LDH isozyme pattern in induced muscle disease (coxsackie group a virus infection)

1967 ◽  
Vol 23 (7) ◽  
pp. 581-582 ◽  
Author(s):  
Sally Kelly ◽  
Arlene Belorit
1961 ◽  
Vol 2 (18) ◽  
pp. 708-711 ◽  
Author(s):  
A. E. Duxbury ◽  
John White ◽  
B. M. Lipscomb ◽  
W. S. Williamson

Cell Reports ◽  
2021 ◽  
Vol 35 (7) ◽  
pp. 109159
Author(s):  
Xiaoyuan Bai ◽  
Wenxian Yang ◽  
Xiaohan Luan ◽  
Huizi Li ◽  
Heqiao Li ◽  
...  

1965 ◽  
Vol 15 (5) ◽  
pp. 668-680 ◽  
Author(s):  
E. Zalan ◽  
A. E. Kelen ◽  
N. A. Labzoffsky

1954 ◽  
Vol 99 (1) ◽  
pp. 21-27 ◽  
Author(s):  
Gilbert Dalldorf ◽  
Rebecca Gifford

Gravid mice become progressively more susceptible to infection with the pancreatic line of Group B-1 Coxsackie virus during the last week of pregnancy. A Group A-8 strain did not have such an effect. The young that survive despite the fact that their mothers are infected with a B-1 strain appear to be normal in the gross and microscopically, to grow at the usual rate, to be free of demonstrable virus, and to be susceptible on challenge with a homologous strain.


PEDIATRICS ◽  
2000 ◽  
Vol 105 (5) ◽  
pp. e60-e60 ◽  
Author(s):  
K. B. Laupland ◽  
H. D. Davies ◽  
D. E. Low ◽  
B. Schwartz ◽  
K. Green ◽  
...  

The Lancet ◽  
1973 ◽  
Vol 301 (7816) ◽  
pp. 1385 ◽  
Author(s):  
Kenneth Vickery
Keyword(s):  
Group A ◽  

2013 ◽  
Vol 209 (2) ◽  
pp. 255-264 ◽  
Author(s):  
Seigo Ueda ◽  
Satoshi Uchiyama ◽  
Tarik Azzi ◽  
Claudine Gysin ◽  
Christoph Berger ◽  
...  

PEDIATRICS ◽  
1953 ◽  
Vol 11 (2) ◽  
pp. 113-119
Author(s):  
LILLIAN P. KRAVIS ◽  
KLAUS HUMMELER ◽  
M. MICHAEL SIGEL ◽  
HAROLD I. LECKS

The causal relationship between certain Coxsackie group A viruses and the disease entity herpangina first reported by Huebner and his associates in 1951 is confirmed. Outbreaks of herpangina in Philadelphia during the summer of 1951 are described. Coxsackie group A viruses of three different immunologic types were isolated from stools or throat swabs, or both, obtained from 17 of the 21 patients involved in the outbreaks. A rise in complement-fixing and neutralizing antibody titers against homologous virus strains during convalescence was demonstrated in 3 patients. Asymptomatic carriers of Coxsackie group A virus are described. Evidence is presented suggesting that two children with herpangina acquired their infection from such a carrier. The criteria used in making a laboratory diagnosis of infection with Coxsackie virus are discussed.


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