Peculiarities of ischemic cardiac arrhythmias in cats against the background of stimulation of sensorimotor cortex and administration of selective opiate receptor agonists

2000 ◽  
Vol 129 (5) ◽  
pp. 423-424 ◽  
Author(s):  
S. D. Mikhailova ◽  
N. V. Glushchenko ◽  
T. M. Semushkina ◽  
G. I. Storozhakov
Keyword(s):  
Sensorimotor Cortex ◽  
Cardiac Arrhythmias ◽  
Opiate Receptor ◽  
Receptor Agonists ◽  
Stimulation Of ◽  
Ischemic Cardiac Arrhythmias ◽  
Stimulation Of Sensorimotor Cortex

Effect of opiate receptor blockade by naloxone on development of ischemic cardiac arrhythmias

1989 ◽  
Vol 107 (5) ◽  
pp. 598-600
Author(s):  
G. I. Kositskii ◽  
S. D. Mikhailova ◽  
S. L. Gorozhanin ◽  
T. M. Semushkina
Keyword(s):  
Cardiac Arrhythmias ◽  
Opiate Receptor ◽  
Receptor Blockade ◽  
Ischemic Cardiac Arrhythmias

c-Fos Expression After Chronic Electrical Stimulation of Sensorimotor Cortex in Rats

2008 ◽  
Vol 11 (3) ◽  
pp. 187-195 ◽  
Author(s):  
Katsunori Shijo ◽  
Yoichi Katayama ◽  
Akiko Yamashita ◽  
Kazutaka Kobayashi ◽  
Hideki Oshima ◽  
...  
Keyword(s):  
Electrical Stimulation ◽  
Sensorimotor Cortex ◽  
Chronic Electrical Stimulation ◽  
Fos Expression ◽  
Stimulation Of ◽  
Stimulation Of Sensorimotor Cortex

Effects of stimulation of sensorimotor cortex on primate spinothalamic neurons

1974 ◽  
Vol 65 (2) ◽  
pp. 351-356 ◽  
Author(s):  
Joe Dan Coulter ◽  
Roger A. Maunz ◽  
William D. Willis
Keyword(s):  
Sensorimotor Cortex ◽  
Stimulation Of ◽  
Stimulation Of Sensorimotor Cortex

SPECIFIC STIMULATION OF MIGRATION OF HUMAN KERATINOCYTES BY μ -OPIATE RECEPTOR AGONISTS

2002 ◽  
Vol 22 (1-4) ◽  
pp. 191-199 ◽  
Author(s):  
Paul L. Bigliardi ◽  
Stanislaus Büchner ◽  
Theo Rufli ◽  
Mei Bigliardi-Qi
Keyword(s):  
Human Keratinocytes ◽  
Opiate Receptor ◽  
Receptor Agonists ◽  
Specific Stimulation ◽  
Μ Opiate Receptor ◽  
Stimulation Of

Entrainment and Annihilation of Reentrant Excitation in a Periodically Stimulated Ring of Excitable Media

1997 ◽  
Vol 36 (04/05) ◽  
pp. 290-293
Author(s):  
L. Glass ◽  
T. Nomura
Keyword(s):  
Cardiac Arrhythmias ◽  
Initial Phase ◽  
Human Heart ◽  
Periodic Wave ◽  
Excitable Media ◽  
One Dimensional ◽  
Clinical Observations ◽  
Periodic Stimulation ◽  
Reentrant Excitation ◽  
Stimulation Of

Abstract:Excitable media, such as nerve, heart and the Belousov-Zhabo- tinsky reaction, exhibit a large excursion from equilibrium in response to a small but finite perturbation. Assuming a one-dimensional ring geometry of sufficient length, excitable media support a periodic wave of circulation. As in the periodic stimulation of oscillations in ordinary differential equations, the effects of periodic stimuli of the periodically circulating wave can be described by a one-dimensional Poincaré map. Depending on the period and intensity of the stimulus as well as its initial phase, either entrainment or termination of the original circulating wave is observed. These phenomena are directly related to clinical observations concerning periodic stimulation of a class of cardiac arrhythmias caused by reentrant wave propagation in the human heart.

Human cortical potentials evoked by stimulation of the median nerve. II. Cytoarchitectonic areas generating long-latency activity

1989 ◽  
Vol 62 (3) ◽  
pp. 711-722 ◽  
Author(s):  
T. Allison ◽  
G. McCarthy ◽  
C. C. Wood ◽  
P. D. Williamson ◽  
D. D. Spencer
Keyword(s):  
Spatial Distribution ◽  
Median Nerve ◽  
Somatosensory Cortex ◽  
Sensorimotor Cortex ◽  
Preceding Paper ◽  
Short Latency ◽  
Cortical Surface ◽  
Long Latency ◽  
Area 3B ◽  
Stimulation Of

1. The anatomic generators of human median nerve somatosensory evoked potentials (SEPs) in the 40 to 250-ms latency range were investigated in 54 patients by means of cortical-surface and transcortical recordings obtained during neurosurgery. 2. Contralateral stimulation evoked three groups of SEPs recorded from the hand representation area of sensorimotor cortex: P45-N80-P180, recorded anterior to the central sulcus (CS) and maximal on the precentral gyrus; N45-P80-N180, recorded posterior to the CS and maximal on the postcentral gyrus; and P50-N90-P190, recorded near and on either side of the CS. 3. P45-N80-P180 inverted in polarity to N45-P80-N180 across the CS but was similar in polarity from the cortical surface and white matter in transcortical recordings. These spatial distributions were similar to those of the short-latency P20-N30 and N20-P30 potentials described in the preceding paper, suggesting that these long-latency potentials are generated in area 3b of somatosensory cortex. 4. P50-N90-P190 was largest over the anterior one-half of somatosensory cortex and did not show polarity inversion across the CS. This spatial distribution was similar to that of the short-latency P25-N35 potentials described in the preceding paper and, together with our and Goldring et al. 1970; Stohr and Goldring 1969 transcortical recordings, suggest that these long-latency potentials are generated in area 1 of somatosensory cortex. 5. SEPs of apparently local origin were recorded from several regions of sensorimotor cortex to stimulation of the ipsilateral median nerve. Surface and transcortical recordings suggest that the ipsilateral potentials are generated not in area 3b, but rather in other regions of sensorimotor cortex perhaps including areas 4, 1, 2, and 7. This spatial distribution suggests that the ipsilateral potentials are generated by transcallosal input from the contralateral hemisphere. 6. Recordings from the periSylvian region were characterized by P100 and N100, recorded above and below the Sylvian sulcus (SS) respectively. This distribution suggests a tangential generator located in the upper wall of the SS in the second somatosensory area (SII). In addition, N125 and P200, recorded near and on either side of the SS, suggest a radial generator in a portion of SII located in surface cortex above the SS. 7. In comparison with the short-latency SEPs described in the preceding paper, the long-latency potentials were more variable and were more affected by intraoperative conditions.

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