Selective impairment of express saccade generation in patients with schizophrenia

1993 ◽  
Vol 97 (2) ◽  
Author(s):  
Jon Currie ◽  
Sarah Joyce ◽  
Paul Maruff ◽  
Ben Ramsden ◽  
Cheryl McArthur-Jackson ◽  
...  
2004 ◽  
Vol 21 (2) ◽  
pp. 119-127 ◽  
Author(s):  
PETER H. SCHILLER ◽  
JOHANNES HAUSHOFER ◽  
GEOFFERY KENDALL

The frequency with which express saccades are generated under a variety of conditions in rhesus monkeys was examined. Increasing the gap time between fixation spot termination and target onset increased express saccade frequency but was progressively less effective in doing so as the number of target positions in the sample was increased. Express saccades were rarely produced when two targets were presented simultaneously and the choice of either of which was rewarded; a temporal asynchrony of only 17 ms between the targets reinstated express saccade generation. Express saccades continued to be generated when the vergence or pursuit systems was coactivated with the saccadic system.


2010 ◽  
Vol 10 (7) ◽  
pp. 505-505
Author(s):  
P. Schiller ◽  
M. Kwak

2020 ◽  
Vol 123 (5) ◽  
pp. 1907-1919 ◽  
Author(s):  
Suryadeep Dash ◽  
Tyler R. Peel ◽  
Stephen G. Lomber ◽  
Brian D. Corneil

Express saccades are the shortest-latency saccade. The frontal eye fields (FEF) are thought to promote express saccades by presetting the superior colliculus. Here, by reversibly inactivating the FEF either unilaterally or bilaterally via cortical cooling, we support this by showing that the FEF plays a facilitative but not critical role in express saccade generation. We also found that FEF inactivation lowered express saccade peak velocity, emphasizing a contribution of the FEF to express saccade kinematics.


1992 ◽  
Vol 32 (1) ◽  
pp. 125-127 ◽  
Author(s):  
Sammanda Ramamoorthy ◽  
Frederick H Leibach ◽  
Virendra B Mahesh ◽  
Vadivel Ganapathy

Neurocase ◽  
1999 ◽  
Vol 5 (5) ◽  
pp. 447-458 ◽  
Author(s):  
Catherine Dumont ◽  
Bernadette Ska ◽  
Alessandra Schiavetto

HAND ◽  
1978 ◽  
Vol os-10 (1) ◽  
pp. 77-81 ◽  
Author(s):  
A. R. M. Upton ◽  
J. Darracott ◽  
F. A. Bianchi

Summary A loss of functional motor axons in the median and ulnar nerves occurred in half of thirty-three patients with rheumatoid arthritis. Weakness of small hand muscles may predispose to the development of ulnar deviation of the fingers in patients with joint disease at the radio-ulnar and metacarpophalangeal joints. There is no evidence that spasm of small hand muscles is a significant cause of ulnar deviation of the fingers in rheumatoid arthritis. Ulnar deviation of the fingers in rheumatoid arthritis is not due to selective impairment of the ulnar nerve or the deep palmar branch of the ulnar nerve even though ulnar deviation of the fingers can occur in association with such lesions and in the absence of joint disease.


1995 ◽  
Vol 27 (2) ◽  
pp. 213-236 ◽  
Author(s):  
S.Z. Rapcsak ◽  
C. Ochipa ◽  
K.C. Anderson ◽  
H. Poizner

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