Labeling of the centromeric region on human chromosome 8 by in situ hybridization

1991 ◽  
Vol 87 (4) ◽  
Author(s):  
Heinz-UlrichG. Weier ◽  
JoeW. Gray ◽  
Hans-Dieter Kleine
2004 ◽  
Vol 171 (4S) ◽  
pp. 156-156
Author(s):  
Chandler D. Dora ◽  
Yasushi Kondo ◽  
Fusheng X. Lan ◽  
Jeffrey M. Slezak ◽  
Erik J. Bergstralh ◽  
...  

1995 ◽  
Vol 71 (3) ◽  
pp. 211-211 ◽  
Author(s):  
E.M.C. Jones ◽  
A. Fernald ◽  
G.I. Bell ◽  
M.M. LeBeau

Genomics ◽  
1994 ◽  
Vol 24 (2) ◽  
pp. 413-414 ◽  
Author(s):  
Xiao-Xiang Zhang ◽  
Rima Rozen ◽  
Matthias A. Hediger ◽  
Paul Goodyer ◽  
Patrice Eydoux

1998 ◽  
Vol 82 (1-2) ◽  
pp. 105-106 ◽  
Author(s):  
T. Okamoto ◽  
T. Ono ◽  
M. Hori ◽  
J.-P. Yang ◽  
T. Tetsuka ◽  
...  

Genomics ◽  
1992 ◽  
Vol 14 (3) ◽  
pp. 740-744 ◽  
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Susanne Schnittger ◽  
V.V.N. Gopal Rao ◽  
Urban Deutsch ◽  
Peter Gruss ◽  
Rudi Balling ◽  
...  

1994 ◽  
Vol 297 (3) ◽  
pp. 441-445 ◽  
Author(s):  
D Hickman ◽  
A Risch ◽  
V Buckle ◽  
N K Spurr ◽  
S J Jeremiah ◽  
...  

Arylamine N-acetyltransferase is encoded at two loci, AAC-1 and AAC-2, on human chromosome 8. The products of the two loci are able to catalyse N-acetylation of arylamine carcinogens, such as benzidine and other xenobiotics. AAC-2 is polymorphic and individuals carrying the slow-acetylator phenotype are more susceptible to benzidine-induced bladder cancer. We have identified yeast artificial chromosome clones encoding AAC-1 and AAC-2 and have used the cloned DNAs as fluorescent probes for in situ hybridization. The hybridization patterns allow assignment of AAC-1 and AAC-2 to chromosome 8p21.3-23.1, a region in which deletions have been associated with bladder cancer [Knowles, Shaw and Proctor (1993) Oncogene 8, 1357-1364].


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