The increased vulnerability of gastric mucosa against intraluminar acid due to high blood ethanol level in rats

1990 ◽  
Vol 25 (3) ◽  
pp. 400-400
Author(s):  
Sunao Kawano ◽  
Eiji Masuda ◽  
Kouichi Nagano ◽  
Nobuhiro Sato ◽  
Takenobu Kamada
Keyword(s):  
Gastric Mucosa ◽  
Ethanol Level ◽  
Blood Ethanol Level

scholarly journals The Effect of Blood Ethanol Level on Mortality in Patients with Trauma

2019 ◽  
Vol 26 (1) ◽  
pp. 53-60
Author(s):  
Burak Altan ◽  
Muhammed İkbal Şaşmaz
Keyword(s):  
Ethanol Level ◽  
Blood Ethanol Level

An Unusually High Blood Ethanol Level in a Living Patient

1977 ◽  
Vol 10 (4) ◽  
pp. 429-431 ◽  
Author(s):  
ALPHONSE POKLIS ◽  
A. Pearson Margaret
Keyword(s):  
Ethanol Level ◽  
Blood Ethanol Level

A Low Blood Ethanol Level Is Associated with Improved Cytokine Production in Aged Mice after Traumatic Injury

2005 ◽  
Vol 59 (4) ◽  
pp. 984-989 ◽  
Author(s):  
Timothy P. Plackett ◽  
Jennifer Jarrett ◽  
Richard L. Gamelli ◽  
Elizabeth J. Kovacs
Keyword(s):  
Cytokine Production ◽  
Traumatic Injury ◽  
Aged Mice ◽  
Ethanol Level ◽  
Blood Ethanol Level

scholarly journals Trace Amine Associated Receptor 1 Modulates Behavioral Effects of Ethanol

2013 ◽  
Vol 7 ◽  
pp. SART.S12110 ◽  
Author(s):  
Laurie J. Lynch ◽  
Katherine A. Sullivan ◽  
Eric J. Vallender ◽  
James K. Rowlett ◽  
Donna M. Platt ◽  
...  
Keyword(s):  
Locomotor Activity ◽  
Treatment Options ◽  
Motor Impairment ◽  
Ethanol Level ◽  
Loss Of Righting Reflex ◽  
Significant Difference ◽  
Trace Amine ◽  
Acute Ethanol ◽  
Righting Reflex ◽  
Blood Ethanol Level

Background Few treatment options for alcohol use disorders (AUDs) exist and more are critically needed. Here, we assessed whether trace amine associated receptor 1 (TAAR1), a modulator of brain monoamine systems, is involved in the behavioral and reinforcement-related effects of ethanol and whether it could potentially serve as a therapeutic target. Methods Wild-type (WT) and TAAR1 knockout (KO) mice (75% C57J/BL6 and 25% 129S1/Sv background) were compared in tests of ethanol consumption (two-bottle choice [TBC]), motor impairment (loss of righting reflex, [LORR], locomotor activity) and ethanol clearance (blood ethanol level [BEL]). Results As compared with WT mice, KO mice displayed (1) significantly greater preference for and consumption of ethanol in a TBC paradigm (3%–11% vol/vol escalating over 10 weeks), with no significant difference observed in TBC with sucrose (1%–3%); (2) significantly greater sedative-like effects of acute ethanol (2.0 or 2.5 g/kg, intraperitoneal [i.p.]) manifested as LORR observed at a lower dose and for longer time, with similar BELs and rates of ethanol clearance; and (3) lower cumulative locomotor activity over 60 minutes in response to an acute ethanol challenge (1.0–2.5 g/kg, i.p.). Conclusions The present findings are the first to implicate TAAR1 in the behavioral and reinforcement-related effects of ethanol and raise the question of whether specific drugs that target TAAR1 could potentially reduce alcohol consumption in humans with AUDs.

scholarly journals Morphometry of Cortical Neurons in Acute Clozapine and Ethanol Poisoning

2021 ◽  
Vol 16 (6) ◽  
pp. 19-30
Author(s):  
A. R. Bashirova ◽  
D. V. Sundukov ◽  
A. S. Babkina ◽  
M. A. Golubev ◽  
I. N. Telipov
Keyword(s):  
Statistical Analysis ◽  
Cortical Neurons ◽  
Morphological Analysis ◽  
Neuronal Damage ◽  
Parametric Methods ◽  
Irreversible Damage ◽  
Ethanol Level ◽  
Hematoxylin And Eosin ◽  
Ethanol Poisoning ◽  
Blood Ethanol Level

The aim of the study is to summarize the histology and morphometry of cortical neurons in acute clozapine and ethanol poisoning.Material and methods. Histological examination of the parietal cortical brain samples of 26 patients died during the Day 1 of acute clozapine and ethanol poisoning (23 males and 3 females aged 22-63 years) was performed. The blood ethanol level was 1.4-4.1%o. The level of clozapine in the blood ranged between 0.24 and 5.8 mg%, in the liver between 0.097 and 6.5 mg%, in kidneys between 0.03 and 3.5 mg%. The cortical samples for morphometric examination were placed in 10% neutral paraformaldehyde, the histological sections were done and stained with hematoxylin and eosin, as well as according to Niessl. The morphological analysis was performed using the video light microscopy. The number of damaged neurons (with separate quantification of reversible, intermediate, and irreversible damage) was assessed. The statistical analysis was done using the non-parametric methods.Results. The signs of brain neuronal damage in acute clozapine and ethanol poisoning, as well as forensic chemical tests, might be used for establishing the direct cause of death.

Alcohol after laparoscopic sleeve gastrectomy: high blood ethanol level or fast metabolism?

2018 ◽  
Vol 14 (11) ◽  
pp. S110
Author(s):  
Angelo AI Iossa ◽  
Maria Chiara Ciccioriccio ◽  
Anna Guida ◽  
Gianfranco Silecchia
Keyword(s):  
Sleeve Gastrectomy ◽  
Laparoscopic Sleeve Gastrectomy ◽  
Ethanol Level ◽  
Blood Ethanol Level
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