Renal glycosuria in renal homograft recipients

1984 ◽  
Vol 62 (18) ◽  
pp. 876-884 ◽  
Author(s):  
F. C. Reubi ◽  
A. Montandon ◽  
K. Farina
JAMA ◽  
1966 ◽  
Vol 198 (3) ◽  
pp. 226-230 ◽  
Author(s):  
F. Milgrom

JAMA ◽  
1965 ◽  
Vol 192 (13) ◽  
pp. 1171 ◽  
Author(s):  
Duncan A. McIntosh
Keyword(s):  

1918 ◽  
Vol XXI (6) ◽  
pp. 705
Author(s):  
A. H. BEARD
Keyword(s):  

1917 ◽  
Vol 153 (1) ◽  
pp. 79-80 ◽  
Author(s):  
JOHN R. MURLIN ◽  
WALTER L. NILES
Keyword(s):  

1973 ◽  
Vol 28 (11) ◽  
pp. 821-822
Author(s):  
HAROLD ZAROWITZ ◽  
STANLEY NEWHOUSE
Keyword(s):  

PEDIATRICS ◽  
1957 ◽  
Vol 20 (2) ◽  
pp. 337-357
Author(s):  
Carolyn F. Piel

As indicated in the preceding sections of this review, it seems evident that renal diabetes insipidus, renal glycosuria, "cystinuria" and renal hyperchloremic acidosis are unquestionably renal tubular diseases. Vitamin D resistant rickets has tentatively been placed in the same category although it is recognized that the evidence for this classification is not yet thoroughly convincing. All of the findings of the Fanconi syndrome seem actually to represent a summation of the single tubular diseases, except "cystinosis." Known renal tubular dysfunction fails to explain the cystine-storage disease, cystinosis.


1966 ◽  
Vol 4 (1) ◽  
pp. 116
Author(s):  
A. B. Cosimi ◽  
J. M. Reckler ◽  
W. R. Waddell
Keyword(s):  

Metabolism ◽  
1971 ◽  
Vol 20 (10) ◽  
pp. 968-975 ◽  
Author(s):  
Louis J. Elsas ◽  
Dietrich Busse ◽  
Leon E. Rosenberg

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