Monitoring the pressure of the sphincter of Oddi by percutaneous transhepatic choledochoscopy: New method for evaluating motor function

1991 ◽  
Vol 26 (4) ◽  
pp. 507-513
Author(s):  
Jun-ichi Kanoh ◽  
Saburo Nakazawa ◽  
Yasuo Naito ◽  
Kazuo Inui ◽  
Yoshihisa Tsukamoto
Keyword(s):  
Motor Function ◽  
Sphincter Of Oddi ◽  
New Method ◽  
Percutaneous Transhepatic Choledochoscopy

Normal sphincter of Oddi motor function

2004 ◽  
Vol 6 (2) ◽  
pp. 163-168 ◽  
Author(s):  
Kinnari Kher ◽  
Moises Guelrud
Keyword(s):  
Motor Function ◽  
Sphincter Of Oddi

scholarly journals Motor function of the opossum sphincter of Oddi.

1983 ◽  
Vol 71 (2) ◽  
pp. 208-220 ◽  
Author(s):  
J Toouli ◽  
W J Dodds ◽  
R Honda ◽  
S Sarna ◽  
W J Hogan ◽  
...  
Keyword(s):  
Motor Function ◽  
Sphincter Of Oddi

scholarly journals The Effect of Propofol on the Canine Sphincter of Oddi

HPB Surgery ◽  
1994 ◽  
Vol 7 (4) ◽  
pp. 297-304 ◽  
Author(s):  
T. H. Baron ◽  
C. B. Dalton ◽  
P. B. Cotton ◽  
G. R. May ◽  
L. G. Milton ◽  
...  
Keyword(s):  
Phase I ◽  
Motor Function ◽  
Sphincter Of Oddi ◽  
Migrating Motor Complex ◽  
Pressure Amplitude ◽  
Low Doses ◽  
Basal Pressure ◽  
Motor Complex ◽  
Single Lumen

To assess the effect of propofol on the canine sphincter of Oddi (SO), sphincter of Oddi manometry (SOM) was performed in fasting dogs which had undergone cholecystectomy and placement of modified Thomas duodenal cannulae. Using two water-perfused, single-lumen manometric catheters, SO and duodenal pressures were measured simultaneously. Baseline SO activity was recorded for at least one complete interdigestive cycle followed by bolus injections of propofol (Diprivan ®) (N = 31) from 0.1 to 4.0 mg/kg during Phase I of the Migrating Motor Complex (MMC).When propofol was administered in bolus doses ≤ 0.5 mg/kg, no change in SO or duodenal motor function was seen. In doses ≥ 0.5 mg/kg, SO basal pressure, amplitude, and frequency of contractions increased significantly. Increases in duodenal activity paralleled SO activity. Our results suggest that propofol in low doses may be useful for sedation during Sphincter of Oddi manometry in humans. Further studies of the effects of propofol on the human sphincter of Oddi are warranted.

An Adrenergic Nervous System in Sea Anemones

1963 ◽  
Vol s3-104 (68) ◽  
pp. 531-534
Author(s):  
ERIK DAHL ◽  
BENGT FALCK ◽  
CLAES VON MECKLENBURG ◽  
HARRY MYHRBERG
Keyword(s):  
Nervous System ◽  
Fluorescence Microscopy ◽  
Motor Function ◽  
New Method ◽  
Sea Anemones ◽  
Fluorescent Substance ◽  
Metridium Senile ◽  
Adrenergic Nervous System ◽  
Oral Disk

A new method for the demonstration of certain mono-amines by means of fluorescence microscopy was applied to the tentacles and oral disk of the sea anemones Metridium senile and Tealia felina. A fluorescent substance was found in the cells and fibres of the tentacular ectodermal nervous system. This nervous system probably has a double sensory and motor function.

The role of antioxidant enzymes in the control of opossum sphincter of Oddi motility

1997 ◽  
Vol 272 (5) ◽  
pp. G1050-G1056 ◽  
Author(s):  
J. J. Cullen ◽  
B. M. Herrmann ◽  
R. M. Thomas ◽  
S. Fang ◽  
J. A. Murray ◽  
...  
Keyword(s):  
Antioxidant Enzymes ◽  
Motor Function ◽  
Circular Muscle ◽  
Sphincter Of Oddi ◽  
Electrical Field Stimulation ◽  
Sod Activity ◽  
Diethyldithiocarbamic Acid ◽  
Sphincter Of Oddi Motility ◽  
Displacement Transducers ◽  
Scavenging Enzymes

Superoxide rapidly oxidizes nitric oxide (NO) to form peroxynitrite, thus terminating the biological activity of NO. The aims of our study were to determine if superoxide alters the motor function of the sphincter of Oddi and to localize the antioxidant enzymes in the sphincter of Oddi. Immunostaining was performed and enzyme activities were measured in the sphincter of Oddi. In physiological experiments, force-displacement transducers recorded tension in the spontaneously contracting sphincter of Oddi and after electrical field stimulation (EFS) of precontracted sphincter of Oddi. Superoxide was generated by the addition of xanthine with xanthine oxidase, superoxide radicals were scavenged by the addition of superoxide dismutase (SOD), and catalase or SOD was inhibited by diethyldithiocarbamic acid. Immunostaining demonstrated SOD and catalase immunoreactivity in ganglia situated at the serosal surface of the circular muscle. Total SOD activity was 202 +/- 12 U/mg. Generation of superoxide or inhibition of SOD increased the contractile frequency and decreased relaxation after EFS. We conclude that superoxide alters sphincter of Oddi motor function, and the presence of superoxide scavenging enzymes in enteric plexuses suggests that they may regulate sphincter of Oddi neuromuscular function by clearing endogenous superoxide.

Effect of histamine on motor function of opossum sphincter of Oddi

1981 ◽  
Vol 241 (2) ◽  
pp. G122-G128 ◽  
Author(s):  
J. Toouli ◽  
W. J. Dodds ◽  
R. Honda ◽  
W. J. Hogan
Keyword(s):  
Smooth Muscle ◽  
Motor Function ◽  
Large Dose ◽  
Contractile Activity ◽  
Sphincter Of Oddi ◽  
Inhibitory Effect ◽  
Excitatory Effect ◽  
Catheter System ◽  
Infused Catheter System ◽  
Stimulation Of

In this study, we evaluated the effect of histamine on phasic contractile activity in the opossum sphincter of Oddi (SO). SO manometry was done in 35 animals, using an infused catheter system with minimal compliance. In anesthetized animals, phasic SO contractions occurred at a frequency of 7.3 +/- 0.3 (SE) contractions/min with an amplitude of 83 +/- 4 mmHg. Intravenous histamine (5-80 micrograms/kg) invariably inhibited the frequency and amplitude of SO phasic contractions. At larger doses, the SO contractions were abolished for several minutes. The SO inhibitory effect of histamine was duplicated by the selective H1-agonist, 2-pyridylethylamine, and abolished by H1-blockade with pyrilamine or neural blockade with tetrodotoxin. After tetrodotoxin, histamine and 2-pyridylethylamine caused an increased frequency and amplitude of SO contractions. This excitatory effect was blocked by pyrilamine. The histamine effects on SO phasic contractions were not altered by metiamide, atropine, phentolamine, propranolol, hexamethonium, or a large dose of nicotine. We conclude that 1) histamine depresses phasic SO contractions in the opossum; 2) histamine's depressant SO effect is mediated by H1 stimulation of noncholinergic, nonadrenergic SO inhibitory nerves, overriding an H1 stimulatory effect on SO smooth muscle; and 3) histamine has no H2-mediated effect on the opossum SO.

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