The intramuscular nerve endings in muscular dystrophy

1965 ◽  
Vol 5 (2) ◽  
pp. 225-242 ◽  
Author(s):  
Hanna Jędrzejowska ◽  
A. G. Johnson ◽  
A. L. Woolf
Brain ◽  
1960 ◽  
Vol 83 (1) ◽  
pp. 10-23 ◽  
Author(s):  
E. R. BICKERSTAFF ◽  
A. L. WOOLF

1931 ◽  
Vol 54 (2) ◽  
pp. 167-169 ◽  
Author(s):  
W. M. Rogers ◽  
A. M. Pappenheimer ◽  
Marianne Goettsch

The nutritional muscle dystrophy of guinea pigs and rabbits is primarily a degeneration of the muscle fibers and is not associated with visible alterations of the peripheral nerves or their motor terminals.


1956 ◽  
Vol 71 (1) ◽  
pp. 173-178 ◽  
Author(s):  
A. L. Woolf ◽  
H. J. Bagnall ◽  
P. Bauwens ◽  
Edwin R. Bickerstaff

1967 ◽  
Vol 45 (5) ◽  
pp. 821-831 ◽  
Author(s):  
G. Beaulieu ◽  
G. B. Frank

The mechanism for the production of the 'twitching' response induced by tetraethylammonium (TEA) of the frog's sartorius muscle was investigated. A linear relation was found between the log TEA concentration and the log of the latent period for the onset of the muscle response. The minimum latent period was 5 to 10 sec. Previously it was suggested that TEA caused the release of acetylcholine from intramuscular nerve endings to produce this response (13). The findings that blocking neuromuscular transmission by d-tubocurarine, by reduced extracellular calcium concentrations, or by repetitive indirect stimulation in the presence of hemicholinium also prevented the TEA-induced muscle 'twitching' response support the above suggestion. Exposing the preparation to procaine concentrations too low to block neuromuscular transmission blocked the TEA-induced response, indicating that this response was due to an effect of TEA on the unmyelinated nerve endings.


2010 ◽  
Vol 32 (6) ◽  
pp. 529-537 ◽  
Author(s):  
X. C. An ◽  
J. H. Lee ◽  
S. Im ◽  
M. S. Lee ◽  
K. Hwang ◽  
...  

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