The blood group substances on gastric mucosal cells of patients with peptic ulcer and gastric carcinoma: A preliminary report

1965 ◽  
Vol 10 (3) ◽  
pp. 211-218 ◽  
Author(s):  
Barbara B. Frank ◽  
Rhoda Little ◽  
Gloria Chen
Keyword(s):  
Peptic Ulcer ◽  
Gastric Carcinoma ◽  
Blood Group ◽  
Preliminary Report ◽  
Gastric Mucosal Cells ◽  
Mucosal Cells ◽  
Blood Group Substances

scholarly journals Peptic ulcer disease: absence of antibodies stimulating the histamine sensitive adenylate cyclase of gastric mucosal cells.

Gut ◽  
1991 ◽  
Vol 32 (6) ◽  
pp. 620-623 ◽  
Author(s):  
P Burman ◽  
S Mardh ◽  
L Loof ◽  
J Naesdal ◽  
F A Karlsson
Keyword(s):  
Peptic Ulcer ◽  
Adenylate Cyclase ◽  
Peptic Ulcer Disease ◽  
Gastric Mucosal Cells ◽  
Ulcer Disease ◽  
Mucosal Cells

Susceptibility to Peptic Ulcer and Blood Group Substances

Digestion ◽  
1969 ◽  
Vol 2 (5) ◽  
pp. 298-303 ◽  
Author(s):  
R. Lambert ◽  
F. Martin
Keyword(s):  
Peptic Ulcer ◽  
Blood Group ◽  
Blood Group Substances

scholarly journals The Relationship Between the Expression of the Epithelial Blood-group Substances and Malignancy: A Preliminary Report

1980 ◽  
Vol 59 (5) ◽  
pp. 831-831 ◽  
Author(s):  
Donald I. George ◽  
Carl T. Hanks ◽  
Richard M. Courtney ◽  
Dennis E. Lopatin
Keyword(s):  
Blood Group ◽  
Preliminary Report ◽  
Blood Group Substances ◽  
The Relationship

The Effect of Psychoanalysis on the Course of Peptic Ulcer: A Preliminary Report

1952 ◽  
Vol 21 (2) ◽  
pp. 185-211 ◽  
Author(s):  
Leonard A. Stine ◽  
A.C. Ivy
Keyword(s):  
Peptic Ulcer ◽  
Preliminary Report

In vitro Action of Gastric Mucosal Lysosomal Enzymes on Intracellular Gastric Glycoproteins

1979 ◽  
Vol 34 (1-2) ◽  
pp. 90-95 ◽  
Author(s):  
Fouad M. Fouad ◽  
D. Waldron-Edward
Keyword(s):  
Cell Wall ◽  
Lysosomal Enzymes ◽  
Gastric Ulceration ◽  
Mucosal Cell ◽  
Gastric Lesions ◽  
Acid Hydrolases ◽  
Gastric Mucosal Cells ◽  
Mucosal Cells ◽  
Specific Increase

Abstract The results show that incubation of gastric mucosal cells from rat at pH ~4.5 or in the presence of aspirin is associated with a specific increase in the activity of some acid-hydrolases. Intracellular glycoproteins, isolated by non-degradative techniques from rat or dog fundic mucosal cells, were found to be potential bio-substrates for these acid-hydrolyses. This may suggest that cleavage of the carbohydrate moieties of the intracellular and mucosal cell wall glycoproteins is a fundamental step in the development of gastric ulceration. A model for gastric lesions is proposed and discussed in the light of the results obtained.

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