Expression of HLA-DR antigens in inflammatory bowel disease mucosa: Role of intestinal lamina propria mononuclear cell-derived interferon ?

1988 ◽  
Vol 33 (12) ◽  
pp. 1528-1536 ◽  
Author(s):  
Qin Ouyang ◽  
Mounif El-Youssef ◽  
Belinda Yen-Lieberman ◽  
Wanda Sapatnekar ◽  
Kenneth R. Youngman ◽  
...  
Keyword(s):  
Inflammatory Bowel Disease ◽  
Lamina Propria ◽  
Mononuclear Cell ◽  
Bowel Disease ◽  
Intestinal Lamina Propria ◽  
Hla Dr ◽  
Inflammatory Bowel ◽  
Lamina Propria Mononuclear Cell

MadCAM1 Expression in Intestinal Lamina Propria Endothelium Varies among Inflammatory Bowel Disease Patients

2017 ◽  
Vol 152 (5) ◽  
pp. S767-S768
Author(s):  
Tomas Zamborsky ◽  
Katarina Berakova ◽  
Barbora Kadleckova ◽  
Dusan Podmanicky ◽  
Zuzana Zelinkova
Keyword(s):  
Inflammatory Bowel Disease ◽  
Lamina Propria ◽  
Bowel Disease ◽  
Intestinal Lamina Propria ◽  
Inflammatory Bowel

Cytokines in canine inflammatory bowel disease

2013 ◽  
Vol 16 (1) ◽  
pp. 165-171 ◽  
Author(s):  
A. Kołodziejska-Sawerska ◽  
A. Rychlik ◽  
A. Depta ◽  
M. Wdowiak ◽  
M. Nowicki ◽  
...  
Keyword(s):  
Inflammatory Bowel Disease ◽  
Mrna Expression ◽  
Lamina Propria ◽  
Bowel Disease ◽  
Unknown Etiology ◽  
Tnf Α ◽  
Chronic Enteropathies ◽  
Inflammatory Bowel ◽  
Ifn Γ

Abstract Canine inflammatory bowel disease is a group of chronic enteropathies characterized by persistent or recurring gastric symptoms with an unknown etiology which are related to histopathological changes in the mucosa of the small and large bowel in the form of cellular infiltration in the mucosal lamina propria. Recent years have witnessed a growing number of investigations into the role of the immune system and, in particular, cytokines in the development of IBD. In this article, the expression of pro-inflammatory (IL-1, IL-2, IL-5, IL-6, IL-12, IL-18, IFN-γ, TNF-α) and anti-inflammatory cytokines (IL-4, IL-10) was compared in canine patients with IBD based on clinical presentation, breed, lamina propria cell infiltrate and histopathological grade. Only selected studies confirmed higher mRNA expression levels of cytokines IL-2, IL-4, IL-5, IL-12p40, IFN-γ, TNF-α and TGF-β in dogs with IBD in comparison with healthy subjects. GSD were strongly represented in most study populations. Dogs with LPE were characterized by elevated levels of IL-1α, IL-1β, IL-2, IL-5, IL-6, IL-12, TNF-α, TGF-β. The present studies of canine patients with LPC revealed the mRNA expression of cytokines IL-1β, IL-2, IL-4, IL-5, IL-6, IL-10, IL-12p35, IL-12p40, IFN-γ, TNF-α, TGF-β. In the reviewed studies, the progression of IBD was not accompanied by changes in the mRNA expression of IL-1α, IL-1β, IL-2, IL-4, IL-5, IL-6, IL-10, IL-12, IL-18, TNF-α, IFN-γ or TGF-β.

scholarly journals P339 MadCAM1 expression in intestinal lamina propria endothelium varies among inflammatory bowel disease patients

2017 ◽  
Vol 11 (suppl_1) ◽  
pp. S249-S249
Author(s):  
T. Zamborsky ◽  
K. Berakova ◽  
B. Kadleckova ◽  
D. Podmanicky ◽  
Z. Zelinkova
Keyword(s):  
Inflammatory Bowel Disease ◽  
Lamina Propria ◽  
Bowel Disease ◽  
Intestinal Lamina Propria ◽  
Inflammatory Bowel

Increased activation of isolated intestinal lamina propria mononuclear cells in inflammatory bowel disease

1991 ◽  
Vol 101 (4) ◽  
pp. 1020-1030 ◽  
Author(s):  
Stefan Schreiber ◽  
Richard P. MacDermott ◽  
Andreas Raedler ◽  
Ralf Pinnau ◽  
Michael J. Bertovich ◽  
...  
Keyword(s):  
Inflammatory Bowel Disease ◽  
Lamina Propria ◽  
Bowel Disease ◽  
Mononuclear Cells ◽  
Intestinal Lamina Propria ◽  
Lamina Propria Mononuclear Cells ◽  
Inflammatory Bowel

The role of osteopontin (OPN/SSP1) gene variants in inflammatory bowel disease

2009 ◽  
Vol 47 (09) ◽  
Author(s):  
J Glas ◽  
J Seiderer ◽  
HP Török ◽  
B Göke ◽  
T Ochsenkühn ◽  
...  
Keyword(s):  
Inflammatory Bowel Disease ◽  
Bowel Disease ◽  
Gene Variants ◽  
Inflammatory Bowel

Nutrition in inflammatory bowel disease

2009 ◽  
Vol 150 (18) ◽  
pp. 839-845 ◽  
Author(s):  
János Banai
Keyword(s):  
Inflammatory Bowel Disease ◽  
Bowel Disease ◽  
Fast Food ◽  
Optimal Growth ◽  
Developed Countries ◽  
Aetiological Factor ◽  
Artificial Nutrition ◽  
Food Preservatives ◽  
Inflammatory Bowel

Aetiology of inflammatory bowel disease (IBD) is complex and probably multifactorial. Nutrition has been proposed to be an important aetiological factor for development of IBD. Several components of the diet (such as sugar, fat, fibre, fruit and vegetable, protein, fast food, preservatives etc.) were examined as possible causative agents for IBD. According to some researchers infant feeding (breast feeding) may also contribute to the development of IBD. Though the importance of environmental factors is evidenced by the increasing incidence in developed countries and in migrant population in recent decades, the aetiology of IBD remained unclear. There are many theories, but as yet no dietary approaches have been proved to reduce the risk of developing IBD. The role of nutrition in the management of IBD is better understood. The prevention and correction of malnutrition, the provision of macro- and micronutrients and vitamins and the promotion of optimal growth and development of children are key points of nutritional therapy. In active disease, the effective support of energy and nutrients is a very important part of the therapy. Natural and artificial nutrition or the combination of two can be choosen for supporting therapy of IBD. The author summarises the aetiological and therapeutic role of nutrition in IBD.

scholarly journals Tu1789 – Treatment Response to Ustekinumab and Vedolizumab in Inflammatory Bowel Disease: the Predictive Role of Gut Microbiota

2019 ◽  
Vol 156 (6) ◽  
pp. S-1124
Author(s):  
Clara Caenepeel ◽  
Sara Vieira-Silva ◽  
Jorge F. Vázquez-Castellanos ◽  
Bram Verstockt ◽  
Marc Ferrante ◽  
...  
Keyword(s):  
Inflammatory Bowel Disease ◽  
Gut Microbiota ◽  
Treatment Response ◽  
Bowel Disease ◽  
Predictive Role ◽  
Inflammatory Bowel

scholarly journals The role of heme oxygenase and carbon monoxide in inflammatory bowel disease

Redox Report ◽  
2010 ◽  
Vol 15 (5) ◽  
pp. 193-201 ◽  
Author(s):  
Tomohisa Takagi ◽  
Yuji Naito ◽  
Kazuhiko Uchiyama ◽  
Toshikazu Yoshikawa
Keyword(s):  
Inflammatory Bowel Disease ◽  
Carbon Monoxide ◽  
Heme Oxygenase ◽  
Bowel Disease ◽  
Inflammatory Bowel
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