Body increases during molt and molting cycle of the oriental brown shrimp Penaeus japonicus

1971 ◽  
Vol 9 (1) ◽  
pp. 31-37 ◽  
Author(s):  
S. Choe
Keyword(s):  
Brown Shrimp ◽  
Molting Cycle ◽  
Penaeus Japonicus

Variation in lipid classes during the molting cycle of the prawn Penaeus japonicus

1977 ◽  
Vol 39 (2) ◽  
pp. 129-136 ◽  
Author(s):  
S. Teshima ◽  
A. Kanazawa ◽  
H. Okamoto
Keyword(s):  
Lipid Classes ◽  
Molting Cycle ◽  
Penaeus Japonicus

scholarly journals The variation of lipids and cholesterol contents in the tissues of prawn, Penaeus japonicus, during the molting cycle.

1976 ◽  
Vol 42 (9) ◽  
pp. 1003-1007 ◽  
Author(s):  
Akio KANAZAWA ◽  
Shin-ichi TESHIMA ◽  
Yoshinobu SAKAMOTO ◽  
Jean-Claude B.GUARY
Keyword(s):  
Molting Cycle ◽  
Penaeus Japonicus

scholarly journals Effect of various levels of squid protein on growth and some biochemical parameters of Penaeus japonicus juveniles.

1987 ◽  
Vol 53 (11) ◽  
pp. 2083-2088 ◽  
Author(s):  
L. E. Cruz-Suarez ◽  
J. Guillaume ◽  
A.Van Wormhoudt
Keyword(s):  
Biochemical Parameters ◽  
Penaeus Japonicus

scholarly journals Identification of Pathways and Key Genes in Venous Remodeling After Arteriovenous Fistula by Bioinformatics Analysis

2020 ◽  
Vol 11 ◽  
Author(s):  
Kong Jie ◽  
Wang Feng ◽  
Zhao Boxiang ◽  
Gong Maofeng ◽  
Zhang Jianbin ◽  
...  
Keyword(s):  
Arteriovenous Fistula ◽  
Pathway Analysis ◽  
Kegg Pathway ◽  
Function Analysis ◽  
Gene Set Enrichment Analysis ◽  
Control Group ◽  
Hub Genes ◽  
Molting Cycle ◽  
Kegg Pathway Analysis ◽  
First Choice

The arteriovenous fistula (AVF) is the first choice for vascular access for hemodialysis of renal failure patients. Venous remodeling after exposure to high fistula flow is important for AVF to mature but the mechanism underlying remodeling is still unknown. The objective of this study is to identify the molecular mechanisms that contribute to venous remodeling after AVF. To screen and identify the differentially expressed genes (DEGs) that may involve venous remodeling after AVF, we used bioinformatics to download the public microarray data (GSE39488) from the Gene Expression Omnibus (GEO) and screen for DEGs. We then performed gene ontology (GO) function analysis, Kyoto Encyclopedia of Genes and Genomes (KEGG) pathway analysis, and gene set enrichment analysis (GSEA) for the functional annotation of DEGs. The protein-protein interaction (PPI) network was constructed and the hub genes were carried out. Finally, we harvested 12 normal vein samples and 12 AVF vein samples which were used to confirm the expressions of the hub genes by immunohistochemistry. A total of 45 DEGs were detected, including 32 upregulated and 13 downregulated DEGs. The biological process (BP) of the GO analysis were enriched in the extrinsic apoptotic signaling pathway, cGMP-mediated pathway signaling, and molting cycle. The KEGG pathway analysis showed that the upregulated DEGs were enriched in glycosaminoglycan biosynthesis and purine metabolism, while the downregulated DEGs were mainly enriched in pathways of glycosaminoglycan biosynthesis, antifolate resistance, and ABC transporters. The GSEA analysis result showed that the top three involved pathways were oxidative phosphorylation, TNFA signaling via NF-K B, and the inflammatory response. The PPI was constructed and the hub genes found through the method of DMNC showed that INHBA and NR4A2 might play an important role in venous remodeling after AVF. The integrated optical density (DOI) examined by immunohistochemistry staining showed that the expression of both INHBA and NR4A2 increased in AVF compared to the control group. Our research contributes to the understanding of the molecular mechanism of venous remodeling after exposure to high fistula flow, which may be useful in treating AVF failure.

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