Acute hyperparathyroidism and vascular thrombosis; An unrecognized association

2010 ◽  
Vol 33 (9) ◽  
pp. 683-683
Author(s):  
A. Franchello ◽  
M. Camandona ◽  
G. Gasparri
1960 ◽  
Vol 39 (1) ◽  
pp. 83-89 ◽  
Author(s):  
Abraham Atsmon ◽  
Menachem Frank ◽  
Paul Nathan ◽  
Andre de Vries

2001 ◽  
Vol 21 (03) ◽  
pp. 82-96 ◽  
Author(s):  
D. Hoppensteadt ◽  
O. Iqbal ◽  
R. L. Bick ◽  
J. Fareed

SummaryThrombotic disorders are the most common cause of death in the United States. About two million individuals die each year from an arterial or venous thrombosis or related disorders. About 80% to 90% of all cases of thrombosis can now be defined with respect to cause. Of these, over 50% occur in patients who harbor a congenital or acquired blood coagulation protein or platelet defect which caused the thrombotic event. It is obviously of major importance to define those individuals harboring such a defect as this allows: 1) appropriate antithrombotic therapy to decrease risks of recurrence; 2) determination of the length of time the patient must remain on therapy for secondary prevention; and 3) allow for testing of family members of those harboring a blood coagulation protein or platelet defect which is hereditary (about 50% of all coagulation and platelet defects mentioned above). Aside from mortality, significant additional morbidity occurs from both arterial or venous thrombotic events, including, but not limited to paralysis (non-fatal thrombotic stroke), cardiac disability (repeated coronary events), loss of vision (retinal vascular thrombosis), fetal waste syndrome (placental vascular thrombosis), stasis ulcers and other manifestations of post-phlebitic syndrome, etc.


2020 ◽  
Author(s):  
Jameel Al- Ata ◽  
Gaser Abdelmohsen ◽  
Saud Bahaidarah ◽  
Naif Alkhushi ◽  
Zaher Zaher

IntroductionNeonates with congenital heart disease are at a high risk of vascular thrombosis. Thrombosis may occur due to vascular injury, increased blood viscosity secondary to polycythemia associated with congenital cyanotic heart diseases, or stasis of blood flow associated with low cardiac output (Schmidt B & Andrew M., Pediatrics 1995; 96: 939–943. Veldman A et al.,Vasc Health Risk Manag 2008; 4: 1337–1348).


1934 ◽  
Vol 30 (10) ◽  
pp. 1027-1029
Author(s):  
A. I. Petchenko

Vascular thrombosis in obstetrics and gynecology is quite common, although not always easy to recognize. In the postpartum period, after many vaginal operations we have massive thrombus formation. Placental vein thrombosis is almost physiological; slowed blood flow, decreased vascular tone after the fetus emerges, blood loss are predisposing moments to thrombosis formation in the postpartum period.


1998 ◽  
Vol 29 (8) ◽  
pp. 663-668
Author(s):  
Darius M Moshfeghi ◽  
Gholam A Peyman ◽  
Andrew A Moshfeghi ◽  
Bahram Khoobehi ◽  
George B Primbs ◽  
...  

BMJ ◽  
1964 ◽  
Vol 2 (5410) ◽  
pp. 675-676 ◽  
Author(s):  
A. Pringle ◽  
E. K. M. Smith

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