scholarly journals Normal fluctuations and the FKG inequalities

1980 ◽  
Vol 74 (2) ◽  
pp. 119-128 ◽  
Author(s):  
C. M. Newman
Keyword(s):  
Normal Fluctuations ◽  
Fkg Inequalities

scholarly journals The nature of large cosmic-ray bursts

1936 ◽  
Vol 154 (881) ◽  
pp. 223-245 ◽  
Keyword(s):  
Cosmic Rays ◽  
Ionization Chamber ◽  
Cosmic Ray ◽  
Conclusive Evidence ◽  
Recording Apparatus ◽  
Ionization Current ◽  
Continuous Record ◽  
Normal Fluctuations

A continuous record of the ionization current produced by cosmic rays in an ionization chamber shows, if the recording apparatus is sufficiently sensitive, occasional relatively large momentary currents superposed on the normal fluctuations. These large momentary currents are generally called “bursts”. They were first discovered by Hoffmann. It has often been assumed that the bursts are of the same nature as the showers which are observed either in the Wilson chamber or by means of three or more coincidence counters; but so far no conclusive evidence, particularly for the larger bursts, has been reported in support of this assumption.

scholarly journals Intracellular Mg2+regulates ADP phosphorylation and adenine nucleotide synthesis in human erythrocytes

1998 ◽  
Vol 274 (5) ◽  
pp. E920-E927 ◽  
Author(s):  
Sarah Page ◽  
Michael Salem ◽  
Maren R. Laughlin
Keyword(s):  
Adenine Nucleotide ◽  
Phosphoglycerate Kinase ◽  
Pentose Phosphate ◽  
Human Erythrocytes ◽  
Maximal Velocity ◽  
Nucleotide Synthesis ◽  
Ion Concentrations ◽  
Net Flux ◽  
Normal Fluctuations ◽  
Phosphorylation Rate

13C- and31P-NMR were used in methylene blue-treated human erythrocytes to determine the dependence on intracellular Mg2+concentration ([Mg2+]i) of the pentose phosphate pathway (PPP), the glycolytic pathway, and adenine nucleotide synthesis. The PPP flux had an [Mg2+]iat half-maximal velocity ([Mg2+]i,0.5) of 0.02 mM, well below the physiological range (0.2–0.7 mM). Flux through the PPP was reduced at higher [Mg2+]ias flux through phosphofructokinase was increased ([Mg2+]i,0.5= 0.16 mM). [Mg2+]i,0.5of phosphoglycerate kinase flux, which equals net ADP phosphorylation rate, was 0.27 mM, well within the physiological [Mg2+]irange. The rate of adenine nucleotide synthesis from [2-13C]glucose-derived ribose 5-phosphate and exogenous adenine also exhibited dependence on [Mg2+]ibut was not saturable up to 1.6 mM. Therefore, net flux through the PPP and glycolytic pathways in erythrocytes is not strongly dependent on [Mg2+]iat physiological ion concentrations, but both ADP phosphorylation and adenine nucleotide synthesis are likely to be regulated by normal fluctuations in [Mg2+]i.

A simple proof of certain FKG inequalities

1974 ◽  
Vol 15 (7) ◽  
pp. 998-999 ◽  
Author(s):  
James L. Monroe
Keyword(s):  
Simple Proof ◽  
Fkg Inequalities

Normal Fluctuations in Pulmonary Artery Pressures and Cardiac Output in Patients with Severe Left Ventricular Dysfunction

2002 ◽  
Vol 1 (2) ◽  
pp. 131-137 ◽  
Author(s):  
Debra K. Moser ◽  
Susan K. Frazier ◽  
Mary A. Woo ◽  
Linda K. Daley
Keyword(s):  
Cardiac Output ◽  
Pulmonary Artery ◽  
Pulmonary Capillary Wedge Pressure ◽  
Left Ventricular Dysfunction ◽  
Ventricular Dysfunction ◽  
Systolic Pressure ◽  
Left Ventricular ◽  
Pulmonary Artery Systolic Pressure ◽  
Pulmonary Capillary ◽  
Normal Fluctuations

Background: One barrier to accurate interpretation of changes in hemodynamic pressures and cardiac output is lack of data about what constitutes a normal fluctuation. Few investigators have examined normal fluctuations in these parameters and none have done so in patients with left ventricular dysfunction. Aims: To describe normal fluctuations in pulmonary artery pressures and cardiac output in patients with left ventricular dysfunction. Methods: Hemodynamically stable advanced heart failure patients ( N=39; 55±6 years old; 62% male) with left ventricular dysfunction (mean ejection fraction 22±5%) were studied. Cardiac output and pulmonary artery pressures were measured every 15 min for 2 h. Results: Mean±standard deviation fluctuations were as follows: pulmonary artery systolic pressure=7±4 mmHg; pulmonary artery diastolic pressure=6±3 mmHg; pulmonary capillary wedge pressure=5±3 mmHg; cardiac output=0.7±0.3 l/min. The coefficient of variation for fluctuations in pulmonary artery systolic pressure was 6.7%, in pulmonary artery diastolic pressure was 9.3%, in pulmonary capillary wedge pressure was 9.2%, and in cardiac output was 7.2%. Conclusions: Values that vary <8% for pulmonary artery systolic pressure, <11% for pulmonary artery diastolic pressure, <12% for pulmonary capillary wedge pressure, and <9% for cardiac output from baseline represent normal fluctuations in these parameters in patients with left ventricular dysfunction.

Gender differences in endothelium-dependent relaxations do not involve NO in porcine coronary arteries

1997 ◽  
Vol 273 (5) ◽  
pp. H2325-H2332 ◽  
Author(s):  
Dustan A. Barber ◽  
Virginia M. Miller
Keyword(s):  
Gender Differences ◽  
Steroid Hormones ◽  
Coronary Arteries ◽  
Isometric Force ◽  
No Synthase ◽  
Sex Steroid Hormones ◽  
Sex Steroid ◽  
Intact Male ◽  
Endogenous Estrogen ◽  
Normal Fluctuations

Experiments were designed to determine whether normal fluctuations in endogenous sex steroid hormones and/or gender affect endothelium-dependent relaxations in coronary arteries, and, if so, to identify endothelium-derived factors contributing to these differences. Coronary arteries from sexually mature, gonadally intact male and female pigs or ovariectomized pigs were prepared either for study of isometric force in organ chambers or for measurement of prostanoids and activity of nitric oxide (NO) synthase. In organ chamber studies, neither the magnitude nor the sensitivity of endothelium-dependent relaxations correlated with endogenous estrogen or progesterone in female pigs. However, relaxations to bradykinin and UK-14304 were significantly greater and/or shifted leftward in arterial rings from female compared with male pigs. Indomethacin (10−5 mol/l) increased endothelium-dependent relaxations only in arteries from male pigs. N G-monomethyl-l-arginine reduced endothelium-dependent relaxations to a similar extent in coronary arteries from either sex. Neither production nor response to thromboxane A2 or prostacyclin differed in coronary arteries from male compared with female pigs. Activity for calcium-dependent or -independent NO synthase was similar in both sexes. These results suggest that normal fluctuations in endogenous sex steroid hormones do not affect endothelium-dependent relaxations in coronary arteries from female pigs. There are, however, gender differences in endothelium-dependent relaxations that are indomethacin sensitive and may be due to cyclooxygenase products other than thromboxane A2 or prostacyclin.

Normal Fluctuations of Biological Membrane Shape as a Coupling Factor for Ordered Monolayer Domains

2019 ◽  
Vol 13 (3) ◽  
pp. 205-211
Author(s):  
T. R. Galimzyanov ◽  
M. A. Kalutsky ◽  
O. V. Kondrashov ◽  
K. V. Pinigin ◽  
R. J. Molotkovsky ◽  
...  
Keyword(s):  
Biological Membrane ◽  
Coupling Factor ◽  
Normal Fluctuations ◽  
Membrane Shape

scholarly journals RETURN TIME STATISTICS OF INVARIANT MEASURES FOR INTERVAL MAPS WITH POSITIVE LYAPUNOV EXPONENT

2009 ◽  
Vol 09 (01) ◽  
pp. 81-100 ◽  
Author(s):  
HENK BRUIN ◽  
MIKE TODD
Keyword(s):  
Lyapunov Exponent ◽  
Invariant Measures ◽  
Return Time ◽  
Continuous Invariant Measure ◽  
Absolutely Continuous ◽  
Interval Maps ◽  
Absolutely Continuous Invariant Measure ◽  
Normal Fluctuations ◽  
Return Time Statistics ◽  
Absolutely Continuous Invariant

We prove that multimodal maps with an absolutely continuous invariant measure have exponential return time statistics around almost every point. We also show a "polynomial Gibbs property" for these systems, and that the convergence to the entropy in the Ornstein–Weiss formula has normal fluctuations. These results are also proved for equilibrium states of some Hölder potentials.

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