Beta-adrenergic mechanisms in arterial hemodynamics: A comparison between normotensive and hypertensive rats

1996 ◽  
Vol 3 (4) ◽  
pp. 286-292 ◽  
Author(s):  
Cheng-Tao Hu ◽  
Chia-Yen Wu ◽  
Hsing I. Chen
Keyword(s):  
Hypertensive Rats ◽  
Beta Adrenergic ◽  
Adrenergic Mechanisms ◽  
Arterial Hemodynamics

Beta-Adrenergic Mechanisms in Action Tremor

1975 ◽  
Vol 293 (19) ◽  
pp. 950-953 ◽  
Author(s):  
Robert R. Young ◽  
John H. Growdon ◽  
Bhagwan T. Shahani
Keyword(s):  
Action Tremor ◽  
Beta Adrenergic ◽  
Adrenergic Mechanisms

Reduced vascular beta-adrenergic receptors in deoxycorticosterone-salt hypertensive rats

1980 ◽  
Vol 29 (11) ◽  
pp. 1465-1468 ◽  
Author(s):  
Elizabeth A. Woodcock ◽  
Christine A. Olsson ◽  
Colin I. Johnston
Keyword(s):  
Adrenergic Receptors ◽  
Beta Adrenergic Receptors ◽  
Hypertensive Rats ◽  
Beta Adrenergic

Alpha- and beta-adrenergic mechanisms in the control of vascular capacitance by the carotid sinus baroreflex system

1994 ◽  
Vol 267 (1) ◽  
pp. H201-H210 ◽  
Author(s):  
K. Shigemi ◽  
M. J. Brunner ◽  
A. A. Shoukas
Keyword(s):  
Adrenergic Receptor ◽  
Carotid Sinus ◽  
Venous Pressure ◽  
Systemic Circulation ◽  
Adrenergic System ◽  
Beta Adrenergic ◽  
Vascular Volume ◽  
Adrenergic Mechanisms ◽  
Vascular Capacitance ◽  
Carotid Sinus Baroreflex

We examined the active and passive contributions of the alpha- and beta-adrenergic receptor mechanisms to the changes in systemic vascular capacitance caused by the carotid sinus baroreflex system in anesthetized, vagotomized dogs. The carotid sinuses were isolated from the systemic circulation and perfused with controlled pressures. To determine the changes in vascular capacitance, a constant flow, constant venous pressure cardiopulmonary bypass was used. The changes in unstressed vascular volume were calculated when carotid sinus pressure was reduced from 200 to 50 mmHg without any adrenergic receptor antagonist, with either an alpha- (phentolamine) or a beta- (propranolol) antagonist and then with both. The reflex change in unstressed vascular volume in the systemic circulation (22.6 +/- 9.0 ml/kg without any antagonist) was reduced by 72% with phentolamine, by 35% with propranolol, and by 73% with both antagonists. Our results suggest that the alpha-adrenergic mechanisms contribute significantly to active changes in systemic venous capacity. In addition, the beta-adrenergic system has very little effect on active changes in venous vessels but does contribute to the overall capacity changes by dilating the hepatic outflow resistance when the carotid sinus baroreflex system is activated.

Beta-adrenergic mechanisms in the nasal mucosa vascular bed

1995 ◽  
Vol 252 (5) ◽  
Author(s):  
J.S. Lacroix ◽  
A.M. Kurt ◽  
S. Auberson ◽  
C. Bretton
Keyword(s):  
Nasal Mucosa ◽  
Beta Adrenergic ◽  
Adrenergic Mechanisms ◽  
Vascular Bed

Role of Beta-Adrenergic Mechanisms in Exercise Training-Induced Metabolic Changes in Respiratory and Locomotor Muscle

1995 ◽  
Vol 16 (01) ◽  
pp. 13-18 ◽  
Author(s):  
S. Powers ◽  
M. Wade ◽  
D. Criswell ◽  
R. Herb ◽  
S. Dodd ◽  
...  
Keyword(s):  
Exercise Training ◽  
Metabolic Changes ◽  
Beta Adrenergic ◽  
Adrenergic Mechanisms
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