The effect of beta-adrenergic blockade on leg blood flow with repeated maximal contractions of the triceps surae muscle group in man

1990 ◽  
Vol 60 (5) ◽  
pp. 360-364 ◽  
Author(s):  
John M. Kowalchuk ◽  
Cliff S. Klein ◽  
Richard L. Hughson
Keyword(s):  
Blood Flow ◽  
Muscle Group ◽  
Triceps Surae ◽  
Adrenergic Blockade ◽  
Beta Adrenergic ◽  
Leg Blood Flow ◽  
Triceps Surae Muscle

scholarly journals Muscle Heating With Megapulse II Shortwave Diathermy and ReBound Diathermy

2013 ◽  
Vol 48 (4) ◽  
pp. 477-482 ◽  
Author(s):  
David O. Draper ◽  
Amanda R. Hawkes ◽  
A. Wayne Johnson ◽  
Mike T. Diede ◽  
Justin H. Rigby
Keyword(s):  
Heat Dissipation ◽  
Subcutaneous Fat ◽  
Muscle Group ◽  
Triceps Surae ◽  
Tissue Temperature ◽  
Measured Temperature ◽  
Temperature Decay ◽  
Triceps Surae Muscle ◽  
Subcutaneous Fat Thickness ◽  
Shortwave Diathermy

Context: A new continuous diathermy called ReBound recently has been introduced. Its effectiveness as a heating modality is unknown. Objective: To compare the effects of the ReBound diathermy with an established deep-heating diathermy, the Megapulse II pulsed shortwave diathermy, on tissue temperature in the human triceps surae muscle. Design:  Crossover study. Setting: University research laboratory. Patients or Other Participants: Participants included 12 healthy, college-aged volunteers (4 men, 8 women; age = 22.2 ± 2.25 years, calf subcutaneous fat thickness = 7.2 ± 1.9 mm). Intervention(s):  Each modality treatment was applied to the triceps surae muscle group of each participant for 30 minutes. After 30 minutes, we removed the modality and recorded temperature decay for 20 minutes. Main Outcome Measure(s): We horizontally inserted an implantable thermocouple into the medial triceps surae muscle to measure intramuscular tissue temperature at 3 cm deep. We measured temperature every 5 minutes during the 30-minute treatment and each minute during the 20-minute temperature decay. Results: Tissue temperature at a depth of 3 cm increased more with Megapulse II than with ReBound diathermy over the course of the treatment (F6,66 = 10.78, P < .001). ReBound diathermy did not produce as much intramuscular heating, leading to a slower heat dissipation rate than the Megapulse II (F20,220 = 28.84, P < .001). Conclusions:  During a 30-minute treatment, the Megapulse II was more effective than ReBound diathermy at increasing deep, intramuscular tissue temperature of the triceps surae muscle group.

LV pressure-volume area and oxygen consumption: evaluation in intact dog by fast CT

1990 ◽  
Vol 258 (4) ◽  
pp. H1208-H1215
Author(s):  
N. Chung ◽  
X. Wu ◽  
K. R. Bailey ◽  
E. L. Ritman
Keyword(s):  
Blood Flow ◽  
Oxygen Consumption ◽  
Myocardial Blood Flow ◽  
Myocardial Oxygen Consumption ◽  
Systolic Pressure ◽  
Left Ventricular ◽  
Adrenergic Blockade ◽  
Ct Scanner ◽  
Beta Adrenergic ◽  
The Relationship

The relationship between left ventricular (LV) myocardial oxygen consumption (MVO2) and LV systolic pressure-volume area (PVA) was investigated in anesthetized closed-chest dogs with intact reflexes and subsequently with beta-adrenergic blockade, with or without simultaneous muscarinic blockade. LV chamber volumes were measured using a fast computerized tomography (CT) scanner (dynamic spatial reconstructor, DSR) at 33-ms intervals. Myocardial blood flow was measured from the DSR scans of aortic root angiograms. With intact reflexes, LV MVO2 (Y) related to PVA (X) values as Y = (4.28 +/- 1.81)X + (1.94 +/- 6.0) (n = 24) (mJ.g-1.cycle-1). With beta-adrenergic blockade, LV MVO2 (Y) related to PVA (X) value as Y = (4.24 +/- 1.03)X - (6.43 +/- 6.5), (n = 9) (mJ.g-1.cycle-1). With beta-adrenergic and muscarinic blockade, LV MVO2 (Y) related to PVA (X) value as Y = (2.84 +/- 1.72)X + (3.51 +/- 5.15), (n = 13) (mJ.g-1.cycle-1). The slopes of these regressions are higher than the slopes demonstrated by others in isolated ventricles but very similar to those demonstrated in open-chest dogs.

Influence of Exchanged Neuromuscular Regulation of M.Gastrocnemius on Leg Bloodflow Regulation During Static Voluntary Contraction of Knee Flexors

2015 ◽  
Vol 6 (2) ◽  
pp. 14-20
Author(s):  
Una Gavrona ◽  
Alvis Paeglītis ◽  
Normunds Vārpa ◽  
Galeja Zinta ◽  
Dzērve Vilnis ◽  
...  
Keyword(s):  
Blood Flow ◽  
Flow Regulation ◽  
Voluntary Contraction ◽  
Triceps Surae ◽  
Local Blood Flow ◽  
Leg Blood Flow ◽  
Blood Flow Regulation ◽  
Static Contraction ◽  
Blood Flow Redistribution ◽  
Knee Flexors

Abstract In literature from 1970th are described experimentaly proved concept of muscle local blood flow redistribution to capillaries feeding active muscle fibres, and loss of this synhrone blood flow redistribution with changes of recruited motor units, caused by mechanical, metabolic or neural regulatory disturbances. On the other hand, from theory of applied kinesiology (AK) it is known that different external or internal challenges coud exchenge neuromuscular regulation and muscle could become weak-testing or become hypertonic. The physiological background of these changes is not jet known. The aim of this study is to find out connections between muscle's neuromuscular regulation and its local blood flow regulation. We investigate blood flow regulation in the leg during static voluntary contraction with contraction force 5% of the knee flexor muscle maximal static voluntary contraction (MVC) in position with knee flexed till 60 deg. In this experiment participate 8 LASE students in age 22-24 with normotonic m. gastrocnemius and 8 LASE students in age 22-26 with weak-tested m. gastrocnemius. Muscle tests were performed according AK testing methode (Frost, 2002.). Leg blood flow was measured with a mercury-in-silastic strain-gauge plethismograph, using the venous-occlusion plethismography Hokanson AI6 (Hokanson, Sumner and Strandness, 1975). Cycle of leg blood flow measurement was 15 seconds repeated twice in every controled minute. Student paired t-test was used to evaluate diferences in the results of blood flow measurements from strong tested and weak tested leg muscles. We find statistically significant (P>95) differences triceps surae during static contraction with contracting force of knee flexors 5% MVC. Volume blood flow in leg with exchanged neuromuscular regulation of knee flexors where statisticaly higher neither blood floww in leg with normal neuromuscuular regulation. These differences was observed all the time of the static contraction. Changes of neuromuscular regulation could cause muscles local blood flow regulatory disturbances. Character of muscle local blood flow could be a quantitatively measurable parameter which reflects changes of neuromuscular regulatory processes of studied muscle.

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