The influence of diuretic agents on the insulin stimulated glucose metabolism of rat adipose tissue in vitro and in vivo

1971 ◽  
Vol 8 (1) ◽  
pp. 278-287
Author(s):  
Wolfgang Heptner ◽  
Gudrun Reuter ◽  
Hans-Martin Kellner ◽  
Otmar Christ
Keyword(s):  
Adipose Tissue ◽  
Glucose Metabolism ◽  
Adipose Tissue In Vitro ◽  
Diuretic Agents ◽  
Rat Adipose Tissue

Effect of pH and CO2 concentration on glucose metabolism by rat adipose tissue in vitro

1968 ◽  
Vol 215 (3) ◽  
pp. 582-586 ◽  
Author(s):  
WJ Longmore ◽  
BR Landau ◽  
ES Baker ◽  
AB Hastings ◽  
DM Lum ◽  
...  
Keyword(s):  
Adipose Tissue ◽  
Glucose Metabolism ◽  
Co2 Concentration ◽  
Effect Of Ph ◽  
Adipose Tissue In Vitro ◽  
Rat Adipose Tissue

HORMONAL REGULATION OF GLUCOSE METABOLISM IN ADIPOSE TISSUE IN VITRO

1965 ◽  
Vol 131 (1 Adipose Tissu) ◽  
pp. 43-58 ◽  
Author(s):  
Bernard R. Landau ◽  
Joseph Katz ◽  
Glenn E. Bartsch ◽  
Lawrence W. White ◽  
Hollis R. Williams
Keyword(s):  
Adipose Tissue ◽  
Glucose Metabolism ◽  
Hormonal Regulation ◽  
Adipose Tissue In Vitro

Hepatocyte differentiation of mesenchymal stem cells from human adipose tissue in vitro promotes hepatic integration in vivo

Gut ◽  
2008 ◽  
Vol 58 (4) ◽  
pp. 570-581 ◽  
Author(s):  
H Aurich ◽  
M Sgodda ◽  
P Kaltwasser ◽  
M Vetter ◽  
A Weise ◽  
...  
Keyword(s):  
Stem Cells ◽  
Mesenchymal Stem Cells ◽  
Adipose Tissue ◽  
Human Adipose Tissue ◽  
Hepatocyte Differentiation ◽  
Adipose Tissue In Vitro

Effects of hormones on cyclic AMP release from rat adipose tissue in vitro

FEBS Letters ◽  
1974 ◽  
Vol 49 (1) ◽  
pp. 65-69 ◽  
Author(s):  
P. Zumstein ◽  
J. Zapf ◽  
E.R. Froesch
Keyword(s):  
Adipose Tissue ◽  
Cyclic Amp ◽  
Adipose Tissue In Vitro ◽  
Rat Adipose Tissue

Metabolic influences on enzymes of glycogen synthesis and breakdown in adipose tissue

1964 ◽  
Vol 207 (6) ◽  
pp. 1215-1220 ◽  
Author(s):  
Alisa Gutman ◽  
Eleazar Shafrir
Keyword(s):  
Adipose Tissue ◽  
Protein Content ◽  
Glycogen Synthesis ◽  
Total Activity ◽  
Epididymal Adipose Tissue ◽  
Control Value ◽  
Prolonged Fast ◽  
Rat Adipose Tissue

Rat adipose tissue from different body sites was shown to contain uridine diphosphoglucose (UDPG)-transglucosylase activity, which on the basis of protein content was comparable to or higher than that reported for muscle or liver. In epididymal adipose tissue, the activity of UDPG-glycogen transglucosylase and phosphorylase, as well as the content of glycogen per wet weight, decreased with increasing age of the animals in parallel with the decrease of tissue protein content. On prolonged fast the activity of UDPG-glycogen transglucosylase and phosphorylase per milligram protein dropped by 25–50% of the control value. On refeeding, the extent of changes was variable but, in general, at 24 hr control or higher levels of activity were reached and at 48 hr the activities were elevated. The ratio of glucose 6-phosphate independent activity of UDPG-glycogen transglucosylase to total activity was not affected by fasting and refeeding or by the administration of glucose with insulin. In adrenalectomized rats, with high adipose tissue glycogen, no change in UDPG-glycogen transglucosylase was found, whereas the levels of phosphorylase were elevated. Epinephrine in vivo and in vitro did not affect the activity of UDPG-glycogen transglucosylase of adipose tissue.

Some hormonal effects on the metabolism of acetate-i-C14 by rat adipose tissue

1960 ◽  
Vol 198 (3) ◽  
pp. 640-644 ◽  
Author(s):  
Rodney D. Orth ◽  
William D. Odell ◽  
Robert H. Williams
Keyword(s):  
Growth Hormone ◽  
Adipose Tissue ◽  
Oxygen Consumption ◽  
Basic Mechanism ◽  
Tissue Lipid ◽  
Hormonal Effects ◽  
Adipose Tissue In Vitro ◽  
Rat Adipose Tissue

The effects of various hormones on the metabolism of acetate-i-C14 by rat adipose tissue in vitro were investigated. Using an albumin-containing medium, it was found that ACTH, growth hormone, epinephrine and glucagon each caused increased oxygen consumption, a decreased incorporation of acetate into tissue lipid, and an increase in the amount of newly synthesized lipid attached to albumin. The basic mechanism involved in the production of such qualitatively similar effects by each of the hormones studied are unknown.

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