Portal Hypertension ◽  
Portal Vein ◽  
Variceal Bleeding ◽  
Gastric Varices ◽  
Idiopathic Portal Hypertension ◽  
Endoscopic Sclerotherapy ◽  
Cyanoacrylate Glue ◽  
Portal Fibrosis ◽  
Vascular Origin ◽  
Glue Injection

Non-cirrhotic portal hypertension (NCPH) is a heterogeneous group of liver disorders of vascular origin, leading to portal hypertension (PHTN) in the absence of cirrhosis.The lesions are generallyvascular, either in the portal vein, its branches or in the peri-sinusoidal area. The majority of diseases included in the category of NCPH are well-characterized disease entities where PHTN is a late manifestation. Two diseases that present only with features of PHTN and are common in developing countries are non-cirrhotic portal fibrosis (NCPF) and extrahepatic portal vein obstruction (EHPVO). Non-cirrhotic portal fibrosis is a syndrome of obscure etiology, characterized by ‘obliterative-portovenopathy’ leading to PHT, massive splenomegaly and well-tolerated episodes of variceal bleeding in young adults from low socioeconomic backgrounds, having near normal hepatic functions. In some parts of the world, NCPFis called idiopathic portal hypertension in Japan or ‘hepatoportalsclerosis’in USA. Because 85–95% of patient with NCPF and EHPVO present with variceal bleeding, treatment involves management with endoscopic sclerotherapy (EST) or variceal ligation (EVL). These therapies are effective in approximately 90–95% of patients. Gastric varices are another common cause of upper gastrointestinal bleeding in these patients and these can be managed with cyanoacrylate glue injection or surgery. The prognosis of patients with NCPF is good and 5 years survival in patients in whom variceal bleeding can be controlled has been reported to be approximately 95–100%.Bangladesh Med J. 2014 Sep; 43 (3): 170-176

scholarly journals Banti’s Syndrome Presenting as Hematemesis - A Case Report

2021 ◽  
Vol 10 (10) ◽  
pp. 749-751
Author(s):  
Aishwarya Ghule ◽  
Sourya Acharya ◽  
Samarth Shukla ◽  
Sunil Kumar ◽  
Parth Godhiwala
Keyword(s):  
Portal Hypertension ◽  
Portal Pressure ◽  
Normal Liver ◽  
Liver Function Tests ◽  
Massive Bleeding ◽  
Idiopathic Portal Hypertension ◽  
Hepatic Veins ◽  
Massive Splenomegaly ◽  
History Of ◽  
Banti's Syndrome

Massive splenomegaly presenting with hypersplenism, pancytopenia and portal hypertension, without any underlying known cause is known as Banti’s syndrome. There are various causes of splenomegaly. When all the known causes of portal hypertension are ruled out, it is termed as Banti’s syndrome. This syndrome was discovered by Guido Banti in 1882 and is named after him. Banti’s syndrome is also known as idiopathic portal hypertension or non-cirrhotic portal fibrosis.1 Banti’s syndrome is commonly found in India and Japan than in the West. 2 There is absence of any haematologic cause, primary hepatic cause or any tumour or mass lesion involving the spleen. Banti had stated that the primary organ involved was spleen and not the liver leading to secondary splenomegaly. Other features include normal liver function tests, varices seen in endoscopy, cytopenia of one or more cell lines, absence of cirrhosis, patent hepatic veins and elevated portal pressure with multiple collaterals. The complications include rupture of varices and massive bleeding. 3 We report a case of a 20-year-old male who presented to us with a history of fever for 7 days and one-episode of hematemesis on the day of admission. All known causes of hypersplenism were ruled out and he was diagnosed to have idiopathic massive splenomegaly with portal hypertension and hypersplenism.

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