Evidence for an increased rate of choline efflux across erythrocyte membranes in Alzheimer's disease

1985 ◽  
Vol 10 (7) ◽  
pp. 909-918 ◽  
Author(s):  
D. Allan Butterfield ◽  
Maren M. Nicholas ◽  
William R. Markesbery
Keyword(s):  
Alzheimer’S Disease ◽  
Alzheimer's Disease ◽  
Erythrocyte Membranes

Membrane fluidity of platelets and erythrocytes in patients with Alzheimer's disease and the effect of small amounts of aluminium on platelet and erythrocyte membranes

1992 ◽  
Vol 17 (8) ◽  
pp. 825-829 ◽  
Author(s):  
Susan J. van Rensburg ◽  
Machteld E. Carstens ◽  
Felix C. V. Potocnik ◽  
Abraham K. Aucamp ◽  
Joshua J. F. Taljaard ◽  
...  
Keyword(s):  
Alzheimer’S Disease ◽  
Alzheimer's Disease ◽  
Membrane Fluidity ◽  
Erythrocyte Membranes

Spin label and biochemical studies of erythrocyte membranes in Alzheimer's disease

1980 ◽  
Vol 45 (2-3) ◽  
pp. 323-330 ◽  
Author(s):  
William R. Markesbery ◽  
Patrick K. Leung ◽  
D.Allan Butterfield
Keyword(s):  
Alzheimer’S Disease ◽  
Alzheimer's Disease ◽  
Spin Label ◽  
Erythrocyte Membranes ◽  
Biochemical Studies

End products of lipid peroxidation in erythrocyte membranes in Alzheimer's disease

2011 ◽  
Vol 30 (3) ◽  
pp. 205-210 ◽  
Author(s):  
Alice Skoumalová ◽  
Pavla Mádlová ◽  
Eva Topinková
Keyword(s):  
Alzheimer’S Disease ◽  
Alzheimer's Disease ◽  
Lipid Peroxidation ◽  
Erythrocyte Membranes ◽  
End Products

Cognitive control constrains memory attributions

2019 ◽  
Vol 42 ◽  
Author(s):  
Colleen M. Kelley ◽  
Larry L. Jacoby
Keyword(s):  
Alzheimer’S Disease ◽  
Older Adults ◽  
Alzheimer's Disease ◽  
Traumatic Brain Injury ◽  
Brain Injury ◽  
Cognitive Control

Abstract Cognitive control constrains retrieval processing and so restricts what comes to mind as input to the attribution system. We review evidence that older adults, patients with Alzheimer's disease, and people with traumatic brain injury exert less cognitive control during retrieval, and so are susceptible to memory misattributions in the form of dramatic levels of false remembering.

Formation of paired helical filaments in Alzheimer's disease

1984 ◽  
Vol 42 ◽  
pp. 302-303
Author(s):  
J. Metuzals ◽  
D. F. Clapin ◽  
V. Montpetit
Keyword(s):  
Alzheimer’S Disease ◽  
Alzheimer's Disease ◽  
Frontal Lobe ◽  
Giant Axon ◽  
Paired Helical Filaments ◽  
Normal Brain ◽  
Protein Assemblies ◽  
Electron Microscopic ◽  
Helical Symmetry ◽  
Structural Levels

Information on the conformation of paired helical filaments (PHF) and the neurofilamentous (NF) network is essential for an understanding of the mechanisms involved in the formation of the primary lesions of Alzheimer's disease (AD): tangles and plaques. The structural and chemical relationships between the NF and the PHF have to be clarified in order to discover the etiological factors of this disease. We are investigating by stereo electron microscopic and biochemical techniques frontal lobe biopsies from patients with AD and squid giant axon preparations. The helical nature of the lesion in AD is related to pathological alterations of basic properties of the nervous system due to the helical symmetry that exists at all hierarchic structural levels in the normal brain. Because of this helical symmetry of NF protein assemblies and PHF, the employment of structure reconstruction techniques to determine the conformation, particularly the handedness of these structures, is most promising. Figs. 1-3 are frontal lobe biopsies.

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