Mechanism of disturbances of catecholamine excretion with saliva in rats with acute starvation

1991 ◽  
Vol 111 (3) ◽  
pp. 301-302 ◽  
Author(s):  
V. V. Mikhailov ◽  
M. A. Gordeeva ◽  
A. G. Rusanova
Keyword(s):  
Catecholamine Excretion ◽  
Acute Starvation

REM sleep and catecholamine excretion: a study in elite athletes

2001 ◽  
Vol 84 (6) ◽  
pp. 521-526 ◽  
Author(s):  
Nikolaus C. Netzer ◽  
David Kristo ◽  
Hartmut Steinle ◽  
Manfred Lehmann ◽  
Kingman P. Strohl
Keyword(s):  
Rem Sleep ◽  
Elite Athletes ◽  
Catecholamine Excretion

scholarly journals THE SEDATIVE EFFECT OF PREMEDICATION AS MEASURED BY CATECHOLAMINE EXCRETION

1966 ◽  
Vol 38 (10) ◽  
pp. 780-786 ◽  
Author(s):  
LEONARDO R. MARTINEZ ◽  
CHRISTOPHER VON EULER ◽  
OLOF P. NORLANDER
Keyword(s):  
Sedative Effect ◽  
Catecholamine Excretion

Modernization and catecholamine excretion of young Samoan adults

1990 ◽  
Vol 31 (7) ◽  
pp. 729-736 ◽  
Author(s):  
Jay D. Pearson ◽  
Joel M. Hanna ◽  
Maureen H. Fitzgerald ◽  
Paul T. Baker
Keyword(s):  
Catecholamine Excretion

AMPK blocks chromatin activation and consequent R-loop formation to protect genome stability following acute starvation

2021 ◽  
Author(s):  
Bing Sun ◽  
McLean Sherrin ◽  
Richard Roy
Keyword(s):  
Dna Damage ◽  
Genome Stability ◽  
Germ Line ◽  
Critical Role ◽  
Significant Proportion ◽  
Loop Formation ◽  
C Elegans ◽  
Chromatin Activation ◽  
Acute Starvation

Abstract During periods of starvation organisms must modify both gene expression and metabolic pathways to adjust to the energy stress. We previously reported that C. elegans that lack AMPK have transgenerational reproductive defects that result from abnormally elevated H3K4me3 levels in the germ line following recovery from acute starvation1. Here we show that H3K4me3 is dramatically increased at promoters, driving aberrant transcription elongation that results in the accumulation of R-loops in the starved AMPK mutants. DRIP-seq analysis demonstrated that a significant proportion of the genome was affected by R-loop formation with a dramatic expansion in the number of R-loops at numerous loci, most pronounced at the promoter-TSS regions of genes in the starved AMPK mutants. The R-loops are transmissible into subsequent generations, likely contributing to the transgenerational reproductive defects typical of these mutants following starvation. Strikingly, AMPK null germ lines show considerably more RAD-51 foci at sites of R-loop formation, potentially sequestering it from its critical role at meiotic breaks and/or at sites of induced DNA damage. Our study reveals a previously unforeseen role of AMPK in maintaining genome stability following starvation, where in its absence R-loops accumulate, resulting in reproductive compromise and DNA damage hypersensitivity.

URINARY CATECHOLAMINE EXCRETION BY HEALTHY CHILDREN

PEDIATRICS ◽  
1967 ◽  
Vol 39 (2) ◽  
pp. 252-257
Author(s):  
Mary L. Voorhess
Keyword(s):  
Urinary Excretion ◽  
Surface Area ◽  
Body Surface Area ◽  
Body Surface ◽  
Age Groups ◽  
Healthy Children ◽  
Urinary Catecholamine ◽  
Catecholamine Excretion ◽  
Daily Output ◽  
The Mean

There is increase in the daily urinary excretion of dopamine (DA), norepinephrine (NE), epinephrine (E), and 3-methoxy-4-hydroxymandelic acid (VMA) with age. The mean output in micrograms per 24 hours for various age groups is as follows: birth to 1 year-DA 60.9 (± 24.3), NE 10.6 (± 3.4), E 1.3 (± 1.2), VMA 569 (± 309); 1 through 5 years—DA 124.1 (± 40.7), NE (18.8 ± 7.0), E 3.2 (± 2.7), VMA 1348 (± 433); 6 through 15 years—DA 169.3 (± 72.6), NE 37.4 (± 16.6), E 4.8 (± 2.4), VMA 2373 (± 698); over 15 years—DA 249.1 (± 74.9), NE 50.7 (± 15.7), E 7.1 (± 3.3), VMA 3192 (± 699). The studies suggest that the daily output of these compounds in the various age groups is similar when related to body surface area after infancy.

Sign in / Sign up

Export Citation Format

Share Document