Experimental carcinoma of the biliary tract induced in dogs by N-ethyl-N’-nitro-N-nitrosoguanidine

1977 ◽  
Vol 12 (2) ◽  
pp. 47-50 ◽  
Author(s):  
Saburo Nakazawa ◽  
Yasuo Naito ◽  
Yoshiki Yamamoto ◽  
Hiroshiko Yamase ◽  
Kunio Sobue ◽  
...  
Keyword(s):  
Biliary Tract ◽  
Experimental Carcinoma

Roentgenology of the Liver and Biliary Tract

1963 ◽  
Vol 45 (5) ◽  
pp. 644-657 ◽  
Author(s):  
Robert E. Wise
Keyword(s):  
Biliary Tract

The Diagnosis of Cancer of the Pancreas, Biliary Tract, and Duodenum by Combined Cytologic and Secretory Methods

1958 ◽  
Vol 34 (6) ◽  
pp. 996-1008 ◽  
Author(s):  
Howard F. Raskin ◽  
Julius Wenger ◽  
Manuel Sklar ◽  
Sylvia Pleticka ◽  
Willard Yarema
Keyword(s):  
Biliary Tract ◽  
Cancer Of The Pancreas ◽  
Diagnosis Of Cancer

Chemotherapy in biliary tract carcinomas: Results in India

2002 ◽  
Vol 29 (6U) ◽  
pp. 46-50 ◽  
Author(s):  
Dinesh C. Doval ◽  
Jagdev S. Sekhon ◽  
Sunil K. Gupta ◽  
Sanjay Gupta ◽  
Bhawana S. Awasthy
Keyword(s):  
Biliary Tract

EGFR and HER2 Expression in advanced Biliary Tract Cancer

2008 ◽  
Vol 46 (09) ◽  
Author(s):  
J Harder ◽  
O Waiz ◽  
M Geissler ◽  
HE Blum ◽  
A Schmitt-Gräff ◽  
...  
Keyword(s):  
Biliary Tract ◽  
Biliary Tract Cancer ◽  
Her2 Expression ◽  
Tract Cancer

Lymph Node Metastasis in the Biliary Tract Carcinoma: CT Evaluation

1994 ◽  
Vol 31 (1) ◽  
pp. 119
Author(s):  
Young Soo Do ◽  
Byung Hee Lee ◽  
Kie Hwan Kim ◽  
Soo Yil Chin ◽  
Ah Ra Lee
Keyword(s):  
Lymph Node ◽  
Lymph Node Metastasis ◽  
Biliary Tract ◽  
Biliary Tract Carcinoma ◽  
Node Metastasis ◽  
Ct Evaluation

Reprogrammed M1 macrophages with inhibited STAT3, STAT6 and/or SMAD3 extends lifespan of mice with experimental carcinoma

2017 ◽  
pp. 4-9
Author(s):  
С.В. Калиш ◽  
С.В. Лямина ◽  
А.А. Раецкая ◽  
И.Ю. Малышев
Keyword(s):  
Tumor Growth ◽  
Culture Medium ◽  
Ehrlich Carcinoma ◽  
Macrophage Phenotype ◽  
M1 Macrophages ◽  
M1 Macrophage ◽  
Ec Cells ◽  
Experimental Carcinoma

Цель исследования. Репрограммирование М1 фенотипа макрофагов с ингибированными факторами транскрипции М2 фенотипа STAT3, STAТ6 и SMAD и оценка их влияния на развитие карциномы Эрлиха (КЭ) in vitro и in vivo. Методика. Рост опухоли иницировали in vitro путем добавления клеток КЭ в среду культивирования RPMI-1640 и in vivo путем внутрибрюшинной инъекции клеток КЭ мышам. Результаты. Установлено, что M1макрофаги и in vitro, и in vivo оказывают выраженный противоопухолевый эффект, который превосходит антиопухолевые эффекты М1, M1, M1 макрофагов и цисплатина. Заключение. М1 макрофаги с ингибированными STAT3, STAT6 и/или SMAD3 эффективно ограничивают рост опухоли. Полученные данные обосновывают разработку новой технологии противоопухолевой клеточной терапии. Objective. Reprogramming of M1 macrophage phenotype with inhibited M2 phenotype transcription factors, such as STAT3, STAT6 and SMAD and assess their impact on the development of Ehrlich carcinoma (EC) in vitro and in vivo . Methods. Tumor growth in vitro was initiated by addition of EC cells in RPMI-1640 culture medium and in vivo by intraperitoneal of EC cell injection into mice. Results. It was found that M1 macrophages have a pronounced anti-tumor effect in vitro , and in vivo , which was greater than anti-tumor effects of M1, M1, M1 macrophages and cisplatin. Conclusion. M1 macrophages with inhibited STAT3, STAT6 and/or SMAD3 effectively restrict tumor growth. The findings justify the development of new anti-tumor cell therapy technology.

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