Role of epidermal growth factor in peptic ulcer healing

1995 ◽  
Vol 40 (11) ◽  
pp. 2497-2504 ◽  
Author(s):  
A. Calabrò ◽  
S. Milani ◽  
I. Paladini ◽  
B. Orsini ◽  
G. Salvadori ◽  
...  
Keyword(s):  
Peptic Ulcer ◽  
Growth Factor ◽  
Epidermal Growth Factor ◽  
Ulcer Healing ◽  
Epidermal Growth

Interaction of Growth Hormone-Releasing Factor and Somatostatin on Ulcer Healing and Mucosal Growth in Rats: Role of Gastrin and Epidermal Growth Factor

Digestion ◽  
1988 ◽  
Vol 41 (3) ◽  
pp. 121-128 ◽  
Author(s):  
S.J. Konturek ◽  
T. Brzozowski ◽  
A. Dembinski ◽  
Z. Warzecha ◽  
P.K. Konturek ◽  
...  
Keyword(s):  
Growth Hormone ◽  
Growth Factor ◽  
Epidermal Growth Factor ◽  
Ulcer Healing ◽  
Growth Hormone Releasing Factor ◽  
Epidermal Growth ◽  
Mucosal Growth

scholarly journals Role of Low Environmental Temperature in Peptic Ulcer Development

2021 ◽  
Vol 9 (3) ◽  
pp. 193-204
Author(s):  
Serah Funke Ige ◽  
◽  
Bolade S. Olateju ◽  
Victor A. Oladipupo ◽  
Adedayo T. Adekola ◽  
...  
Keyword(s):  
Helicobacter Pylori ◽  
Peptic Ulcer ◽  
Growth Factor ◽  
Epidermal Growth Factor ◽  
Growth Factor Receptor ◽  
Cold Temperature ◽  
Peptic Ulcers ◽  
Epidermal Growth ◽  
H Pylori

Peptic ulcers are described as acid-induced lesions found in the stomach and duodenum caused bythe imbalance between the defending factors of the mucosa and the attacking factors such ashydrochloric acid in gastric juice, with Helicobacter Pylori (H. Pylori) and Non-Steroidal Anti-Inflammatory Drugs NSAIDs. They exhibit seasonal patterns in their occurrence, with higheroccurrence in winter and spring and a low occurrence in summer. Temperature plays a major role intheir occurrence, some of which have resulted in increased morbidity in some number of diseases,such as gastrointestinal bleeding, caused by an increase in air pressure, dry air (relative humidity)occurring from cold air, and also its actions on the protective effect helicobacter pylori in the humanbody. Their actions excite the adrenal gland marrow and the sympathetic nerve, causing rapidsecretion of adrenaline and non-adrenaline, angiotensin II and endothelin, resulting in damage tothe mucosa epithelial, caused by the contraction effect of the adrenal agents on the duodenalmucosa and blood vessel. It causes low expression of Epidermal Growth Factor Receptor (EGFR),Epidermal Growth Factor (EGF), Heat Shock Protein (HSP) 70, Occludin, Nitric Oxide Synthase(NOS), in the gastric mucosa, in extremely cold temperature than those in extremely hottemperature, increasing the gastric acid secretion in extremely cold temperature than in extremelyhot temperature. Therefore, this review aims to give general insight into the role of low temperaturein peptic ulcer development and further consideration in the treatment of peptic ulcer diseases.

The Role of Epidermal Growth Factor Formulation on Stress Ulcer Healing of the Gastric Mucosa

Surgery Today ◽  
2002 ◽  
Vol 32 (10) ◽  
pp. 880-883 ◽  
Author(s):  
K. Gonca Akbulut ◽  
Bilge Gönül ◽  
Ali Türkyilmaz ◽  
Nevin Celebi
Keyword(s):  
Growth Factor ◽  
Gastric Mucosa ◽  
Epidermal Growth Factor ◽  
Ulcer Healing ◽  
Stress Ulcer ◽  
Epidermal Growth

scholarly journals Differential Pathways of Angiotensin II-Induced Extracellularly Regulated Kinase 1/2 Phosphorylation in Specific Cell Types: Role of Heparin-Binding Epidermal Growth Factor

2004 ◽  
Vol 18 (8) ◽  
pp. 2035-2048 ◽  
Author(s):  
Bukhtiar H. Shah ◽  
Akin Yesilkaya ◽  
J. Alberto Olivares-Reyes ◽  
Hung-Dar Chen ◽  
László Hunyady ◽  
...  
Keyword(s):  
Angiotensin Ii ◽  
Growth Factor ◽  
Epidermal Growth Factor ◽  
Cell Types ◽  
Specific Cell ◽  
Heparin Binding ◽  
Epidermal Growth
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