Conversion of the rabbit gracilis muscle for transposition as a neoanal sphincter by electrical stimulation

Surgery Today ◽  
1995 ◽  
Vol 25 (3) ◽  
pp. 233-236 ◽  
Author(s):  
Tomoo Shatari ◽  
Tatsuo Teramoto ◽  
Masaki Kitajima ◽  
Haruyuki Minamitani
1993 ◽  
Vol 80 (10) ◽  
pp. 1342-1346 ◽  
Author(s):  
B. D. George ◽  
N. S. Williams ◽  
J. Patel ◽  
M. Swash ◽  
E. S. Watkinst

ASAIO Journal ◽  
1993 ◽  
Vol 39 (3) ◽  
pp. M486-M488 ◽  
Author(s):  
Tomoo Shatari ◽  
Tatsuo Teramoto ◽  
Masahiko Watanabe ◽  
Masaki Kitajima ◽  
Haruyuki Minamitani

ASAIO Journal ◽  
1993 ◽  
Vol 39 (3) ◽  
pp. M486-M488 ◽  
Author(s):  
TOMOO SHATARI ◽  
TATSUO TERAMOTO ◽  
MASAHIKO WATANABE ◽  
MASAKI KlTAJIMA ◽  
HARUYUKI MlNAMITANl

1965 ◽  
Vol 208 (3) ◽  
pp. 417-424 ◽  
Author(s):  
Lawrence D. Dorr ◽  
Michael J. Brody

The hypothesis that sympathetic innervation to skeletal muscle vasculature contains functionally distinct adrenergic and cholinergic fibers was investigated utilizing the dog isolated perfused gracilis muscle. The use of hemicholinium in an attempt to abolish cholinergic dilatation, but not adrenergic constriction, in response to intermittent nerve stimulation was not successful. Continuous nerve stimulation produced vasoconstriction which was maintained for the duration of stimulation. Conversely, when cholinergic vasodilatation was unmasked, continuous stimulation resulted in a dilator response which disappeared rapidly. These experiments suggested that release of the adrenergic transmitter could not be dependent upon an intermediate cholinergic link in the sympathetic nerve. This postulate was supported further by experiments utilizing electrical stimulation of medullary vasoconstrictor areas. Whereas cholinergic vasodilatation was unmasked routinely by peripheral stimulation following reserpine, guanethidine or ß-TM 10, this response was never seen when medullary vasoconstrictor neurons were activated following these agents. It was concluded that sympathetic cholinergic nerves to skeletal muscle vessels possess a purely vasodilator function, and do not play an intermediate role involving release of the adrenergic transmitter.


1997 ◽  
Vol 15 (5) ◽  
pp. 320-328 ◽  
Author(s):  
Michael B. Chancellor ◽  
John P. F. A. Heesakkers ◽  
Rudi A. Janknegt

Author(s):  
I. Taylor ◽  
P. Ingram ◽  
J.R. Sommer

In studying quick-frozen single intact skeletal muscle fibers for structural and microchemical alterations that occur milliseconds, and fractions thereof, after electrical stimulation, we have developed a method to compare, directly, ice crystal formation in freeze-substituted thin sections adjacent to all, and beneath the last, freeze-dried cryosections. We have observed images in the cryosections that to our knowledge have not been published heretofore (Figs.1-4). The main features are that isolated, sometimes large regions of the sections appear hazy and have much less contrast than adjacent regions. Sometimes within the hazy regions there are smaller areas that appear crinkled and have much more contrast. We have also observed that while the hazy areas remain still, the regions of higher contrast visibly contract in the beam, often causing tears in the sections that are clearly not caused by ice crystals (Fig.3, arrows).


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