Volume control during periodic changes of blood volume in the alert rat

1984 ◽  
Vol 79 (5) ◽  
pp. 572-578 ◽  
Author(s):  
O. Aziz ◽  
E. Sommer
Keyword(s):  
Blood Volume ◽  
Volume Control ◽  
Alert Rat ◽  
Periodic Changes

Short Time Dialysis with Continuous Blood Volume Control

1990 ◽  
Vol 13 (2) ◽  
pp. 83-86 ◽  
Author(s):  
S. Stiller ◽  
U. Schallenberg ◽  
U. Gladziwa ◽  
E. Ernst ◽  
H. Mann
Keyword(s):  
Blood Volume ◽  
Volume Control ◽  
Short Time ◽  
Blood Volume Control

Blood Volume Control by Biofeedback and Dialysis-Induced Symptomatology

Nephron ◽  
2002 ◽  
Vol 92 (3) ◽  
pp. 605-609 ◽  
Author(s):  
Chantalle Wolkotte ◽  
Daxenos R. Hassell ◽  
Karin Moret ◽  
Paul G. Gerlag ◽  
A. Warmold van den Wall Bake ◽  
...  
Keyword(s):  
Blood Volume ◽  
Volume Control ◽  
Blood Volume Control

Simultaneous Blood Temperature Control and Blood Volume Control Reduces Intradialytic Symptoms

2011 ◽  
Vol 34 (4) ◽  
pp. 357-364 ◽  
Author(s):  
Ljubiša Veljančicć ◽  
Jovan Popović ◽  
Milan Radović ◽  
Peter Ahrenholz ◽  
Wolfgang Ries ◽  
...  
Keyword(s):  
Blood Volume ◽  
Temperature Control ◽  
Volume Control ◽  
Blood Temperature ◽  
Blood Volume Control ◽  
Intradialytic Symptoms

SuO015REDUCING INTRADIALYTIC COMPLICATIONS WITH AUTOMATED TARGET BLOOD VOLUME CONTROL

2019 ◽  
Vol 34 (Supplement_1) ◽  
Author(s):  
Susanne Kron ◽  
Daniel Schneditz ◽  
Til Leimbach ◽  
Joachim Kron
Keyword(s):  
Blood Volume ◽  
Volume Control ◽  
Blood Volume Control

scholarly journals THE PHYSIOLOGICAL RESPONSE OF THE CIRCULATORY SYSTEM TO EXPERIMENTAL ALTERATIONS

1925 ◽  
Vol 42 (5) ◽  
pp. 661-679 ◽  
Author(s):  
Emile Holman ◽  
Claude S. Beck
Keyword(s):  
Blood Pressure ◽  
Blood Volume ◽  
Peripheral Resistance ◽  
Circulatory System ◽  
Blood Stream ◽  
Volume Flow ◽  
Volume Control ◽  
Total Blood ◽  
Full Compensation ◽  
The Right

An abnormal communication, experimentally produced between the right and left ventricles, causes a deflection of part of the blood stream into the shorter pulmonary circuit. Proceeding pari passu with the increase in volume flow of blood through this shorter circuit, there occurs a gradual enlargement of the heart limited to that part of the circulatory system through which the deflected blood passes; namely, the left ventricle, the right ventricle, the pulmonary artery, and the left auricle. There is also a demonstrable hypertrophy of the right and left ventricles, which presumably is the result of the increased effort necessary to propel forward an increased volume flow of blood, since it cannot be attributed to an increased peripheral resistance. Immediately after the production of the defect, the right auricle and aorta become smaller than usual, conforming in size to the decreased volume flow of blood through them. As full compensation for the deflected flow occurs by an increase in total blood volume, they return to their normal size. If full compensation has not occurred they remain smaller than normal (Dog X 11). The changes incident to the establishment of an opening in the septum are entirely dependent upon the size of the defect, and hence, upon the extent of the volume of blood deflected into the shorter circuit. Commensurate with the volume of blood deflected, there is a fall in general blood pressure. If the animal survives the immediate fall in blood pressure, certain compensatory adjustments occur which reestablish a more normal blood pressure: (a) an immediate increase in pulse rate; (b) a gradual increase in total blood mass. The increase in blood volume is directly commensurate with the size of the defect. The pulse returns to a normal rate when complete compensation through an increase in blood volume has been attained. It is suggested that the enlargement of the heart seen clinically in so called "idiopathic hypertrophy," "essential hypertension," and also in certain cases of cardiorenal disease, may be due to an increase in total blood mass following some interference with the mechanism for its control. The seat of this impairment in blood volume control may be: (a) in a chemical alteration in the blood; (b) in a diseased function of the kidneys which may be responsible for a decreased elimination, or for a change in the chemical composition of the blood; or (c) in an abnormal stimulation of the organs producing the cellular elements of the blood.

Impaired Autonomic Blood Pressure and Blood Volume Control in Chronic Renal Failure

2010 ◽  
pp. 291-297
Author(s):  
Guido Grassi ◽  
Raffaella Dell’Oro ◽  
Fosca Quarti-Trevano ◽  
Giuseppe Mancia
Keyword(s):  
Blood Pressure ◽  
Renal Failure ◽  
Chronic Renal Failure ◽  
Blood Volume ◽  
Volume Control ◽  
Blood Volume Control

Hematocrit and Blood Volume Control during Cardiopulmonary Bypass with the Use of Hemofiltration

1984 ◽  
Vol 60 (5) ◽  
pp. 478-480 ◽  
Author(s):  
Peter L. Klineberg ◽  
Chin A. Kam ◽  
David C. Johnson ◽  
Timothy B. Cartmill ◽  
John H. Brown
Keyword(s):  
Cardiopulmonary Bypass ◽  
Blood Volume ◽  
Volume Control ◽  
Blood Volume Control

scholarly journals Blood Volume Monitoring: A Clinical Tool to Guide Ultrafiltration in Volume Control and Optimisation of Intradialytic Blood Pressure

2021 ◽  
pp. 70-78
Author(s):  
Stephen Mahony ◽  
Frank Ward
Keyword(s):  
Blood Volume ◽  
Extracellular Volume ◽  
Volume Overload ◽  
Dry Weight ◽  
Volume Status ◽  
Volume Control ◽  
Current Evidence ◽  
Chronic Haemodialysis ◽  
Intradialytic Hypotension ◽  
Arterial Blood

The importance of extracellular volume control and avoidance of volume overload has been well documented in relation to the management of patients with chronic haemodialysis. Chronic volume overload results in poorly controlled hypertension, increased cardiovascular events, and increased all-cause mortality. Traditional methods of dry weight assessment have relied on clinical assessment to guide volume status. The challenge of achieving the balance between dry weights and preventing intradialytic complications is a formidable one. In order to achieve this, reproducible and sensitive methods are desirable to aid objective quantification of volume status. One such method is by the use of blood volume monitoring, which is achieved by real-time calculation of changes in relative blood volume via a cuvette placed in the arterial blood-line, which can be used to guide ultrafiltration targets during the haemodialysis session. This review article examines the use of blood volume monitoring as a tool to guide ultrafiltration during dialysis and to examine the current evidence to supports its use in assessing dry weight and in preventing intradialytic hypotension events.

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