Na transport compartment in rabbit urinary bladder

1988 ◽  
Vol 411 (6) ◽  
pp. 681-687 ◽  
Author(s):  
Adolf D�rge ◽  
Peter Wienecke ◽  
Franz Beck ◽  
Brigitte W�rndl ◽  
Roger Rick ◽  
...  
Keyword(s):  
Urinary Bladder ◽  
Na Transport ◽  
Rabbit Urinary Bladder

Aldosterone stimulates active Na+ transport in rabbit urinary bladder by both genomic and non-genomic processes

2005 ◽  
Vol 510 (3) ◽  
pp. 181-186 ◽  
Author(s):  
Timothy J. Burton ◽  
Dermot M.F. Cooper ◽  
Bryony Dunning-Davies ◽  
Dina Mansour ◽  
Nanako Masada ◽  
...  
Keyword(s):  
Urinary Bladder ◽  
Na Transport ◽  
Rabbit Urinary Bladder

Effects of serosal anion composition on the permeability properties of rabbit urinary bladder

1989 ◽  
Vol 256 (6) ◽  
pp. F1125-F1134
Author(s):  
P. J. Donaldson ◽  
L. K. Chen ◽  
S. A. Lewis
Keyword(s):  
Urinary Bladder ◽  
Basolateral Membrane ◽  
Anion Channel ◽  
Membrane Properties ◽  
Lag Phase ◽  
Na Transport ◽  
Transepithelial Na Transport ◽  
Bladder Replacement ◽  
Lag Period ◽  
Rabbit Urinary Bladder

This study describes the effects of serosal Cl- and HCO3- substitution on transepithelial Na+ transport and basolateral membrane properties of the rabbit urinary bladder. Replacement of Cl- with NO3-, SCN-, and Br- had no effect on transepithelial Na+ transport or the basolateral membrane potential (Vbl). However, gluconate, isethionate, and cyclamate (anions that were shown previously to be not as permeable as Cl- through the basolateral membrane anion channel), decreased transepithelial Na+ transport and depolarized Vbl. Replacement of HCO3- also produced a decrease in transepithelial Na+ transport and a depolarization of Vbl. Utilizing conventional and K+-specific microelectrodes, we found the depolarization to be due to a reduction in basolateral K+ conductance. This depolarization was reversible only when Cl- was returned to the serosal solution, the normally permeant anion NO3- being unable to affect repolarization, suggesting that both the K+ and Cl- conductance are reduced during depolarization. A lag period of some 4 min preceded the repolarization of Vbl. The Na+-H+ exchange blocker amiloride prolonged the lag phase associated with repolarization, whereas niflumic acid, a Cl-(-)HCO3- exchange blocker (in red blood cells) reduced the magnitude of Vbl repolarization. Because of the possible involvement of the exchangers it is believed that the lag phase represents a volume-dependent and/or pH-dependent reactivation of the basolateral membrane conductances.

The mechanism of Na+ transport by rabbit urinary bladder

1976 ◽  
Vol 28 (1) ◽  
pp. 41-70 ◽  
Author(s):  
Simon A. Lewis ◽  
Douglas C. Eaton ◽  
Jared M. Diamond
Keyword(s):  
Urinary Bladder ◽  
Na Transport ◽  
Rabbit Urinary Bladder

Cyclical estrogen and free radical damage to the rabbit urinary bladder

2009 ◽  
Vol 21 (4) ◽  
pp. 489-494 ◽  
Author(s):  
Alexandra Rehfuss ◽  
Catherine Schuler ◽  
Christina Maxemous ◽  
Robert E. Leggett ◽  
Robert M. Levin
Keyword(s):  
Free Radical ◽  
Urinary Bladder ◽  
Free Radical Damage ◽  
Radical Damage ◽  
Rabbit Urinary Bladder
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