scholarly journals The prevalence of diabetic impotence

Diabetologia ◽  
1980 ◽  
Vol 18 (4) ◽  
Author(s):  
D.K. McCulloch ◽  
I.W. Campbell ◽  
F.C. Wu ◽  
R.J. Prescott ◽  
B.F. Clarke
Keyword(s):  
Diabetic Impotence

Permanent Diabetic Impotence

BMJ ◽  
1962 ◽  
Vol 1 (5276) ◽  
pp. 487-487 ◽  
Author(s):  
H. Ayad
Keyword(s):  
Diabetic Impotence

Trial of Pentoxifylline for Diabetic Impotence

Diabetes Care ◽  
1995 ◽  
Vol 18 (3) ◽  
pp. 345-352 ◽  
Author(s):  
W. J. Georgitis ◽  
J. A. Merenich
Keyword(s):  
Diabetic Impotence

scholarly journals Diabetic impotence.

BMJ ◽  
1980 ◽  
Vol 280 (6212) ◽  
pp. 483-483
Author(s):  
J Bancroft ◽  
F C Wu
Keyword(s):  
Diabetic Impotence

Sexual dysfunction in the diabetic BB/WOR rat: a role of central neuropathy

1997 ◽  
Vol 272 (1) ◽  
pp. R259-R267 ◽  
Author(s):  
K. T. McVary ◽  
C. H. Rathnau ◽  
K. E. McKenna
Keyword(s):  
Sexual Function ◽  
Diabetic Rats ◽  
Nerve Fibers ◽  
Diabetic Rat ◽  
Penile Erections ◽  
Reflex Testing ◽  
Diabetic Impotence ◽  
Central Neuropathy ◽  
Sexual Reflexes

The pathophysiological mechanisms of diabetic impotence remain obscure. We have presented an analysis of sexual function in a diabetic rat (BB/WOR) model characterized by diffuse neuropathic changes without a confounding vasculopathy that allows us to define the neural components of erectile failure. Copulatory behavioral testing demonstrated that diabetic males were severely impaired: the controls mounted three times more than the diabetics and had about one-half the latency to first mount. The diabetics had about one-fourth the number of intromissions and took nearly twice as long to achieve first ejaculation. The number of ejaculations was drastically reduced as well. We examined sexual reflexes in the anesthetized acutely spinalized rat. These experiments tested the integrity of spinal circuits controlling sexual function. Reflex testing demonstrated that spinal sexual reflexes were also severely impaired: the onset latency of reflexes was more than doubled, and the duration of reflexes was less than one-half. More than one-half of the diabetic rats showed no penile erections. Neural studies showed even more derangement in reflex measures in rats, without erection. Nerve conduction velocity experiments suggested a peripheral neuropathic change in hypogastric nerve and motor pudendal nerve fibers. These dysfunctional findings were seen without any androgen deficiency. These results indicate that diabetic impotence in this model reflects central and peripheral neuropathic disease processes.

The Aldose Reductase Inhibitor in the Treatment of Diabetic Impotence

1987 ◽  
Vol 137 (6) ◽  
Author(s):  
Culley C. Carson ◽  
Antoinette E. Porretta ◽  
Steven H. Herman ◽  
Rudy T. Andriani ◽  
Mark R. Feinglos
Keyword(s):  
Aldose Reductase ◽  
Reductase Inhibitor ◽  
Aldose Reductase Inhibitor ◽  
Diabetic Impotence

The Value of Testing for Unmyelinated Fibre, Sensory Neuropathy in Diabetic Impotence

1988 ◽  
Vol 61 (1) ◽  
pp. 63-67 ◽  
Author(s):  
CLARE J. FOWLER ◽  
Z. ALI ◽  
R. S. KIRBY ◽  
J. P. PRYOR
Keyword(s):  
Sensory Neuropathy ◽  
Unmyelinated Fibre ◽  
Diabetic Impotence

Glycosylation may be an Important pathogenic mechanism in diabetic impotence.

1995 ◽  
Vol 86 (3) ◽  
pp. 564
Author(s):  
Nam-Wee Kour ◽  
Michitaka Yajima ◽  
Shang-Sen Lee ◽  
Tom F. Lue
Keyword(s):  
Pathogenic Mechanism ◽  
Diabetic Impotence
Sign in / Sign up

Export Citation Format

Share Document