Use of the internal jugular vein approach in balloon dilatation angioplasty of pulmonary artery stenosis in children

1996 ◽  
Vol 17 (2) ◽  
pp. 82-85 ◽  
Author(s):  
H. Senzaki ◽  
K. Koike ◽  
T. Isoda ◽  
A. Ishizawa ◽  
T. Hishi ◽  
...  
1996 ◽  
Vol 17 (2) ◽  
pp. 82-85 ◽  
Author(s):  
H. Senzaki ◽  
K. Koike ◽  
T. Isoda ◽  
A. Ishizawa ◽  
T. Hishi ◽  
...  

Author(s):  
Qiang Zheng ◽  
Xiaoke Shang ◽  
Nianguo Dong ◽  
Jiawei Shi

Abstract Background COVID-19 continues to be a pandemic worldwide. Lung transplantation is the last option to increase life expectancy of end-stage COVID-19 patients. Branch pulmonary artery stenosis (PAS) is a rare complication after lung transplantation with an extremely poor prognosis. The current trend in the management of branch PAS is percutaneous balloon angioplasty and/or stent implantation, rather than high-risk reoperation with a lower success rate. Case summary The subject was a 54-year-old male with severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection who underwent a double-lung transplantation. He suffered hypoxemia and right heart dysfunction following the operation. Right cardiac catheterization and pulmonary angiography examination revealed severe stenosis of the right branch pulmonary artery. Due to immunosuppression and reduced coagulation function, the patient underwent pulmonary artery balloon dilatation and stent implantation, and ultimately recovered well. Discussion The combination of balloon dilatation and stent implantation is a good alternative to reoperation for patients with COVID-19.


2021 ◽  
Vol 11 (1) ◽  
Author(s):  
Cuilan Hou ◽  
Junmin Zheng ◽  
Wei liu ◽  
Lijian Xie ◽  
Xiaomin Sun ◽  
...  

AbstractCongenital heart defects, one of the most common birth defects, affect approximately 1% of live birth globally and remain the leading cause of infant mortality in developed countries. Utilizing the pathogenicity score and inheritance mode from whole exome sequencing results, a heterozygous mutation (NM_001278939.1: c.1939G>T, p.Gly647Ter) in elastin (ELN) was identified among 6,440 variants in a female proband born with an atrial septal defect accompanied by pulmonary artery stenosis. Results of RT-PCR showed that the mutation (NM_001278939.1: c.1939G>T, p.Gly647Ter) did not affect the expression levels of ELN mRNA but increased protein level. The content of ELN truncate (functional component) was significantly lower in both the intracellular and extracellular compartments after mutation. These results indicate that the ELN mutation (NM_001278939.1: c.1939G>T, p.Gly647Ter) affected the protein truncate, which may be a functional component of ELN and play crucial roles for this pedigree. Here we report of an ELN heterozygous variant associated with congenital heart disease accompanied with pulmonary artery stenosis, which is less common. Based on our results, we speculate that this may be the main molecular mechanism underlying the mutation-led functional changes, and propose that the decrease of ELN protein level may cause this pedigree vascular abnormality, especially pulmonary artery stenosis, and reinforce the view that ELN insufficiency is the primary cause of these vascular lesions. This may be the main molecular mechanism underlying the mutation-led functional changes. Thus, systematic analysis not only enables us to better understand the etiology of this disease but also contributes to clinical and prenatal diagnosis.


2020 ◽  
Vol 6 (1) ◽  
Author(s):  
Akiko Tomita ◽  
Shoko Takada ◽  
Tomoko Fujimoto ◽  
Mitsuo Iwasaki ◽  
Yukio Hayashi

2010 ◽  
Vol 12 (S1) ◽  
Author(s):  
Isra Valverde ◽  
Thomas Krasemann ◽  
Eric Rosenthal ◽  
Tarique Hussain ◽  
Hannah Bellsham-Revell ◽  
...  

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