Spontaneous regression of cerebral arteriovenous malformations: clinical and angiographic analysis with review of the literature

2002 ◽  
Vol 44 (1) ◽  
pp. 11-16 ◽  
Author(s):  
S. Lee ◽  
P. Vilela ◽  
R. Willinsky ◽  
K. TerBrugge
1993 ◽  
Vol 123 (3-4) ◽  
pp. 101-112 ◽  
Author(s):  
R. Deruty ◽  
I. Pelissou-Guyotat ◽  
C. Mottolese ◽  
Y. Bascoulergue ◽  
D. Amat

2014 ◽  
Vol 81 (1) ◽  
pp. 83-90 ◽  
Author(s):  
Nitin Agarwal ◽  
Jacqueline C. Guerra ◽  
Nihar B. Gala ◽  
Prateek Agarwal ◽  
Alexandros Zouzias ◽  
...  

Neurosurgery ◽  
1988 ◽  
Vol 23 (6) ◽  
pp. 781-785 ◽  
Author(s):  
M. Nazek ◽  
T. I. Mandybur ◽  
S. Kashiwagi

Abstract A peculiar nonneoplastic oligodendroglial proliferative abnormality associated with cerebral arteriovenous malformations (AVMs) was present in three patients. Histological examination of biopsy material revealed dense oligodendroglial tissue reminiscent of oligodendroglioma in the white matter adjoining the AVMs. Careful consideration of clinical and pathological features suggested that the evidence was insufficient to qualify the lesion as truly neoplastic (oligodendroglioma); rather, a tissue collapse or a hamartomatous proliferation could be considered to be its cause. The literature contains 14 instances of various vascular malformations associated with primary brain tumors, 5 of which were diagnosed as oligodendrogliomas. It is possible, however, that some of the cases reported in the literature constitute oligodendroglial abnormality similar to that observed in our cases rather than genuine oligodendrogliomas. Attention is drawn to this interesting and prognostically important phenomenon.


1999 ◽  
Vol 5 (1_suppl) ◽  
pp. 157-160 ◽  
Author(s):  
S. Hirai ◽  
S. Mine ◽  
E. Kobayashi ◽  
I. Yamakami ◽  
A. Yamaura

To find out lesions responsible for hemorrhage in arteriovenous malformations (AVMs), a retrospective study of angioarchitecture around the nidus was conducted in 27 patients who underwent conservative treatment. Comparison of angiograms revealed disappearance of an intranidal aneurysmal dilatation after the hemorrhagic events in two cases. The hematomas were adjacent to the dilatation, and no subarachnoid hemorrhage was evident. Obstruction of venous drainage, noticed in a case of spontaneous regression of AVM, was not demonstrated in the cases of hemorrhage. The intranidal aneurysmal dilatation is likely to have caused the hemorrhage in our cases. Careful endavascular embolization using proper materials should be indicated for an intranidal aneurysmal dilatation to prevent subsequent hemorrhage.


2004 ◽  
Vol 62 (4) ◽  
pp. 324-330 ◽  
Author(s):  
Norberto Andaluz ◽  
John S. Myseros ◽  
Sumeer Sathi ◽  
Kerry R. Crone ◽  
John M. Tew

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