Alkenyl and alkyl ether phospholipids in pig mesenteric lymph node lymphocytes

Lipids ◽  
1980 ◽  
Vol 15 (6) ◽  
pp. 475-478 ◽  
Author(s):  
Takayuki Sugiura ◽  
Yasuo Masuzawa ◽  
Keizo Waku
Keyword(s):  
Lymph Node ◽  
Mesenteric Lymph Node ◽  
Alkyl Ether ◽  
Mesenteric Lymph ◽  
Ether Phospholipids

An estimate of Salmonella prevalence on Illinois swine farms using mesenteric lymph node cultures

1999 ◽  
Author(s):  
D. Damman ◽  
P. B. Bahnson ◽  
R. M. Weigel ◽  
R. E. Isaacson ◽  
H. F. Troutt ◽  
...  
Keyword(s):  
Lymph Node ◽  
Mesenteric Lymph Node ◽  
Swine Farms ◽  
Mesenteric Lymph

scholarly journals Rotavirus infection causes mesenteric lymph node hypertrophy independently of type I interferon or TNFa in mice

2020 ◽  
Author(s):  
Joy Nakawesi ◽  
Konjit Getachew Muleta ◽  
Dragos‐Christian Dasoveanu ◽  
Bengt Johansson‐Lindbom ◽  
Katharina Lahl
Keyword(s):  
Lymph Node ◽  
Mesenteric Lymph Node ◽  
Type I Interferon ◽  
Type I ◽  
Rotavirus Infection ◽  
Mesenteric Lymph

scholarly journals IFN-γ Secreted by CD103+Dendritic Cells Leads to IgG Generation in the Mesenteric Lymph Node in the Absence of Vitamin A

2011 ◽  
Vol 186 (12) ◽  
pp. 6999-7005 ◽  
Author(s):  
Jae-Hoon Chang ◽  
Hye-Ran Cha ◽  
Sun-Young Chang ◽  
Hyun-Jeong Ko ◽  
Sang-Uk Seo ◽  
...  
Keyword(s):  
Dendritic Cells ◽  
Lymph Node ◽  
Vitamin A ◽  
Mesenteric Lymph Node ◽  
Mesenteric Lymph ◽  
Ifn Γ

Evidence for the involvement of a bone marrow-derived cell population in the immune expulsion ofTrichinella spiralis

Parasitology ◽  
1977 ◽  
Vol 74 (3) ◽  
pp. 225-234 ◽  
Author(s):  
D. Wakelin ◽  
Margaret M. Wilson
Keyword(s):  
Bone Marrow ◽  
Lymph Node ◽  
Cell Population ◽  
Mesenteric Lymph Node ◽  
Trichinella Spiralis ◽  
Mesenteric Lymph ◽  
Lymph Node Cells

When mice were irradiated immediately before infection withTrichinella spiralisthere was a profound and long-lasting interference with their ability to expel adult worms from the intestine. Irradiation given after the fifth day of infection was progressively less effective in this respect. The ability to expel worms was not restored when mesenteric lymph node cells (MLNC) were transferred (a) on the day of infection in mice irradiated one day previously, or (b) on day 7 of an infection in mice irradiated on day 6, even though the MLNC transferred immunity to intact recipients. Transfer of bone marrow (BM) alone was also without effect. However, worm explusion was restored if, following irradiation and injection of BM, 10 days were allowed for BM differentiation before transfer of MLNC. This restoration was effective even after lethal levels of irradiation and was clearly dependent upon a donor-derived BM component cooperating with, or responding to, the activity of the transferred MLNC. The possibility that the BM component is non-lymphoid in nature is discussed.

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