Intramuscular pressure changes during and after revascularization of the femoral arteries in humans

1983 ◽  
Vol 7 (5) ◽  
pp. 646-651 ◽  
Author(s):  
J. T. Christenson ◽  
P. Qvarfordt
Keyword(s):  
Intramuscular Pressure ◽  
Femoral Arteries ◽  
Pressure Changes

Application of Consistent Massage-Like Perturbations on Mouse Calves and Monitoring the Resulting Intramuscular Pressure Changes

10.3791/59475 ◽  
2019 ◽  
Author(s):  
Naoyoshi Sakitani ◽  
Takahiro Maekawa ◽  
Kumiko Saitou ◽  
Katsuhiko Suzuki ◽  
Shuhei Murase ◽  
...  
Keyword(s):  
Intramuscular Pressure ◽  
Pressure Changes

INTRAMUSCULAR PRESSURE CHANGES IN SHOCK

1945 ◽  
Vol 143 (1) ◽  
pp. 89-96 ◽  
Author(s):  
W. Kleinberg ◽  
W. W. Swingle ◽  
H. W. Hays
Keyword(s):  
Intramuscular Pressure ◽  
Pressure Changes

Intramuscular pressure changes during contraction in relation to muscle spindles

1965 ◽  
Vol 209 (5) ◽  
pp. 891-899 ◽  
Author(s):  
Charles F. Bridgman ◽  
Earl Eldred
Keyword(s):  
Silicone Oil ◽  
Motor Units ◽  
Muscle Spindles ◽  
Pressure Change ◽  
Displacement Transducer ◽  
Hypodermic Needle ◽  
Intramuscular Pressure ◽  
Low Viscosity ◽  
Single Motor Units ◽  
Pressure Changes

Pressure changes induced by stretch or contraction of the tennuissimus and calf muscles of the cat were studied in relation to postulated mechanism for pressure activation of muscle spindles. Pressure was monitored through tubing filled with low-viscosity silicone oil connecting a hypodermic needle tip to a low-volume displacement transducer. Distribution of changes was detected by moving the needle to different sites in the muscle. Both stretch and total contraction produced essentially uniform pressure increases throughout the muscle. In fractional contractions, including those of single motor units, the center of contraction was marked by strong increases in pressure and the peripheral zones by weaker biphasic and negative changes. Over much of the muscle no change was detectable during contraction of a motor unit. In general the strongest gradient of pressure change seemed directed perpendicular to the axis of the contracting unit. It was concluded that conditions favoring the operation of a pressure mechanism in the spindle might exist in subtotal, but not in total contraction of the muscle.

The Patterns of Simultaneous Intraluminal Pressure Changes in the Human Proximal Small Intestine

1964 ◽  
Vol 47 (3) ◽  
pp. 258-268 ◽  
Author(s):  
Gerald Friedman ◽  
Jerome D. Waye ◽  
Leonard A. Weingarten ◽  
Henry D. Janowitz
Keyword(s):  
Small Intestine ◽  
Intraluminal Pressure ◽  
Proximal Small Intestine ◽  
Pressure Changes

Heart Rate Signal Decomposition

2000 ◽  
Vol 39 (02) ◽  
pp. 200-203
Author(s):  
H. Mizuta ◽  
K. Yana
Keyword(s):  
Blood Pressure ◽  
Heart Rate ◽  
Pressure Change ◽  
Normal Subjects ◽  
Signal Decomposition ◽  
Large Individual ◽  
Heart Rate Fluctuations ◽  
Type Pattern ◽  
Signal Cancellation ◽  
Pressure Changes

Abstract:This paper proposes a method for decomposing heart rate fluctuations into background, respiratory and blood pressure oriented fluctuations. A signal cancellation scheme using the adaptive RLS algorithm has been introduced for canceling respiration and blood pressure oriented changes in the heart rate fluctuations. The computer simulation confirmed the validity of the proposed method. Then, heart rate fluctuations, instantaneous lung volume and blood pressure changes are simultaneously recorded from eight normal subjects aged 20-24 years. It was shown that after signal decomposition, the power spectrum of the heart rate showed a consistent monotonic 1/fa type pattern. The proposed method enables a clear interpretation of heart rate spectrum removing uncertain large individual variations due to the respiration and blood pressure change.

Effect of Ridogrel on Vascular Contractions Gaused by Vasoactive Substances Released during Platelet Activation

1990 ◽  
Vol 64 (01) ◽  
pp. 091-096 ◽  
Author(s):  
W J Janssens ◽  
F J S Cools ◽  
L A M Hoskens ◽  
J M Van Nueten
Keyword(s):  
Smooth Muscle ◽  
Blood Vessel ◽  
Platelet Activation ◽  
Prostaglandin F2α ◽  
Thromboxane A2 ◽  
Femoral Arteries ◽  
Vasoactive Substances ◽  
Caudal Artery ◽  
Isolated Rat ◽  
Rat Platelets

SummaryRidogrel (6.3 × 10−6 to 10−4 M) inhibited contractions of isolated rat caudal arteries and rabbit femoral arteries caused by U-46619. The slope of an Arunlakshana-Schild plot (pA2-value: 3.4 × 10−6 M) on the caudal artery was slightly higher than one (1.14). This effect was maximal within}D min of incubation of the blood vessel with the compound and easily reversible. Ridogrel antagonised contractions of isolated rabbit femoral arteries caused by prostaglandin Fzo2α in the same concentration range. Ridogrel also inhibited contractions induced by aggregating rat platelets on isolated rat caudal arteries (itt the presence of ketanserin 4 × 10−7 M) and on isolated rabbit pulmonary and femoral arteries (in the absence of ketanserin). Ridogrel had no effect on Ca2+-induced contractions in depolarised isolated rabbit femoral arteries, and at 10−4 M antagonised serotonin-induced contractions in this blood vessel. Its effect on serotonin-induced contractions was statistically significant but very small on isolated rat caudal arteries. These observations indicate that ridogrel is an antagonist of prostaglandin endoperoxide/thromboxane A2 and prostaglandin F2α raCeptors on vascular smooth muscle.

Platelet Adhesion in Polytetrafluoroethylene (PTFE) Vascular Grafts In Vivo and the Influence of Increased Intramuscular Pressure-An Experimental Model

1983 ◽  
Vol 50 (04) ◽  
pp. 881-884 ◽  
Author(s):  
J T Christenson ◽  
P Qvarfordt ◽  
S-E Strand ◽  
D Arvidsson ◽  
T Sjöberg ◽  
...  
Keyword(s):  
Blood Flow ◽  
Graft Failure ◽  
Small Diameter ◽  
Graft Material ◽  
Intramuscular Pressure ◽  
Initial Flow ◽  
Early Graft ◽  
Wick Technique ◽  
Early Graft Failure

SummaryThrombogenicity of graft material is involved in early graft failure in small diameter grafts. The frequently seen postoperative swelling of the leg after distal revascularization may cause an increased intramuscular pressure and early graft failure.Pairs of 4 mm polytetrafluoroethylene (PTFE) grafts were implanted. Autologous platelets were labeled with mIn-oxine. Platelet adhesiveness onto the grafts were analyzed from gamma camera images. Intramuscular pressures were measured with wick technique. Blood flow was measured. One graft served as control the other as test graft. Ninety minutes after declamping the i. m. pressure was increased in the test-leg to 30 mmHg, and later to 60 mmHg.In the control-graft platelet uptake increased to a maximum 60 min after declamping. Blood flow and i.m. pressure remained uneffected. The test-grafts were initially similar but when i.m. pressure was increased to 30 mmHg activity in the grafts increased significantly. Blood flow decreased with 12% of initial flow. When i. m. pressure was raised to 60 mmHg platelet uptake continued to increase.An increased intramuscular pressure of 30 mmHg or more significantly increase the amount of platelets adhering onto PTFE grafts, emphasizing the need for measuring intramuscular pressures after lower limb vascular revascularizations.

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