Polymorphism of the major histocompatibility locus in the wild Norway rat

1976 ◽  
Vol 3 (1) ◽  
pp. 193-200 ◽  
Author(s):  
John W. Shonnard ◽  
Donald V. Cramer ◽  
Paul E. Poloskey ◽  
Heinz W. Kunz ◽  
Thomas J. Gill
Keyword(s):  
Major Histocompatibility ◽  
Norway Rat ◽  
Major Histocompatibility Locus ◽  
In The Wild ◽  
Histocompatibility Locus

A High Density Association Study of the Extended Major Histocompatibility Locus in Ulcerative Colitis

2007 ◽  
Vol 123 ◽  
pp. S172-S173
Author(s):  
Philippe Goyette ◽  
Todd Green ◽  
Paul de Bakker ◽  
Daniel Mirel ◽  
Christine Stevens ◽  
...  
Keyword(s):  
Ulcerative Colitis ◽  
Association Study ◽  
High Density ◽  
Major Histocompatibility ◽  
Major Histocompatibility Locus ◽  
Histocompatibility Locus

scholarly journals Distinctive urinary odors governed by the major histocompatibility locus of the mouse.

1981 ◽  
Vol 78 (9) ◽  
pp. 5817-5820 ◽  
Author(s):  
M. Yamaguchi ◽  
K. Yamazaki ◽  
G. K. Beauchamp ◽  
J. Bard ◽  
L. Thomas ◽  
...  
Keyword(s):  
Major Histocompatibility ◽  
Major Histocompatibility Locus ◽  
Histocompatibility Locus

scholarly journals HL-A Frequencies in Patients with Multiple Sclerosis

1974 ◽  
Vol 1 (4) ◽  
pp. 211-213 ◽  
Author(s):  
D.W. Paty ◽  
H. Mervart ◽  
B. Campling ◽  
C.G. Rand ◽  
C.R. Stiller
Keyword(s):  
Multiple Sclerosis ◽  
Autoimmune Disease ◽  
Virus Infection ◽  
Major Histocompatibility ◽  
Histocompatibility Antigens ◽  
Experimental Animals ◽  
Major Histocompatibility Locus ◽  
Slow Virus ◽  
Histocompatibility Locus

SUMMARY:The histocompatibility antigens (HL-A) have been determined in 100 multiple sclerosis (M.S.) patients and 143 randomly selected controls. In the M.S. group there was a statistically significant increase in the frequency of HL-A 7 and W 18 with an insignificant increase in HL-A 3. The variance from normal HL-A patterns in the M.S. population may play some role in establishing the substrate for this disease. Studies in experimental animals have shown that susceptibility to autoimmune disease and to virus infection is linked to the major histocompatibility locus. This has interesting implications for both the “slow virus” and the “autoimmune” theories of the etiology of multiple sclerosis.

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