Some characteristics of premature and usual periods of rapid sleep during daytime narcoleptic attacks

1978 ◽  
Vol 9 (3) ◽  
pp. 247-252 ◽  
Author(s):  
M. L. Rait ◽  
L. P. Latash ◽  
N. N. Yakhno ◽  
A. M. Vein
Keyword(s):  
Rapid Sleep

Some indices of brain activity in ?rapid? sleep preceded or not preceded by ?-sleep

1975 ◽  
Vol 79 (4) ◽  
pp. 375-378
Author(s):  
L. P. Latash ◽  
M. L. Rait ◽  
A. M. Vein ◽  
N. N. Yakhno
Keyword(s):  
Brain Activity ◽  
Rapid Sleep

scholarly journals More Rapid Sleep Onset with Lingual-Spray vs Oral-Tablet Delivery Zolpidem

2019 ◽  
Vol 10 (07) ◽  
pp. 329-342
Author(s):  
Robert B. Raffa ◽  
Gerwin Westfield
Keyword(s):  
Sleep Onset ◽  
Oral Tablet ◽  
Rapid Sleep

A Psychoanalytic note on a Specific Delusion of time in Psychotic Depression

1966 ◽  
Vol 11 (1_suppl) ◽  
pp. 221-228
Author(s):  
J. Sarwer-Foner Gerald
Keyword(s):  
Biological Rhythms ◽  
Organ System ◽  
Psychotic Depression ◽  
Depressed Patients ◽  
Depressed State ◽  
The Future ◽  
Psychoanalytically Oriented Psychotherapy ◽  
Rapid Sleep

1) The specific delusion of the eternity of the depressed state presented by psychotic depressed patients in all aspects of their behaviour and thought during their illness, is described and discussed. It consists of the conviction that their depressed state will never end and will continue for an eternity. 2) The hopelessness and helplessness of such patients are directly linked with this delusion (cf. text). 3) The findings of the author, based on psychoanalytically-oriented psychotherapy and psychoanalysis differ from those presented in the existentialist literature (cf. text). Time is neither regularly ‘speeded up’ nor ‘slowed down’, ‘stagnant’ nor ‘dammed up’, nor is the ‘future blocked’ as reported by the existentialist phenomenologists. Rather, patients perceive their future in the deluded conviction that it is an unending depression that will continue unabated forever or for eternity. 4) The author considers the nature of this delusion. For him, the delusion of the permanence of the depressed state results from a marked regression to fixations in the earliest oral phase, in which the passage of time can only be measured by changes in the biological rhythms of the organ system denoting such change. Thus a state continues ‘forever’ until it is succeeded by a new organic state of function, e.g. sleep to wakefulness, feeding to satiation and very rapid sleep, etc. (cf. text). This regression explains the nature and form of the cathexis present in the patient's depressed state. Helplessness is felt to be a marked regression of aggressive cathexis to a late oral (pre-anal) stage—one prior to the development of sphincter control and mastery (cf. text). 5) The author also discusses some aspects of technique in using the above concepts in psychotherapy and psycho-analysis, of this aspect of their illness, of such patients (cf. text). 6) The patients' poor motivation to continue on-going psychotherapy once out of the depressed state, and the specific psychodynamic reasons for this, are discussed (cf. text).

scholarly journals Distal skin vasodilation promotes rapid sleep onset in preterm neonates

2017 ◽  
Vol 26 (5) ◽  
pp. 572-577 ◽  
Author(s):  
Lucile Barcat ◽  
Pauline Decima ◽  
Emilie Bodin ◽  
Stephane Delanaud ◽  
Erwan Stephan-Blanchard ◽  
...  
Keyword(s):  
Sleep Onset ◽  
Preterm Neonates ◽  
Rapid Sleep

scholarly journals Coordinated activity of sleep and arousal neurons for stabilizing sleep/wake states in Drosophila

2018 ◽  
Author(s):  
Jinfei D. Ni ◽  
Tyler H. Ogunmowo ◽  
Hannah Hackbart ◽  
Ahmed Elsheikh ◽  
Adishthi S. Gurav ◽  
...  
Keyword(s):  
Neuronal Activity ◽  
Ex Vivo ◽  
Central Complex ◽  
List Type ◽  
Shaped Body ◽  
Sleep Homeostat ◽  
The Brain ◽  
Rapid Sleep

SummaryThe output arm of the sleep homeostat in Drosophila is a group of neurons with projections to the dorsal fan-shaped body (dFSB) of the central complex in the brain. However, neurons that regulate the sleep homeostat remain poorly understood. Using neurogenetic approaches combined with ex vivo Ca2+ imaging, we identify two groups of sleep-regulatory neurons that modulate the activity of the sleep homeostat in an opposing fashion. The sleep-promoting neurons activate the sleep homeostat with glutamate, whereas the arousal-promoting neurons down-regulate the sleep homeostat’s output with dopamine. Co-activating these two inputs leads to frequent shifts between sleep and wake states. We also show that dFSB sleep homeostat neurons release the neurotransmitter GABA that inhibits octopaminergic arousal neurons. Taken together, we suggest coordinated neuronal activity of sleep- and arousal-promoting neurons is essential for stabilizing sleep/wake states.HighlightsGlutamate released by AstA neurons activates dFSBAstAR1 sleep-promoting neuronsDopamine down-regulates the activity of dFSBAstAR1 neuronsSimultaneous glutamate and dopamine input causes rapid sleep and awake swingsGABA released by dFSBAstAR1 neurons promotes sleep by inhibiting arousal neurons

Hypnotic Efficacy of Triazolam and Methyprylon in Insomniac In-Patients

1976 ◽  
Vol 4 (1) ◽  
pp. 55-58 ◽  
Author(s):  
P Lomen ◽  
O I Linet
Keyword(s):  
Side Effects ◽  
Sleep Onset ◽  
Double Blind ◽  
Hypnotic Effect ◽  
Sleep Parameters ◽  
Hypnotic Efficacy ◽  
Rapid Sleep

The hypnotic effect of a new triazolobenzodiazepine, triazolam ( Halcion®) 0.5 mg and methyprylon 300 mg was compared in twenty oncologic inpatient volunteers with insomnia using the preference technique. On the first night of the two-night trial, triazolam or methyprylon was given on a double-blind basis and on the second night the patients received the alternate medication. Following each trial night the patients were interviewed in regard to their sleep. Of the seventeen patients who completed the study, eleven patients preferred triazolam, three preferred methyprylon and three had no preference ( p = 0.057). Analysis of the various sleep parameters showed that triazolam helped the patients sleep more than methyprylon ( p = 0.013), induced more rapid sleep onset ( p = 0.003), gave a longer duration of sleep ( p = 0.013). The treatment was considered a success if the patient went to sleep in thirty minutes or less and slept for at least six hours. Triazolam was more successful than methyprylon in this respect ( p = 0.012). There were no side-effects reported on either of the drugs.

Brain catecholamines and serotonin in rapid sleep deprivation

1969 ◽  
Vol 4 (4) ◽  
pp. 659-661 ◽  
Author(s):  
Raúl Hernández-Peón ◽  
RenéRaúl Drucker ◽  
Adela Ramírez Del Angel ◽  
Beatriz Chavez ◽  
Pedro Serrano
Keyword(s):  
Sleep Deprivation ◽  
Brain Catecholamines ◽  
Rapid Sleep

LightSleepNet: A Lightweight Deep Model for Rapid Sleep Stage Classification with Spectrograms

2021 ◽  
Author(s):  
Dongdong Zhou ◽  
Qi Xu ◽  
Jian Wang ◽  
Jiacheng Zhang ◽  
Guoqiang Hu ◽  
...  
Keyword(s):  
Sleep Stage ◽  
Deep Model ◽  
Stage Classification ◽  
Sleep Stage Classification ◽  
Rapid Sleep
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